Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Miguel A Lopez-Nevot"'
Autor:
Amalia Lamana, Alejandro Balsa, Blanca Rueda, Ana M Ortiz, Laura Nuño, Maria Eugenia Miranda-Carus, Maria F Gonzalez-Escribano, Miguel A Lopez-Nevot, Dora Pascual-Salcedo, Javier Martin, Isidoro González-Álvaro
Publikováno v:
PLoS ONE, Vol 7, Iss 8, p e43661 (2012)
BACKGROUND: The number of copies of the HLA-DRB1 shared epitope, and the minor alleles of the STAT4 rs7574865 and the PTPN22 rs2476601 polymorphisms have all been linked with an increased risk of developing rheumatoid arthritis. In the present study,
Externí odkaz:
https://doaj.org/article/3b1c4ed959dc4aae8414ed181cc6eb87
Autor:
Steven G.E. Marsh, Miguel Angel Lopez Nevot, Francisco Ruiz-Cabello, Isabel Maleno, Laura Paco, Teresa Rodríguez, Graham Pawelec, Rosa Mendez, Chrissy h. Roberts, Federico Garrido, I. Anthony Dodi
Publikováno v:
Cancer Immunology, Immunotherapy. 54:141-148
Malignant transformation of cells is frequently associated with abnormalities in human leukocyte antigen (HLA) expression. MHC class I loss or down-regulation in cancer cells is a major immune escape route used by a large variety of human tumours to
Autor:
Jose A, Lopez-Escamez, Pablo, Saenz-Lopez, Lourdes, Acosta, Antonia, Moreno, Irene, Gazquez, Herminio, Perez-Garrigues, Alicia, Lopez-Nevot, Miguel A, Lopez-Nevot
Publikováno v:
The Laryngoscope. 120(1)
Bilateral Meniere's disease (BMD) is a severe disease that usually results in bilateral severe or profound sensorineural hearing loss and chronic disequilibrium with loss of vestibular function. We examined single nucleotide polymorphisms (SNPs) in t
Autor:
Natalia, Aptsiauri, Teresa, Cabrera, Angel, Garcia-Lora, Miguel Angel, Lopez-Nevot, Francisco, Ruiz-Cabello, Federico, Garrido
Publikováno v:
International review of cytology. 256
MHC class I antigens play a crucial role in the interaction of tumor cells with the host immune system, in particular, in the presentation of peptides as tumor-associated antigens to cytotoxic lymphocytes (CTLs) and in the regulation of cytolytic act
Autor:
Javier, Martin, Cristina, Perez-Armengol, Jose A, Miranda-Filloy, Jose R, Vilchez, Miguel A, Lopez-Nevot, Carlos, Garcia-Porrua, Miguel A, Gonzalez-Gay
Publikováno v:
The Journal of rheumatology. 33(2)
Giant cell arteritis (GCA) is a vasculitis preferentially involving large and middle-sized arteries in the elderly. The nuclear factor of k-light polypeptide gene enhancer in B cells (NF-kB) is a family of 5 proteins expressed in most cells that func
Autor:
Miguel A, Gonzalez-Gay, Javier, Oliver, Elena, Sanchez, Carlos, Garcia-Porrua, Laura, Paco, Miguel A, Lopez-Nevot, William E R, Ollier, Javier, Martin
Publikováno v:
The Journal of rheumatology. 32(11)
To assess the contribution of 2 polymorphisms within the inducible nitric oxide (NOS2A) promoter region to susceptibility to giant cell arteritis (GCA).One hundred three patients with biopsy-proven GCA and 198 ethnically matched controls from the Lug
Autor:
Blanca, Rueda, Miguel A, Lopez-Nevot, Maria J, Lopez-Diaz, Carlos, Garcia-Porrua, Javier, Martín, Miguel A, Gonzalez-Gay
Publikováno v:
The Journal of rheumatology. 32(9)
Angiogenesis, the formation of new blood vessels, may play a role in giant cell arteritis (GCA), the most common type of systemic vasculitis in the elderly in Western countries. Vascular endothelial growth factor (VEGF) is one of the most important p
Autor:
Miguel A, Gonzalez-Gay, Javier, Oliver, Gisela, Orozco, Carlos, Garcia-Porrua, Miguel A, Lopez-Nevot, Javier, Martin
Publikováno v:
The Journal of rheumatology. 32(8)
To assess the possible association between PTPN22 1858C(R)T polymorphism and susceptibility to giant cell arteritis (GCA) and to determine if this polymorphism is implicated in the clinical expression of this vasculitis.Ninety-six patients with biops
Autor:
Javier, Martin, Laura, Paco, Maria P, Ruiz, Miguel A, Lopez-Nevot, Carlos, Garcia-Porrua, Mahsa M, Amoli, Maria C, Calviño, William E R, Ollier, Miguel A, Gonzalez-Gay
Publikováno v:
The Journal of rheumatology. 32(6)
To assess the contribution of 2 polymorphisms within the inducible nitric oxide (NOS2A) promoter region to the susceptibility to Henoch-Schönlein purpura (HSP), and to determine if implications exist with severe systemic complications of HSP, in par
Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.
K zobrazení výsledku je třeba se přihlásit.