Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Micaela Johanna Glat"'
Publikováno v:
PLoS ONE, Vol 11, Iss 2, p e0148170 (2016)
Multiple System Atrophy (MSA) is a sporadic neurodegenerative disorder characterized by parkinsonism, cerebellar ataxia and dysautonomia, in various combinations. In MSA with parkinsonism (MSA-P), the degeneration is mainly restricted to the substant
Externí odkaz:
https://doaj.org/article/8a0f5c9edd95464e9f3306663e6f84ba
Autor:
Martin Häring, Ana Cicvaric, Sonali N. Reisinger, Spyros Sideromenos, Francisco J. Monje, Orsolya Horvath, Martin Bilban, Daniela D. Pollak, Micaela Johanna Glat, Sophia Derdak
Publikováno v:
Molecular psychiatry
The signal transducer and activator of transcription 3 (STAT3) signalling pathway is activated through phosphorylation by Janus kinases in response to a diverse set of immunogenic and non-immunogenic triggers. Several distinct lines of evidence propo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f6eac6430de9e1b5d9826d91e642225
http://europepmc.org/articles/PMC8505245
http://europepmc.org/articles/PMC8505245
Publikováno v:
Journal of neural transmission (Vienna, Austria : 1996). 127(2)
Multiple system atrophy (MSA) is a sporadic neurodegenerative disorder characterized by striatonigral degeneration and olivopontocerebellar atrophy. The main hallmark of MSA is the aggregation of alpha-synuclein in oligodendrocytes, which contributes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e7aa12545b2a582ef157a865d406708f
https://pubmed.ncbi.nlm.nih.gov/32065333
https://pubmed.ncbi.nlm.nih.gov/32065333
Autor:
Anat Boehm-Cagan, Daniel M. Michaelson, Ori Liraz, Daniel Offen, Shiran Salomon-Zimri, Yael Barhum, Tali Ben-Zur, Micaela Johanna Glat, Ishai Luz
Publikováno v:
Journal of Alzheimer's Disease. 53:1443-1458
Apolipoprotein E4 (ApoE4), the most prevalent genetic risk factor for Alzheimer's disease (AD), is associated with increased neurodegeneration and vascular impairments. Vascular endothelial growth factor (VEGF), originally described as a key angiogen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cff38fcde71f1960136c268f52ccf670
https://doi.org/10.3233/jad-160182
https://doi.org/10.3233/jad-160182
Publikováno v:
Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia. 26
Glial fibrillary acidic protein (GFAP) has been shown to be increased in the cerebrospinal fluid (CSF) of patients suffering from neurological diseases involving the activation of astrocytes, but has not been studied in amyotrophic lateral sclerosis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::beddb00dc11bcf30a1ec555f42c69885
https://pubmed.ncbi.nlm.nih.gov/26602604
https://pubmed.ncbi.nlm.nih.gov/26602604
Autor:
Tali Ben-Zur, Israel Steiner, Micaela Johanna Glat, Felix Benninger, Yael Barhum, David Yaffe, Elena Kogan, Daniel Offen
Publikováno v:
Journal of molecular neuroscience : MN. 58(1)
Sciatic nerve damage is a common medical problem. The main causes include direct trauma, prolonged external nerve compression, and pressure from disk herniation. Possible complications include leg numbness and the loss of motor control. In mild cases
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ed653920be1732243f83e975c4712e15
https://pubmed.ncbi.nlm.nih.gov/26385386
https://pubmed.ncbi.nlm.nih.gov/26385386
Publikováno v:
Journal of Nanobiotechnology
Background Tau dysfunction is believed to be the primary cause of neurodegenerative disorders referred to as tauopathies, including Alzheimer’s disease, Pick’s disease, frontotemporal dementia and Parkinsonism. The role of microglial cells in the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5a863a080d87c50e20cafb0ea981b954
https://doi.org/10.1186/1477-3155-11-32
https://doi.org/10.1186/1477-3155-11-32
Autor:
Daniel Offen, Micaela Johanna Glat
Publikováno v:
Stem cells and development. 22(10)
Alzheimer's disease (AD) is a progressive neurological disorder characterized by the aggregation of two proteins, amyloid-β and hyperphosphorylated tau, and by neuronal and synaptic loss. Although some drugs have been shown to slow the progression o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aa5eccae5af2580f3c43e40bde5bb55a
https://pubmed.ncbi.nlm.nih.gov/23320452
https://pubmed.ncbi.nlm.nih.gov/23320452