Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Michela Palmisano"'
Autor:
Alessandra Gargano, Bolanle Fatimat Olabiyi, Michela Palmisano, Andreas Zimmer, Andras Bilkei-Gorzo
Publikováno v:
Frontiers in Neuroscience, Vol 17 (2023)
IntroductionAging is associated with a decline in cognitive abilities, including memory and attention. It is generally accepted that age-related histological changes such as increased neuroinflammatory glial activity and a reduction in the number of
Externí odkaz:
https://doaj.org/article/3129527f49f449b5adef632c098a64e7
Autor:
Michela Palmisano, Alessandra Gargano, Bolanle Fatimat Olabiyi, Beat Lutz, Andras Bilkei-Gorzo
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 1, p 26 (2022)
Endocannabinoid system activity declines with age in the hippocampus, along with the density of the cannabinoid receptor type-1 (CB1). This process might contribute to brain ageing, as previous studies showed that the constitutive deletion of the CB1
Externí odkaz:
https://doaj.org/article/4e35024927f14593ba32303e693ad90c
Autor:
Prakash Nidadavolu, Andras Bilkei-Gorzo, Michael Krämer, Britta Schürmann, Michela Palmisano, Eva C. Beins, Burkhard Madea, Andreas Zimmer
Publikováno v:
Frontiers in Aging Neuroscience, Vol 13 (2021)
Decline in cognitive performance, an aspect of the normal aging process, is influenced by the endocannabinoid system (ECS). Cannabinoid receptor 1 (CB1) signaling diminishes with advancing age in specific brain regions that regulate learning and memo
Externí odkaz:
https://doaj.org/article/331c0a3df30e4845b83752232033d0a3
Autor:
Madhusmita P. Sahu, Yago Pazos-Boubeta, Anna Steinzeig, Katja Kaurinkoski, Michela Palmisano, Olgierd Borowecki, Timo Petteri Piepponen, Eero Castrén
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 14 (2021)
Neurotrophin brain-derived neurotrophic factor (BDNF) and neurotransmitter serotonin (5-HT) regulate each other and have been implicated in several neuronal mechanisms, including neuroplasticity. We have investigated the effects of BDNF on serotonerg
Externí odkaz:
https://doaj.org/article/2e04428e57ba411096daf434288f7bde
Publikováno v:
Oncology Reviews, Vol 6, Iss 1 (2012)
Hematopoiesis, the process by which the hematopoietic stem cells and progenitors differentiate into blood cells of various lineages, involves complex interactions of transcription factors that modulate the expression of downstream genes and mediate p
Externí odkaz:
https://doaj.org/article/76322914300f45aa92cce8987f9a9913
Autor:
Michela Palmisano, Tiziana Grafone, Emanuela Ottaviani, Nicoletta Testoni, Michele Baccarani, Giovanni Martinelli
Publikováno v:
Haematologica, Vol 92, Iss 9 (2007)
NPM1 mutations have been reported to be the most frequent mutations in acute myeloid leukemia (AML). They are associated with a wide spectrum of morphologic subtypes of AML, normal karyotype and FLT3 mutations. The high frequency of NPM1 mutations mi
Externí odkaz:
https://doaj.org/article/82efb5cbacbe493dbf67e2b31b706861
Autor:
Madhusmita P, Sahu, Yago, Pazos-Boubeta, Anna, Steinzeig, Katja, Kaurinkoski, Michela, Palmisano, Olgierd, Borowecki, Timo Petteri, Piepponen, Eero, Castrén
Publikováno v:
Frontiers in Molecular Neuroscience
Neurotrophin brain-derived neurotrophic factor (BDNF) and neurotransmitter serotonin (5-HT) regulate each other and have been implicated in several neuronal mechanisms, including neuroplasticity. We have investigated the effects of BDNF on serotonerg
Autor:
Michela Palmisano, Tiziana Grafone, Michele Baccarani, Sergio Storti, Giovanni Martinelli, Chiara Nicci, Ottaviani Emanuela, Alberto M. Martelli
Publikováno v:
Hematological oncology. 26(3)
FLT3 mutation and overexpression in most acute myeloid leukaemia (AML) patients make this tyrosine kinase receptor an attractive therapeutic target. FLT3 kinase inhibitors are actually in clinical trials, thus it is critical to develop a reproducible
Autor:
Nicoletta Testoni, Stefania Paolini, Tiziana Grafone, Emanuela Ottaviani, Cristina Papayannidis, Michela Palmisano, Matteo Renzulli, Michele Baccarani, Giovanni Martinelli
Acute myeloid leukemia (AML) remains the most common form of leukemia and the most common cause of leukemia death. Although conventional chemotherapy can cure between 25 and 45% of AML patients, the majority of patients die after relapse or of compli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::178bfe1e957ed65669e0b2d2ddc6db71
http://hdl.handle.net/11585/68493
http://hdl.handle.net/11585/68493
Autor:
Giovanni Martinelli, Emanuela Ottaviani, Michele Baccarani, Michela Palmisano, Tiziana Grafone, Nicoletta Testoni
Publikováno v:
Haematologica. 92:1268-1269
NPM1 mutations have been reported to be the most frequent mutations in acute myeloid leukemia (AML). They are associated with a wide spectrum of morphologic subtypes of AML, normal karyotype and FLT3 mutations. The high frequency of NPM1 mutations mi