Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Michael R. Quinn"'
Publikováno v:
Clinical Immunology. 102:179-184
Previously, we described the inhibition of proinflammatory mediators such as nitric oxide, tumor necrosis factor-alpha (TNF-alpha), and prostaglandin E2 by taurine chloramine (Tau-Cl) in activated rodent macrophages. We also demonstrated that Tau-Cl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::01d089a382246a80f996643cd298416e
https://doi.org/10.1006/clim.2001.5160
https://doi.org/10.1006/clim.2001.5160
Publikováno v:
Immunology Letters. 70:9-14
Taurine monochloramine (Tau-Cl) is formed through the actions of a halide-dependent myeloperoxidase system associated with polymorphonuclear leukocytes (PMN). Tau-Cl inhibits production of inflammatory mediators by activated macrophages, and PMN. Rec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c9348734e1672f4cf3782f1500a54ea3
https://doi.org/10.1016/s0165-2478(99)00119-4
https://doi.org/10.1016/s0165-2478(99)00119-4
Publikováno v:
Journal of Leukocyte Biology. 61:161-166
Recent studies demonstrate that taurine chloramine (Thu-Cl) inhibits production of nitric oxide (NO) and other proinflammatory mediators in cultured macrophages when added to the media at the time of activation. Because Thu-Cl may react with various
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a8816a0b1c94b5e97359d56467d1ef75
https://doi.org/10.1002/jlb.61.2.161
https://doi.org/10.1002/jlb.61.2.161
Publikováno v:
Immunopharmacology. 34:89-95
Taurine chloramine (Tau-C1) inhibits production of nitric oxide (NO) by activated peritoneal macrophages and attenuates accumulation of tumor necrosis factor-alpha (TNF-α) in the culture media, similar to that previously reported for activated RAW 2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::af902172a69949d1072862355528a0a5
https://doi.org/10.1016/0162-3109(96)00113-0
https://doi.org/10.1016/0162-3109(96)00113-0
Publikováno v:
Advances in experimental medicine and biology. 526
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a1d7e1ee62bd2bf903189d9533ba0131
https://pubmed.ncbi.nlm.nih.gov/12908620
https://pubmed.ncbi.nlm.nih.gov/12908620
Publikováno v:
Advances in experimental medicine and biology. 526
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e912bd8d026a0c222648c3acbd581e50
https://pubmed.ncbi.nlm.nih.gov/12908618
https://pubmed.ncbi.nlm.nih.gov/12908618
Publikováno v:
Advances in experimental medicine and biology. 526
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::5acfd77359d30e6afe477d3678bda5cc
https://pubmed.ncbi.nlm.nih.gov/12908621
https://pubmed.ncbi.nlm.nih.gov/12908621
Publikováno v:
Advances in Experimental Medicine and Biology ISBN: 9781461349136
Taurine chloramine (Tau-Cl) is produced through the actions of a halide-dependent myeloperoxidase system (MPO) that is associated primarily with polymorphonuclear leukocytes (PMN)1–3. Tau-Cl may be synthesized directly by MPO within the intracellul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::068fa538bd5bb11bc95f4c9bac8f7059
https://doi.org/10.1007/978-1-4615-0077-3_45
https://doi.org/10.1007/978-1-4615-0077-3_45
Publikováno v:
Advances in Experimental Medicine and Biology ISBN: 9781461349136
Taurine protects against tissue damage in a variety of models involving inflammation, especially the lung1,2. The mechanism of taurine protection is not well understood but the ability of taurine to attenuate the toxic effects of HOCl/OCl- by formati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::057fa01d06a7e8dc50997aedc9c5de1e
https://doi.org/10.1007/978-1-4615-0077-3_42
https://doi.org/10.1007/978-1-4615-0077-3_42
Publikováno v:
Advances in Experimental Medicine and Biology ISBN: 9781461349136
Microglia are considered to be brain resident macrophages and along with astrocytes are the major immunoresponsive cells in the CNS1,2. When activated by bacterial endotoxin or cytokines, microglia respond rapidly by proliferating, changing morpholog
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c2ab4d355701579bcc99a2f1c9bdc4c5
https://doi.org/10.1007/978-1-4615-0077-3_44
https://doi.org/10.1007/978-1-4615-0077-3_44