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pro vyhledávání: '"Meyer, Ione"'
Autor:
Surana, Sunaina, Tosolini, Andrew P., Meyer, Ione F.G., Fellows, Alexander D., Novoselov, Sergey S., Schiavo, Giampietro
Publikováno v:
In Toxicon 1 June 2018 147:58-67
Autor:
Luisier, Raphaelle, Tyzack, Giulia E., Hall, Claire E., Mitchell, Jamie S., Devine, Helen, Taha, Doaa M., Malik, Bilal, Meyer, Ione, Greensmith, Linda, Newcombe, Jia, Ule, Jernej, Luscombe, Nicholas M., Patani, Rickie
Publikováno v:
Nature Communications, Vol 9, Iss 1, Pp 1-15 (2018)
Nature Communications
Nature Communications
Mutations causing amyotrophic lateral sclerosis (ALS) strongly implicate ubiquitously expressed regulators of RNA processing. To understand the molecular impact of ALS-causing mutations on neuronal development and disease, we analysed transcriptomes
Akademický článek
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Autor:
Tyzack, Giulia E., Hall, Claire E., Sibley, Christopher R., Cymes, Tomasz, Forostyak, Serhiy, Carlino, Giulia, Meyer, Ione F., Schiavo, Giampietro, Zhang, Su-Chun, Gibbons, George M., Newcombe, Jia, Patani, Rickie, Lakatos, András
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-17 (2017)
Nature Communications
Tyzack, G E, Hall, C E, Sibley, C R, Cymes, T, Forostyak, S, Carlino, G, Meyer, I F, Schiavo, G, Zhang, S-C, Gibbons, G M, Newcombe, J, Patani, R & Lakatos, A 2017, ' A neuroprotective astrocyte state is induced by neuronal signal EphB1 but fails in ALS models ', Nature Communications, vol. 8, no. 1, 1164 . https://doi.org/10.1038/s41467-017-01283-z
Nature Communications
Tyzack, G E, Hall, C E, Sibley, C R, Cymes, T, Forostyak, S, Carlino, G, Meyer, I F, Schiavo, G, Zhang, S-C, Gibbons, G M, Newcombe, J, Patani, R & Lakatos, A 2017, ' A neuroprotective astrocyte state is induced by neuronal signal EphB1 but fails in ALS models ', Nature Communications, vol. 8, no. 1, 1164 . https://doi.org/10.1038/s41467-017-01283-z
Astrocyte responses to neuronal injury may be beneficial or detrimental to neuronal recovery, but the mechanisms that determine these different responses are poorly understood. Here we show that ephrin type-B receptor 1 (EphB1) is upregulated in inju
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0791e6d4f0b64270b621eb863174cb0b
http://hdl.handle.net/10044/1/54622
http://hdl.handle.net/10044/1/54622
Autor:
Schiavo, Giampietro, Meyer, Ione, Novoselov, Sergey, Surana, Sunaina, Tiknaz, Deniz, Tosolini, Andrew
Publikováno v:
In Toxicon 25 December 2016 123 Supplement:S75-S75
Autor:
Tyzack, Giulia E, Raphaelle Luisier, Doaa M Taha, Neeves, Jacob, Modic, Miha, Mitchell, Jamie S, Meyer, Ione, Greensmith, Linda, Newcombe, Jia, Ule, Jernej, Luscombe, Nicholas M, Patani, Rickie
Mutations causing amyotrophic lateral sclerosis (ALS) clearly implicate ubiquitously expressed and predominantly nuclear RNA binding proteins, which form pathological cytoplasmic inclusions in this context. However, the possibility that wild-type RNA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8a419600eb9196aae6b6cea86632c76a
Autor:
Tyzack GE; John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, E.D. Adrian Building, Forvie Site, Robinson Way, Cambridge, CB2 0PY, UK.; Department of Molecular Neuroscience, UCL Institute of Neurology, University College London, London, WC1N 3BG, UK., Hall CE; Department of Molecular Neuroscience, UCL Institute of Neurology, University College London, London, WC1N 3BG, UK., Sibley CR; Division of Brain Sciences, Imperial College London, Burlington Danes Building Du Cane Road, London, W12 0NN, UK., Cymes T; John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, E.D. Adrian Building, Forvie Site, Robinson Way, Cambridge, CB2 0PY, UK., Forostyak S; Institute of Experimental Medicine ASCR and Charles University in Prague, Department of Neuroscience, Videnská 1083, Prague 4, 142 20, Czech Republic., Carlino G; Department of Molecular Neuroscience, UCL Institute of Neurology, University College London, London, WC1N 3BG, UK., Meyer IF; Sobell Department of Motor Neuroscience & Movement Disorders, UCL Institute of Neurology, University College London, London, WC1N 3BG, UK., Schiavo G; Sobell Department of Motor Neuroscience & Movement Disorders, UCL Institute of Neurology, University College London, London, WC1N 3BG, UK.; UK Dementia Research Institute at UCL, UCL Institute of Neurology, University College London, London, WC1N 3BG, UK., Zhang SC; Waisman Center, University of Wisconsin, 1500 Highland Avenue, Madison, WI, 53705, USA., Gibbons GM; John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, E.D. Adrian Building, Forvie Site, Robinson Way, Cambridge, CB2 0PY, UK., Newcombe J; Department of Neuroinflammation, UCL Institute of Neurology, University College London, London, WC1N 1PJ, UK., Patani R; Department of Molecular Neuroscience, UCL Institute of Neurology, University College London, London, WC1N 3BG, UK. rickie.patani@ucl.ac.uk.; The Francis Crick Institute, 1 Midland Road, London, NW1 1AT, UK. rickie.patani@ucl.ac.uk., Lakatos A; John van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, E.D. Adrian Building, Forvie Site, Robinson Way, Cambridge, CB2 0PY, UK. AL291@cam.ac.uk.; Addenbrooke's Hospital, Cambridge University Hospitals, Hills Road, Cambridge, CB2 0QQ, UK. AL291@cam.ac.uk.
Publikováno v:
Nature communications [Nat Commun] 2017 Oct 27; Vol. 8 (1), pp. 1164. Date of Electronic Publication: 2017 Oct 27.