Zobrazeno 1 - 10
of 30
pro vyhledávání: '"Miho Odaka"'
Autor:
Shintaro Suzuki, Koushi Ieki, Satoshi Matsukura, Masatsugu Kurokawa, Miho Odaka, Tetsuya Homma, Mitsuru Adachi, Munehiro Yamaguchi, Shin Watanabe, Yoshitaka Yamamoto
Publikováno v:
Chinese Journal of Cancer Research. 23:232-235
A 79-year-old man was diagnosed as stage IV colon cancer and treated with a modified FOLFOX6 (mFOLFOX6) regimen. On the 12th cycle, we observed erythema and dyspnea. Radiographs showed ground grass opacities. Blood tests showed elevated levels of eos
Autor:
Shin Watanabe, Miho Odaka, Shintaro Suzuki, Hiroko Takeuchi, Koushi Ieki, Masayuki Sato, Munehiro Yamaguchi, Masatsugu Kurokawa, Tetsuya Homma, Mio Kawaguchi, Satoshi Matsukura, Mitsuru Adachi
Publikováno v:
International Archives of Allergy and Immunology. 155:12-20
Background: Interleukin (IL)-33, a new member of the IL-1 cytokine family, has been recognized as a key cytokine that enhances T helper 2-balanced immune regulation through its receptor ST2; however, the function and relationship of the IL-33 and ST2
Autor:
Mitsuru Adachi, Miho Odaka, Satoshi Matsukura, Tsukasa Ohnishi, Shin Watanabe, Tetsuya Homma, Shintaro Suzuki, Takashi Hirose, Mio Kawaguchi, Koushi Ieki, Masayuki Sato, Teruaki Kimura, Robert P. Schleimer, Masatsugu Kurokawa
Publikováno v:
International Archives of Allergy and Immunology. 152:9-17
Background: CCL5/RANTES contributes to prolonged eosinophilic inflammation and asthma exacerbation after a viral infection. We studied the mechanism of CCL5 expression using viral product double-stranded RNA (dsRNA), a ligand of Toll-like receptor 3
Autor:
Tsuyoshi Kasama, Mio Kawaguchi, Mitsuru Adachi, Shin Watanabe, Hiroko Takeuchi, Miho Odaka, Robert P. Schleimer, Hideki Kuga, Tetsuya Homma, Shintaro Suzuki, Koushi Ieki, Fumio Kokubu, Kyoko Nohtomi, Masatsugu Kurokawa, Satoshi Matsukura
Publikováno v:
International Archives of Allergy and Immunology. 143:84-88
Background: Airway smooth muscle (ASM) cells may contribute to the pathogenesis of asthma including airway inflammation and remodeling. We focused our study on the regulation of chemokine expression by cytokines and analyzed the mechanisms of eotaxin
Autor:
Satoshi Matsukura, Koushi Ieki, Mio Kawaguchi, Shintaro Suzuki, Shin Watanabe, Tetsuya Homma, Hiroko Takeuchi, Miho Odaka, Hideki Kuga, Masatsugu Kurokawa, Kyoko Nohtomi, Mitsuru Adachi, Fumio Kokubu
Publikováno v:
International Archives of Allergy and Immunology. 143:80-83
Background: We hypothesized that synthetic double-stranded (ds)RNA may mimic viral infection and reported that dsRNA stimulates expression of inflammatory chemokines through a receptor of dsRNA Toll-like receptor (TLR) 3 in airway epithelial cells. I
Autor:
Yoshiko Sasaki, Mio Kawaguchi, Fumio Kokubu, Hidekazu Ota, Shintaro Suzuki, Shin Watanabe, Masatsugu Kurokawa, Koshi Ieki, Hiroko Takeuchi, Miho Odaka, Tetsuya Homma, Shau Ku Huang, Satoshi Matsukura, Mitsuru Adachi
Publikováno v:
International Archives of Allergy and Immunology. 143:89-94
Background: Interleukin (IL)-17F is a recently discovered cytokine and is derived from a panel of limited cell types, such as activated CD4+ T cells, basophils, and mast cells. IL-17F is known to induce several cytokines and chemokines. However, its
Autor:
Miho Odaka, Hideki Kuga, Mio Kawaguchi, Koushi Ieki, S. Suzuki, Shin Watanabe, Tsuyoshi Kasama, Fumio Kokubu, Masatsugu Kurokawa, Hiroko Takeuchi, Satoshi Matsukura, Mitsuru Adachi
Publikováno v:
Clinical Experimental Allergy. 36:1049-1062
Background We hypothesized that synthetic double-stranded (ds)RNA may mimic viral infection and induce expression of genes related to inflammation in airway epithelial cells. Objective We analysed what gene was up-regulated by synthetic dsRNA poly I
Autor:
Mitsuru Adachi, S. Suzuki, Mio Kawaguchi, Koushi Ieki, Cristiana Stellato, Fumio Kokubu, Satoshi Matsukura, Miho Odaka, Hideki Kuga, Robert P. Schleimer, Hiroko Takeuchi, Shin Watanabe
Publikováno v:
Journal of Allergy and Clinical Immunology. 111:1337-1344
Background: Eotaxin is a chemokine that binds with high affinity and specificity to the chemokine receptor CCR3 and plays an important role in the pathogenesis of allergic disease. Objective: We studied the regulation of eotaxin expression by the T H
Autor:
Fumio Kokubu, Masatsugu Kurokawa, Hiroko Takeuchi, Robert P. Schleimer, Miho Odaka, Mio Kawaguchi, Hideki Kuga, Mitsuru Adachi, Kyoko Notomi, Satoshi Matsukura, M. W. Johnson, Tetsuya Homma
Chemokines ligands of CCR3 including eotaxin/CC chemokine ligand 11 (CCL11) may contribute to the pathogenesis of asthma. These chemokines and a growth factor (TGF-beta) may be involved in the process of airway remodelling.We analysed the effects of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9bb3323e7bcae308edabbc2319c0e95f
https://europepmc.org/articles/PMC3712283/
https://europepmc.org/articles/PMC3712283/
Autor:
Mio Kawaguchi, Shin Watanabe, Miho Odaka, S. Suzuki, Tetsuya Homma, Hiroko Takeuchi, Satoshi Matsukura, Mitsuru Adachi, Koushi Ieki, Takashi Hirose, Masatsugu Kurokawa, Shau Ku Huang, Masayuki Sato, Satoshi Konno
Publikováno v:
International archives of allergy and immunology. 149
Background: Osteopontin (OPN) contributes to the development of T helper 1 (Th1)-mediated immunity and Th1-associated diseases. However, the role of OPN in bronchial asthma is unclear. Corticosteroids reduce airway inflammation, as reflected by the l