Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Microvessels/cytology"'
Autor:
Jan C. M. van Hest, Edwin de Jong, Loai K. E. A. Abdelmohsen, Inge S. Zuhorn, David S. Williams
Publikováno v:
Journal of Controlled Release, 289, 14-22. Elsevier
Journal of Controlled Release, 289, 14-22. Elsevier Bedrijfsinformatie b.v.
Journal of Controlled Release, 289, 14-22. Elsevier Bedrijfsinformatie b.v.
The delivery of therapeutics to the brain is greatly hampered by the blood-brain barrier (BBB). The use of nanoparticles that can cross the BBB via the process of receptor-mediated transcytosis at blood-brain barrier endothelial cells seems a promisi
Publikováno v:
Allergy. 63:1610-1616
Background: Some severe asthma patients are characterized by elevated levels of tumor necrosis factor alpha (TNF-α) and neutrophilic inflammation in the airways. Although such phenotypic changes in asthma might contribute to corticosteroid refractor
Group B Streptococcus Hijacks the Host Plasminogen System to Promote Brain Endothelial Cell Invasion
Autor:
Patrick Trieu-Cuot, Kwang Sik Kim, Margarida Lima, Adília Ribeiro, Paula Ferreira, Joana Alves, Vanessa Magalhães, Elva Bonifácio Andrade
Publikováno v:
PLoS ONE
PLoS ONE, 2013, 8 (5), pp.e63244. ⟨10.1371/journal.pone.0063244⟩
PLoS ONE, Vol 8, Iss 5, p e63244 (2013)
PLoS ONE, Public Library of Science, 2013, 8 (5), pp.e63244. ⟨10.1371/journal.pone.0063244⟩
Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
Agência para a Sociedade do Conhecimento (UMIC)-FCT-Sociedade da Informação
instacron:RCAAP
PLoS ONE, 2013, 8 (5), pp.e63244. ⟨10.1371/journal.pone.0063244⟩
PLoS ONE, Vol 8, Iss 5, p e63244 (2013)
PLoS ONE, Public Library of Science, 2013, 8 (5), pp.e63244. ⟨10.1371/journal.pone.0063244⟩
Repositório Científico de Acesso Aberto de Portugal (Repositórios Cientìficos)
Agência para a Sociedade do Conhecimento (UMIC)-FCT-Sociedade da Informação
instacron:RCAAP
Group B Streptococcus (GBS) is the leading cause of meningitis in neonates. We have previously shown that plasminogen, once recruited to the GBS cell surface and converted into plasmin by host-derived activators, leads to an enhancement of bacterial