Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Michèle Heimburger"'
Autor:
Jean-Jacques Mercadier, Jean-Baptiste Michel, Y. Chen, Christine Choqueux, Brigitte Escoubet, Fabrice Prunier, Michèle Heimburger, Barnabas Gellen
Publikováno v:
European Journal of Heart Failure. 7:739-747
Background In most animal models of chronic hemodynamic overload of the left ventricle (LV) as well as in human end stage heart failure, the sarcoplasmic reticulum (SR) Ca2+-ATPase (SERCA2a) mRNA levels are decreased in parallel with increased atrial
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae6bdbb35e1445f99a06862798ee41e8
https://doi.org/10.1016/j.ejheart.2004.10.007
https://doi.org/10.1016/j.ejheart.2004.10.007
Autor:
Jean-Baptiste Michel, Michèle Heimburger, Jean-Jacques Mercadier, Ying Chen, Fabrice Prunier, Bruno Riou, Benoît Vivien, Warner S. Simonides, Christophe Lenoir, Wolfgang-Michael Franz, Christine Choqueux, Brigitte Escoubet, Barnabas Gellen, Julien Amour
Publikováno v:
Circulation, 109(15), 1898-1903. Lippincott Williams and Wilkins
Chen, Y, Escoubet, B, Prunier, F, Amour, J, Simonides, W S, Vivien, B, Lenoir, C, Heimburger, M, Choqueux, C, Gellen, B, Riou, B, Michel, J B, Franz, W M & Mercadier, J J 2004, ' Constitutive Cardiac Overexpression of Sarcoplasmic/Endoplasmic Reticulum Ca 2+-ATPase Delays Myocardial Failure after Myocardial Infarction in Rats at a Cost of Increased Acute Arrhythmias ', Circulation, vol. 109, no. 15, pp. 1898-1903 . https://doi.org/10.1161/01.CIR.0000124230.60028.42
Chen, Y, Escoubet, B, Prunier, F, Amour, J, Simonides, W S, Vivien, B, Lenoir, C, Heimburger, M, Choqueux, C, Gellen, B, Riou, B, Michel, J B, Franz, W M & Mercadier, J J 2004, ' Constitutive Cardiac Overexpression of Sarcoplasmic/Endoplasmic Reticulum Ca 2+-ATPase Delays Myocardial Failure after Myocardial Infarction in Rats at a Cost of Increased Acute Arrhythmias ', Circulation, vol. 109, no. 15, pp. 1898-1903 . https://doi.org/10.1161/01.CIR.0000124230.60028.42
Background— Heart failure often complicates myocardial infarction (MI), and sarcoplasmic/endoplasmic reticulum Ca 2+ -ATPase (SERCA2a) is underexpressed in the failing myocardium. We examined the effect of preexisting cardiac SERCA2a protein overex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fe34c80e8f263b8670d662a39cfcf8aa
https://doi.org/10.1161/01.cir.0000124230.60028.42
https://doi.org/10.1161/01.cir.0000124230.60028.42
Autor:
Anne-Marie Lompré, Nathalie Chossat, Michel Perricaudet, Philippe Jourdon, Michèle Heimburger, Thierry Ragot, Damien Logeart, Stéphane N. Hatem, Jean-Jacques Mercadier, Frank Griscelli
Publikováno v:
Cardiovascular Research. 49:288-297
Objective: We examined the functional consequences of expressing adult rabbit fast skeletal sarcoplasmic reticulum (SR) Ca2+-ATPase (SERCA1a) in isolated adult rat ventricular myocytes. Methods: Myocytes were infected with a recombinant adenovirus ha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b2ef969392b429e5843f13cfd9fdd4be
https://doi.org/10.1016/s0008-6363(00)00234-0
https://doi.org/10.1016/s0008-6363(00)00234-0
Autor:
Michel Aubier, John C. Reed, Maryla Krajewska, Stanislaw Krajewski, Catherine Rucker-Martin, Jean-Jacques Mercadier, Michèle Heimburger, Thierry Folliguet, Stéphane N. Hatem, Christine Aimé-Sempé
Publikováno v:
Journal of the American College of Cardiology. 34:1577-1586
OBJECTIVES The aim of the present study was to determine if myocytes can die by apoptosis in fibrillating and dilated human atria. BACKGROUND The cellular remodeling that occurs during atrial fibrillation (AF) may reflect a degree of dedifferentiatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f1d5d8147cb9039ef321517db1820d60
https://doi.org/10.1016/s0735-1097(99)00382-4
https://doi.org/10.1016/s0735-1097(99)00382-4
Autor:
Jean-Jacques Mercadier, Isabelle Espinasse, Jean-Baptiste Michel, Jean-François Giudicelli, Christine Richer, Françoise Samson, Michèle Heimburger, Marie-Claude Bourin, Nicole Defer, Jacques Hanoune, Vadim Iourgenko, Eric Pussard
Publikováno v:
Cardiovascular Research. 42:87-98
Objective: To address the effect of longstanding left ventricular (LV) hypertrophy and failure on LV adenylyl cyclase (AC) gene expression, mRNA concentrations of the main cardiac AC isoforms were measured in the non-infarcted area of LV from rats wi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5abe3ad0f581ab3e91987e981172c54e
https://doi.org/10.1016/s0008-6363(98)00283-1
https://doi.org/10.1016/s0008-6363(98)00283-1
Autor:
René Pariente, Michel Aubier, Stefano Schiaffino, C. Wisnewsky, Simonetta Ausoni, Rolana Marrash, Jean-Jacques Mercadier, Michèle Heimburger, Ketty Schwartz
Publikováno v:
Europe PubMed Central
In striated muscle, chronic increases in workload result in changes in myosin phenotype. The aim of this study was to determine whether such changes occur in the diaphragm of patients with severe chronic obstructive pulmonary disease, a situation cha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a9556ea17167f66e67927e6389d9593
https://doi.org/10.1152/ajplung.1998.274.4.l527
https://doi.org/10.1152/ajplung.1998.274.4.l527
Autor:
Damien Logeart, Renée Ventura-Clapier, Michèle Heimburger, Vlad Ciobotaru, Christophe Heymes, Brigitte Escoubet, Liliane Louedec, Pierre Bedossa, Jean-Baptiste Michel, Jean-Jacques Mercadier
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 324(1)
We investigated the effects of long-term heart rate reduction (HRR) on pressure overload-induced heart failure. Pressure overload of the left ventricle was induced in 21-day-old rats by banding the ascending aorta. HRR was induced for 3 months with i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::68b6d678ae6348a6643c981ea47c6922
https://pubmed.ncbi.nlm.nih.gov/17901295
https://pubmed.ncbi.nlm.nih.gov/17901295
Autor:
Damien Logeart, Jean-Baptiste Michel, Thierry Ragot, Jean-Jacques Mercadier, Michèle Heimburger, Brigitte Escoubet, Liliane Louedec, Laurent Vinet
Publikováno v:
AJP-Heart and Circulatory Physiology
AJP-Heart and Circulatory Physiology, American Physiological Society, 2006,-, ⟨10.1152/ajpheart.00411.2006⟩
AJP-Heart and Circulatory Physiology, American Physiological Society, 2006,-, ⟨10.1152/ajpheart.00411.2006⟩
The aim of this study was to examine the efficiency of adenovirus-mediated overexpression of sarco(endo)plasmic reticulum Ca2+-ATPase (SERCA1a) gene in a realistic model based on percutaneous intracoronary delivery and on noninvasive functional monit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c7ce9f4cda4e0f80589ff6ef011466c7
https://hal.archives-ouvertes.fr/hal-00093360
https://hal.archives-ouvertes.fr/hal-00093360
Autor:
Guy Michel Mazmanian, Nicolas Bonnet, Alain Serraf, Catherine Rucker-Martin, F. Samson, Dimitri O Levitsky, Michèle Heimburger, Jean-Jacques Mercadier
Publikováno v:
Pediatric research. 48(1)
Congenital aortic coarctation is well tolerated by the fetus because the foramen ovale and ductus arteriosus equalize intracardiac and great arteries pressures and shunts. The pathologic consequences only emerge after birth with closure of the forame
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2da626d74aad8d9b227b074500e2b89
https://pubmed.ncbi.nlm.nih.gov/10879799
https://pubmed.ncbi.nlm.nih.gov/10879799
Autor:
Damien Logeart, Michel Perricaudet, Nathalie Chossat, Jean-Jacques Mercadier, Nathalie Nevo, Catherine Rucker-Martin, Hedi Haddada, Michèle Heimburger, Stéphane N. Hatem
Publikováno v:
Human gene therapy. 11(7)
Efficient and homogeneous gene transfer to cardiac myocytes is a major target in myocardial gene therapy. The aim of this study was to determine the conditions permitting efficient, homogeneous, adenovirus-mediated gene transfer to cardiac myocytes,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dd3c1a4085169c2672d26b416e5c1105
https://pubmed.ncbi.nlm.nih.gov/10811230
https://pubmed.ncbi.nlm.nih.gov/10811230