Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Michael Ziemba"'
Autor:
Michael Ziemba, Molly Barkhouse, Kitipong Uaesoontrachoon, Mamta Giri, Yetrib Hathout, Utkarsh J Dang, Heather Gordish-Dressman, Kanneboyina Nagaraju, Eric P Hoffman
Publikováno v:
PLoS ONE, Vol 16, Iss 2, p e0246507 (2021)
Duchenne muscular dystrophy is initiated by dystrophin deficiency, but downstream pathophysiological pathways such as membrane instability, NFĸB activation, mitochondrial dysfunction, and induction of TGFβ fibrosis pathways are thought to drive the
Externí odkaz:
https://doaj.org/article/ce5d6cb03d0e4faca8116cb2ca183f89
Autor:
Dan Cox, Eric P. Hoffman, Alan J Russell, Michael Ziemba, Benjamin L. Barthel, Volker Straub, Marissa L Barbieri
Publikováno v:
Musclenerve. 64(1)
INTRODUCTION One of the hallmarks of injured skeletal muscle is the appearance of elevated skeletal muscle proteins in circulation. Human skeletal muscle generally consists of a mosaic of slow (type I) and fast (type IIa, IIx/d) fibers, defined by th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::194239f41ef430f75f43689f63eafb92
https://pubmed.ncbi.nlm.nih.gov/34076279
https://pubmed.ncbi.nlm.nih.gov/34076279
Autor:
Yetrib Hathout, Utkarsh J. Dang, Eric P. Hoffman, Molly Barkhouse, Mamta Giri, Heather Gordish-Dressman, Kitipong Uaesoontrachoon, Michael Ziemba, Kanneboyina Nagaraju
Publikováno v:
PLoS ONE, Vol 16, Iss 2, p e0246507 (2021)
PLoS ONE
PLoS ONE
Duchenne muscular dystrophy is initiated by dystrophin deficiency, but downstream pathophysiological pathways such as membrane instability, NFĸB activation, mitochondrial dysfunction, and induction of TGFβ fibrosis pathways are thought to drive the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3af7fff4b4919d057bd1818c7c2bf550
https://doaj.org/article/ce5d6cb03d0e4faca8116cb2ca183f89
https://doaj.org/article/ce5d6cb03d0e4faca8116cb2ca183f89
Autor:
Cinrg Vamorolone, Michael Ziemba, Yetrib Hathout, Eric P. Hoffman, Laurie S. Conklin, Paula R. Clemens, Investigators, Utkarsh J. Dang
Publikováno v:
Human Molecular Genetics
Duchenne muscular dystrophy (DMD) is caused by loss of dystrophin in muscle, and while all patients share the primary gene and biochemical defect, there is considerable patient–patient variability in clinical symptoms. We sought to develop multivar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ecc1c0dc28762049332443eb2f28ea1c
https://pubmed.ncbi.nlm.nih.gov/32592467
https://pubmed.ncbi.nlm.nih.gov/32592467