Muscle-specific interaction of caveolin isoforms: differential complex formation between caveolins in fibroblastic vs. muscle cells

Autor: Federica Sotgia, Philippe G. Frank, Alex W. Cohen, Michela Battista, Michael P. Lisanti, William Schubert, Hyangkyu Lee, Michelle W.-C. Cheung, Franco Capozza

Publikováno v: American Journal of Physiology-Cell Physiology. 288:C677-C691

It is generally well accepted that caveolin-3 expression is muscle specific, whereas caveolin-1 and -2 are coexpressed in a variety of cell types, including adipocytes, endothelial cells, epithelial cells, and fibroblasts. Caveolin-1 and -2 are known

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aedd694c9afccadbac6e0b7c634e921e
https://doi.org/10.1152/ajpcell.00232.2004

Combined Loss of INK4a and Caveolin-1 Synergistically Enhances Cell Proliferation and Oncogene-induced Tumorigenesis

Autor: Philipp E. Scherer, Richard G. Pestell, Alex W. Cohen, Puneeth Iyengar, Babak Razani, Terence M. Williams, Hyangkyu Lee, Michelle W.-C. Cheung, Michael P. Lisanti

Publikováno v: Journal of Biological Chemistry. 279:24745-24756

Tumorigenesis is a multistep process that involves a series of genetic changes or "multiple hits," leading to alterations in signaling, proliferation, immortalization, and transformation. Many of the molecular factors that govern tumor initiation and

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::7aa0a309ca8e765df14dcbea0502745b
https://doi.org/10.1074/jbc.m402064200

Role of caveolin-1 in the regulation of lipoprotein metabolism

Autor: Michael P. Lisanti, Michelle W.-C. Cheung, Philippe G. Frank, Stephanos Pavlides, Kristin M. Daumer

Publikováno v: American Journal of Physiology-Cell Physiology
American Journal of Physiology-Cell Physiology, American Physiological Society, 2008, 295 (1), pp.C242-C248. ⟨10.1152/ajpcell.00185.2008⟩

Lipoprotein metabolism plays an important role in the development of several human diseases, including coronary artery disease and the metabolic syndrome. A good comprehension of the factors that regulate the metabolism of the various lipoproteins is

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::712a84244a521336121517ca3ce6a475
https://pubmed.ncbi.nlm.nih.gov/18508910

Combined loss of INK4a and caveolin-1 synergistically enhances cell proliferation and oncogene-induced tumorigenesis: role of INK4a/CAV-1 in mammary epithelial cell hyperplasia

Autor: Terence M, Williams, Hyangkyu, Lee, Michelle W-C, Cheung, Alex W, Cohen, Babak, Razani, Puneeth, Iyengar, Philipp E, Scherer, Richard G, Pestell, Michael P, Lisanti

Publikováno v: The Journal of biological chemistry. 279(23)

Tumorigenesis is a multistep process that involves a series of genetic changes or "multiple hits," leading to alterations in signaling, proliferation, immortalization, and transformation. Many of the molecular factors that govern tumor initiation and

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::47e5b9c63094643a57fd2916e32fb412
https://pubmed.ncbi.nlm.nih.gov/15044451

Caveolin-3 knock-out mice develop a progressive cardiomyopathy and show hyperactivation of the p42/44 MAPK cascade

Autor: Stephen M. Factor, Richard N. Kitsis, Herbert B. Tanowitz, George J. Christ, Baiyu Tang, Michael P. Lisanti, David S. Park, Michelle W.-C. Cheung, Madhulika Chandra, Alex W. Cohen, Jamshid Shirani, Scott Eric Woodman, Linda A. Jelicks

Publikováno v: The Journal of biological chemistry. 277(41)

A growing body of evidence suggests that muscle cell caveolae may function as specialized membrane micro-domains in which the dystrophin-glycoprotein complex and cellular signaling molecules reside. Caveolin-3 (Cav-3) is the only caveolin family memb

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fe958f9b698b010b25dcad7d349f2f6a
https://pubmed.ncbi.nlm.nih.gov/12138167