Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Miguel Miron-Mendoza"'
Autor:
Miguel Miron-Mendoza, Kara Poole, Sophie DiCesare, Emi Nakahara, Meet Paresh Bhatt, John D. Hulleman, Walter Matthew Petroll
Publikováno v:
Cells, Vol 13, Iss 13, p 1094 (2024)
Vimentin has been reported to play diverse roles in cell processes such as spreading, migration, cell–matrix adhesion, and fibrotic transformation. Here, we assess how vimentin impacts cell spreading, morphology, and myofibroblast transformation of
Externí odkaz:
https://doaj.org/article/6e2992c7c5164c4183bca6ee0999bf15
Effects of Topography and PDGF on the Response of Corneal Keratocytes to Fibronectin-Coated Surfaces
Autor:
Kevin H. Lam, Tarik Z. Shihabeddin, Jacob A. Awkal, Alex M. Najjar, Miguel Miron-Mendoza, Daniel P. Maruri, Victor D. Varner, W. Matthew Petroll, David W. Schmidtke
Publikováno v:
Journal of Functional Biomaterials, Vol 14, Iss 4, p 217 (2023)
During corneal wound healing, corneal keratocytes are exposed to both biophysical and soluble cues that cause them to transform from a quiescent state to a repair phenotype. How keratocytes integrate these multiple cues simultaneously is not well und
Externí odkaz:
https://doaj.org/article/e883c5af1a8e435d83ab39f304bac3cc
Autor:
Miguel Miron-Mendoza, Dalia Vazquez, Nerea García-Rámila, Hikaru R. Ikebe, W. Matthew Petroll
Publikováno v:
Bioengineering, Vol 7, Iss 3, p 89 (2020)
We previously reported that corneal fibroblasts within 3D fibrin matrices secrete, bind, and organize fibronectin into tracks that facilitate cell spreading and migration. Other cells use these fibronectin tracks as conduits, which leads to the devel
Externí odkaz:
https://doaj.org/article/22bbfd5d9eff41bfb8b9a30c35b351ea
ECM Stiffness Controls the Activation and Contractility of Corneal Keratocytes in Response to TGF-β1
Autor:
Victor D. Varner, W. Matthew Petroll, Miguel Miron-Mendoza, David W. Schmidtke, Daniel P. Maruri, Pouriska B. Kivanany, Joshua M. Hack
Publikováno v:
Biophys J
After surgery or traumatic injury, corneal wound healing can cause a scarring response that stiffens the tissue and impairs ocular function. This fibrosis is caused in part by the activation of corneal keratocytes from a native mechanically quiescent
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1014fd6a47aad94298aad00c3474fd60
https://europepmc.org/articles/PMC7677126/
https://europepmc.org/articles/PMC7677126/
Autor:
W. Matthew Petroll, Dalia Vazquez, Hikaru R. Ikebe, Nerea García-Rámila, Miguel Miron-Mendoza
Publikováno v:
Bioengineering
Bioengineering, Vol 7, Iss 89, p 89 (2020)
Volume 7
Issue 3
Bioengineering, Vol 7, Iss 89, p 89 (2020)
Volume 7
Issue 3
We previously reported that corneal fibroblasts within 3D fibrin matrices secrete, bind, and organize fibronectin into tracks that facilitate cell spreading and migration. Other cells use these fibronectin tracks as conduits, which leads to the devel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6f29bf7a32fb2be42113f5965626ad91
Publikováno v:
Matrix Biology. 64:69-80
We previously reported that fibroblasts migrating within 3-D collagen matrices move independently, whereas fibroblasts within 3-D fibrin matrices form an interconnected network. Similar networks have been identified previously during in vivo corneal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e5c8d82fa20534f51f2bcdaf1841f312
https://doi.org/10.1016/j.matbio.2017.06.001
https://doi.org/10.1016/j.matbio.2017.06.001
Publikováno v:
Biomaterials. 31:6425-6435
In three dimensional collagen matrices, cell motile activity results in collagen translocation, cell spreading and cell migration. Cells can penetrate into the matrix as well as spread and migrate along its surface. In the current studies, we quantit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5d92387a75b5fd3b0e10c03911f6f83
https://doi.org/10.1016/j.biomaterials.2010.04.064
https://doi.org/10.1016/j.biomaterials.2010.04.064
Autor:
Blaine Bisel, Joachim Seemann, Nobuhiro Nakamura, Yanzhuang Wang, Shin-ichiro Yoshimura, Danming Tang, Yi Xiang, Miguel Miron-Mendoza, Jen Hsuan Wei
Publikováno v:
The Journal of Cell Biology
Directed cell migration requires the orientation of the Golgi and centrosome toward the leading edge. We show that stimulation of interphase cells with the mitogens epidermal growth factor or lysophosphatidic acid activates the extracellular signal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::00c3c34fa3d78228522cae1caba629d2
http://europepmc.org/articles/PMC2528584
http://europepmc.org/articles/PMC2528584
The generation of cellular forces and the application of these physical forces to the ECM play a central role in mediating matrix patterning and remodeling during fundamental processes such as developmental morphogenesis and wound healing. In additio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::101f23931cc71d8887650564c1e5ce44
https://europepmc.org/articles/PMC4379425/
https://europepmc.org/articles/PMC4379425/
Extracellular matrix (ECM) supplies both physical and chemical signals to cells and provides a substrate through which fibroblasts migrate during wound repair. To directly assess how ECM composition regulates this process, we used a nested 3D matrix
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0359920b399c3d8186c5f2a67339e972
https://europepmc.org/articles/PMC3571722/
https://europepmc.org/articles/PMC3571722/