Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Michiro Wakabayashi"'
Autor:
Hiroyuki Inoshita, Satoshi Horikoshi, Misao Matsushita, Yasuhiko Tomino, Kisara Onda, Masaya Ishii, Isao Ohsawa, Michiro Wakabayashi, Gaku Kusaba, Hiroyuki Ohi
Publikováno v:
Therapeutic Apheresis and Dialysis. 15:441-447
Mannose-binding lectin (MBL), L-ficolin and MBL associated serine protease-2 (MASP-2) are molecules involved in initiation of the lectin pathway (LP) in the complement system. Although MBL deficiency is observed in almost 10% of healthy people, studi
Autor:
Ichiyu Shou, Mitumine Fukui, Kunimi Maeda, Yasuhiko Tomino, Michiro Wakabayashi, Chieko Hamada, Takeshi Hayashi, Satoshi Horikoshi
Publikováno v:
Journal of International Medical Research. 37:205-213
The oral adsorbent AST-120 has been widely used in Japan to delay the initiation of dialysis therapy in patients with chronic renal failure. This study evaluated the long-term effects of AST-120 in patients with chronic renal failure who had not prev
Autor:
Kisara Onda, Isao Ohsawa, Hiroyuki Ohi, Satoshi Horikoshi, Yasuhiko Tomino, Teizo Fujita, Mariko Tamano, Michiro Wakabayashi
Publikováno v:
Journal of Clinical Laboratory Analysis. 21:77-84
IgA nephropathy (IgAN) is the most common form of chronic glomerulonephritis. Although glomerular deposition of complement components is well known, the evidence of serological complement activation in IgAN is inconclusive. We hypothesized that serum
Publikováno v:
Nephron Experimental Nephrology. 104:e89-e95
Background: Complement receptor type 1 on erythrocytes (E-CR1) plays important roles not only in the regulation of complement activation, but also the clearance of immune complexes. Reduced E-CR1 was previously found in patients undergoing hemodialys
Autor:
Kisara Onda, Mariko Tamano, Hiroyuki Ohi, Satoshi Horikoshi, Kunimi Maeda, Michiro Wakabayashi, Yasuhiko Tomino
Publikováno v:
Juntendo Medical Journal. 51:334-343
Autor:
Teizo Fujita, Isao Ohsawa, Mariko Tamano, Hiroyuki Ohi, Yasuhiko Tomino, Satoshi Horikoshi, Kisara Onda, Akie Toki, Satoshi Mano, Michiro Wakabayashi
Publikováno v:
BMC Nephrology
BMC Nephrology, Vol 12, Iss 1, p 64 (2011)
BMC Nephrology, Vol 12, Iss 1, p 64 (2011)
Background Glomerular damage in IgA nephropathy (IgAN) is mediated by complement activation via the alternative and lectin pathways. Therefore, we focused on molecules stabilizing and regulating the alternative pathway C3 convertase in urine which mi
Autor:
Masaya, Ishii, Isao, Ohsawa, Hiroyuki, Inoshita, Gaku, Kusaba, Kisara, Onda, Michiro, Wakabayashi, Hiroyuki, Ohi, Satoshi, Horikoshi, Misao, Matsushita, Yasuhiko, Tomino
Publikováno v:
Therapeutic apheresis and dialysis : official peer-reviewed journal of the International Society for Apheresis, the Japanese Society for Apheresis, the Japanese Society for Dialysis Therapy. 15(5)
Mannose-binding lectin (MBL), L-ficolin and MBL associated serine protease-2 (MASP-2) are molecules involved in initiation of the lectin pathway (LP) in the complement system. Although MBL deficiency is observed in almost 10% of healthy people, studi
Autor:
Yukihiko Takeda, Chieko Hamada, Satoshi Horikoshi, Toshinao Tsuge, Yasuhiko Tomino, Michiro Wakabayashi, T Yamazaki, Tomohito Nishitani, Y Tanaka, Kenji Satake, H Fukuda, Kunimi Maeda, A Ohtani, Takuya Seto, Atushi Kurusu, Ichiyu Shou
Publikováno v:
Clinical nephrology. 68(6)
Aims In this study, dose-response of the serum potassium-lowering effect of a calcium polystyrene sulfonate (PS) preparation was investigated. Changes in the serum potassium level were also examined with or without application of a RAAS inhibitor, wh
Autor:
Tatsuya Aoki, Kaori Yamada, Kunimi Maeda, Kazuhiko Funabiki, Shigeru Kaneko, Keiko Obayashi, Michiro Wakabayashi, Yasuhiko Tomino, Shinji Hagiwara, Masakazu Matsumoto, Mitsuo Tanimoto, Tomohito Gohda, Takamichi Ito, Satoshi Horikoshi, Maki Murakoshi
Publikováno v:
Nephrology (Carlton, Vic.). 11(1)
SUMMARY: Background: In type 2 diabetic nephropathy, there is no animal model which has been completely matched with humans. Advanced glycation end products (AGE) and transforming growth factor-beta (TGF-β) are closely related to hyperglycaemia and