Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Miaoqian Shi"'
Publikováno v:
Biochemical and biophysical research communications. 632
Myocardial infarction is still a leading cause of morbidity and mortality worldwide, but its pathogenesis has not been fully understood. In the study, we attempted to explore the effects of E3 ligase tripartite motif 16 (TRIM16) on myocardial ischemi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::709de058dd8ab0fc6a8aaf4bcb2e816b
https://pubmed.ncbi.nlm.nih.gov/36208489
https://pubmed.ncbi.nlm.nih.gov/36208489
Publikováno v:
Aging (Albany NY)
Aberrant proliferation and migration of vascular smooth muscle cells contributes to cardiovascular diseases (CVDs), including atherosclerosis. MicroRNA-223 (miR-223) protects against atherosclerotic CVDs. We investigated the contribution of miR-223 t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::88cc4dd6c494a94497e75871c9f1dc1d
http://europepmc.org/articles/PMC7803580
http://europepmc.org/articles/PMC7803580
Publikováno v:
International Journal of Biological Sciences, Vol 8, Iss 2, Pp 236-248 (2012)
Objectives: Hypertrophy has been shown to be associated with arrhythmias which can be caused by abnormal remodeling of the Kv4-family of transient potassium channels. Inhibitors of 3-hydroxy-3-methylglutaryl coenzyme A reductase (statins) have recent
Externí odkaz:
https://doaj.org/article/471ff232454a4557a0902a9e18707b74
Publikováno v:
Molecular Medicine Reports
High‑mobility group box 1 (HMGB1) is released by necrotic cells and serves an important role in cardiovascular pathology. However, the effects of HMGB1 in cardiomyocyte hypertrophy remain unclear. Therefore, the aim of the present study was to inve
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70b884f945e7b10fa0b9d1f162aa9c76
https://doi.org/10.3892/mmr.2021.11853
https://doi.org/10.3892/mmr.2021.11853
Autor:
Jian Zhang, Dongwei Zhang, Zheng Qiangsun, Fei-Fei Su, Wei Zhang, Xue Li, Haichang Wang, Miaoqian Shi
Publikováno v:
International Journal of Medical Sciences
Cardiac hypertrophy is an independent predictor of cardiovascular morbidity and mortality. To identify the mechanisms by which simvastatin inhibits cardiac hypertrophy induced by pressure overload, we determined effects of simvastatin on 14-3-3 prote
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4acd889668b77fc6e6c95aee7dd1b796
https://doi.org/10.7150/ijms.28106
https://doi.org/10.7150/ijms.28106
Autor:
Miaoqian Shi, Xiong-Tao Liu, Wangang Guo, Qiang-sun Zheng, Hongtao Wang, Fei-Fei Su, Zifan Lu
Publikováno v:
Mediators of Inflammation
Mediators of Inflammation, Vol 2012 (2012)
Mediators of Inflammation, Vol 2012 (2012)
Cardiac hypertrophy is an independent predictor of cardiovascular morbidity and mortality. In recent years, evidences suggest that high-mobility group box 1 (HMGB1) protein, an inflammatory cytokine, participates in cardiac remodeling; however, the i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6debfd568f0454ebe564e7803fade779
https://doi.org/10.1155/2012/805149
https://doi.org/10.1155/2012/805149
Publikováno v:
International Journal of Biological Sciences
Objectives: Hypertrophy has been shown to be associated with arrhythmias which can be caused by abnormal remodeling of the Kv4-family of transient potassium channels. Inhibitors of 3-hydroxy-3-methylglutaryl coenzyme A reductase (statins) have recent
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70c4cb8008f10ad6c8a2ced4affbdc49
http://europepmc.org/articles/PMC3258563
http://europepmc.org/articles/PMC3258563