Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Miaodan Li"'
Autor:
Miaodan Li, Shumin Chen, Xue Shi, Chenfei Lyu, Yongfang Zhang, Miaoqin Tan, Chen Wang, Nailiang Zang, Xiaoxi Liu, Yafang Hu, Jiangang Shen, Liang Zhou, Yong Gu
Publikováno v:
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-13 (2018)
Abstract Background Blood–brain barrier (BBB) breakdown and inflammatory responses are the major causes of tissue-type plasminogen activator (tPA)-induced hemorrhagic transformation (HT), while high-mobility group box 1 (HMGB1) exacerbates inflamma
Externí odkaz:
https://doaj.org/article/3e4c2b5d0fd94547852454fc3423843d
Autor:
Chenfei Lyu, Yongfang Zhang, Minhua Gu, Yusheng Huang, Guanghui Liu, Chen Wang, Miaodan Li, Shumin Chen, Suyue Pan, Yong Gu
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 12 (2018)
Background: Innate immune response to neuronal death is one of the key events of the pathogenesis of ischemic brain injury. Interleukin-1 receptor-associated kinase (IRAK)-M, encoded by gene Irak3, negatively regulates toll-like receptor signaling by
Externí odkaz:
https://doaj.org/article/59aff9130bc748a3a7dde9eb54982922
Publikováno v:
Experimental and Therapeutic Medicine
Electroacupuncture (EA) may stimulate neurogenesis in animal models of ischemic stroke; however, the associated mechanisms are not clear. The present study aimed to evaluate the neurogenesis efficacy of EA on ischemic stroke and the underlying associ
Publikováno v:
Frontiers in Plant Science, Vol 15 (2024)
In the process of applying exotic plants to wetland ecological restoration, insufficiently evaluated alien species may exhibit strong competitiveness and fecundity. Once introduced, they can displace native flora, disrupt the original ecological bala
Externí odkaz:
https://doaj.org/article/69fbdbaa9d194793a9ac0598cf88d27c
Autor:
Liang Zhou, Miaoqin Tan, Xiao-Xi Liu, Nailiang Zang, Yongfang Zhang, Chenfei Lyu, Yafang Hu, Chen Wang, Yong Gu, Xue Shi, Jiangang Shen, Miaodan Li, Shumin Chen
Publikováno v:
Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-13 (2018)
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-13 (2018)
Background Blood–brain barrier (BBB) breakdown and inflammatory responses are the major causes of tissue-type plasminogen activator (tPA)-induced hemorrhagic transformation (HT), while high-mobility group box 1 (HMGB1) exacerbates inflammatory dama
Autor:
Miaodan Li, Shumin Chen, Shi, Xue, Chenfei Lyu, Yongfang Zhang, Miaoqin Tan, Wang, Chen, Nailiang Zang, Xiaoxi Liu, Yafang Hu, Jiangang Shen, Zhou, Liang, Gu, Yong
Figure S1. Western blot analysis indicated serum HMGB1 levels were elevated after thrombolysis in stroke patients and rats. Sera from human and rats received thrombolysis were incubated with Protein A/G MagBeads (GenScript, Piscataway, NJ) to remove
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::05b03f636fa529bdf898435177a49b0f
Publikováno v:
Neuroscience. 360
Excessive inflammatory response produced after cardiac arrest and cardiopulmonary resuscitation (CA/CPR) is one of major causes of cerebral injury. High mobility group box 1 (HMGB1) is a pro-inflammatory cytokine and its role in brain injury after CA