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pro vyhledávání: '"Michael Reinbold"'
Autor:
Josef Krieglstein, Tobias Kewitz, Uwe Kirchhefer, Oliver Hofnagel, Gabriele Weissen-Plenz, Michael Reinbold, Susanne Klumpp
Publikováno v:
PLoS ONE, Vol 5, Iss 3, p e9561 (2010)
BackgroundMono-unsaturated fatty acids (MUFAs) like oleic acid have been shown to cause apoptosis of cultured endothelial cells by activating protein phosphatase type 2C alpha and beta (PP2C). The question arises whether damage of endothelial or othe
Externí odkaz:
https://doaj.org/article/640c217328584e2bbf7d3c91fcdc2398
Autor:
Michael Reinbold
Wenn hinter der Dorfidylle mörderische Abgründe lauern: Der Kriminalroman »Tod im Heidekloster« von Michael Reinbold jetzt als eBook bei dotbooks. Norddeutschland, 1948. Der Kirchenorganist einer kleinen Gemeinde bei Lüneburg treibt tot im Klost
Publikováno v:
Molecular nutritionfood research. 52(5)
Certain unsaturated fatty acids (UFAs), cleaved from lipoproteins, are known to activate the serine/threonine protein phosphatase type 2C (PP2C) alpha- and beta-isoforms. To investigate the role of UFAs in apoptosis of endothelial cells, we coculture
Autor:
Michael Reinbold, Tobias Kewitz, Birgit Hufnagel, Stefanie Schwarz, Josef Krieglstein, Susanne Klumpp, Melanie Dworak
Publikováno v:
European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences. 35(5)
In previous work we have demonstrated that protein phosphatase type 2C (PP2C) alpha and beta can be activated by mono-unsaturated fatty acids (MUFAs) leading to apoptosis of cultured endothelial cells. In the present paper we could show that saturate
Autor:
Uwe Kirchhefer, Susanne Klumpp, Gabriele Weißen-Plenz, Tobias Kewitz, Oliver Hofnagel, Josef Krieglstein, Michael Reinbold
Publikováno v:
PLoS ONE, Vol 5, Iss 3, p e9561 (2010)
PLoS ONE
PLoS ONE
BackgroundMono-unsaturated fatty acids (MUFAs) like oleic acid have been shown to cause apoptosis of cultured endothelial cells by activating protein phosphatase type 2C alpha and beta (PP2C). The question arises whether damage of endothelial or othe