Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Michael J Vanden Oever"'
Publikováno v:
Pediatric Research. 83:318-324
Epidermolysis bullosa is classified as a genodermatosis, an inherited genetic skin disorder that results in severe, chronic skin blistering with painful and life-threatening complications. Although there is currently no cure for epidermolysis bullosa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ec15c37f68f9b7a9c4e007c12ab3f87
https://doi.org/10.1038/pr.2017.244
https://doi.org/10.1038/pr.2017.244
Publikováno v:
The Journal of investigative dermatology. 136(10)
Recessive dystrophic epidermolysis bullosa (RDEB) is a complex inherited skin disorder caused by loss-of-function mutations in the COL7A1 gene. For an effective treatment of this disorder to be realized, both a thorough understanding of the regulatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a48daa29fba5d4ab452795a1995eaa33
https://pubmed.ncbi.nlm.nih.gov/27328306
https://pubmed.ncbi.nlm.nih.gov/27328306
Autor:
Michael J Vanden Oever, Jin Young Han
Publikováno v:
Journal of Virology. 84:3116-3120
Herpes simplex virus type 2 (HSV-2) induces apoptosis in T cells by a caspase-dependent mechanism. Apoptosis can occur via extrinsic (death receptor) and/or intrinsic (mitochondrial) pathways. Here, we show that the initiator caspase for the intrinsi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::780980e791c2752b117a2aca1b189033
https://doi.org/10.1128/jvi.01726-09
https://doi.org/10.1128/jvi.01726-09
Autor:
Jakub Tolar, Michael J Vanden Oever
Publikováno v:
F1000Prime Reports
Epidermolysis bullosa is a group of inherited disorders that can be both systemic and life-threatening. Standard treatments for the most severe forms of this disorder, typically limited to palliative care, are ineffective in reducing the morbidity an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::136b3d6237c4652965290b0aac43af7e
https://doi.org/10.12703/p6-35
https://doi.org/10.12703/p6-35