Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Michinori, Koebisu"'
Autor:
Osamu Nozawa, Muneaki Miyata, Hajime Shiotani, Takeshi Kameyama, Ryouhei Komaki, Tatsuhiro Shimizu, Toshihiko Kuriu, Yutaro Kashiwagi, Yuka Sato, Michinori Koebisu, Atsu Aiba, Shigeo Okabe, Kiyohito Mizutani, Yoshimi Takai
Publikováno v:
Development. 150
Ramified, polarized protoplasmic astrocytes interact with synapses via perisynaptic astrocyte processes (PAPs) to form tripartite synapses. These astrocyte-synapse interactions mutually regulate their structures and functions. However, molecular mech
Autor:
Osamu, Nozawa, Muneaki, Miyata, Hajime, Shiotani, Takeshi, Kameyama, Ryouhei, Komaki, Tatsuhiro, Shimizu, Toshihiko, Kuriu, Yutaro, Kashiwagi, Yuka, Sato, Michinori, Koebisu, Atsu, Aiba, Shigeo, Okabe, Kiyohito, Mizutani, Yoshimi, Takai
Publikováno v:
Development (Cambridge, England).
Ramified, polarized protoplasmic astrocytes interact with synapses via perisynaptic astrocyte processes (PAPs) to form tripartite synapses. These astrocyte-synapse interactions mutually regulate their structures and functions. However, molecular mech
Publikováno v:
Folia Pharmacologica Japonica. 156:114-119
Orexin receptor antagonists have been approved for insomnia, and the insomnia pharmacotherapy is being greatly progressed. Orexin is a neuropeptide produced in the lateral hypothalamic area, and its physiological role has been suggested to be a key m
Publikováno v:
Nihon yakurigaku zasshi. Folia pharmacologica Japonica. 156(2)
Orexin receptor antagonists have been approved for insomnia, and the insomnia pharmacotherapy is being greatly progressed. Orexin is a neuropeptide produced in the lateral hypothalamic area, and its physiological role has been suggested to be a key m
Autor:
Michinori, Koebisu, Takayuki, Ishida
Publikováno v:
Nihon yakurigaku zasshi. Folia pharmacologica Japonica. 155(4)
Parkinson's disease is a neurodegenerative disorder characterized by the degenerative loss of dopaminergic neurons in the substantia nigra. Dopamine deficiency is thought to disrupt motor control of the basal ganglia and cause characteristic motor sy