Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Michael J. Brabec"'
Autor:
Jeanne Manson, Michael J. Brabec, Judy Buelke-Sam, Gary P. Carlson, Robert E. Chapin, John B. Favor, Lawrence J. Fischer, Dale Hattis, Peter S.J. Lees, Sally Perreault-Darney, Joe Rutledge, Thomas J. Smith, Raymond R. Tice, Peter Working
Publikováno v:
Birth Defects Research Part B: Developmental and Reproductive Toxicology. 74:17-113
Jeanne Manson, Michael J. Brabec, Judy Buelke-Sam, Gary P. Carlson, Robert E. Chapin, John B. Favor, Lawrence J. Fischer, Dale Hattis, Peter S.J. Lees, Sally Perreault-Darney, Joe Rutledge, Thomas J. Smith, Raymond R. Tice and Peter Working Universit
Autor:
Marie Stiborová, Jitka Ulrichová, Jaroslav Vicar, Vladimír Krečman, Vilím Šimánek, Helena Rýdlová, Daniela Walterová, Pavel Kosina, Václav Lichnovský, Michael J. Brabec
Publikováno v:
Food and Chemical Toxicology. 42:85-91
Sanguinaria canadesis, Chelidonium majus and Macleya cordata have been used for centuries as alternative medicines. Currently the extracts from these medicinal plants are components of veterinary and human phytopreparations, and of oral-hygiene agent
Publikováno v:
Cell Biology and Toxicology. 6:63-79
Two cell types central to the support of spermatogenesis, the Sertoli cell and the interstitial (Leydig) cell, were isolated from the same cohort of young male rats and challenged with cadmium chloride to compare their susceptibility to the metal. Bo
Autor:
Michael J. Brabec, Elizabeth D. Dolci
Publikováno v:
Experimental and Molecular Pathology. 28:96-106
Administration of chloramphenicol early in Ccl, intoxication prevents lipid peroxidation of endoplasmic reticulum membranes. Conversely, a sulfamoyl analog, Tevenel, was ineffective in preventing the lipoperoxidative process. Likewise, in an in vitro
Publikováno v:
Toxicology Letters. 24:71-78
40 h after an acute dose of CCl4 (11.3 mmol/kg), the incorporation of [1-3H]ethanolamine into rat hepatic microsomal phospholipids was inhibited to 70% of control. Incorporation into phospholipids of the inner and outer mitochondrial membranes was 30
Publikováno v:
Toxicology and Applied Pharmacology. 76:56-61
Exposure to 2-methoxyethanol (ME) or its major metabolite, methoxyacetic acid (MA), results in spermatocyte depletion and testicular atrophy in experimental animals. The site of spermatogenesis is within the seminiferous tubule. Sertoli cells support
Publikováno v:
Biochemical and Biophysical Research Communications. 76:189-195
The activity of sulfite oxidase increased in intact rat hepatic mitochondria and decreased in solubilized mitochondria during poisoning by CCl 4 . During acute damage the sulfite oxidase activity of the post-mitochondrial supernate increased, while t
Autor:
Michael J. Brabec, Robert H. De Wit
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 824:256-261
Hepatic mitochondria isolated from rats 40 h after dosage with 1.1 ml/kg CCl4 are uncoupled and display structural damage. Mitochondrial function returns during hepatic recovery. Because the products of mitochondrial protein synthesis are essential t
Publikováno v:
Life Sciences. 37:1221-1224
Although anesthetized animals are awakened when subjected to increased pressure, compression does not result in antagonism of all phenomena associated with these drugs. It has recently been demonstrated that halothane's inhibition of respiration of i