Výsledky vyhledávání - "Melissa A. Riddle"

Role of Phosphoinositide 3-OH Kinase p110β in Skeletal Myogenesis

Autor: Luis A. Leandry, Jean J. Zhao, David R. Piper, Ronald W. Matheny, Christine M. Lynch, Melissa A. Riddle-Kottke, Alyssa Geddis, Mary N. Abdalla

Publikováno v: Molecular and Cellular Biology. 35:1182-1196

Phosphoinositide 3-OH kinase (PI3K) regulates a number of developmental and physiologic processes in skeletal muscle; however, the contributions of individual PI3K p110 catalytic subunits to these processes are not well-defined. To address this quest

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::acf857f9a3a7496bb9cb290369d0169d
https://doi.org/10.1128/mcb.00550-14

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Hydrogen sulfide dilates rat mesenteric arteries by activating endothelial large-conductance Ca2+-activated K+ channels and smooth muscle Ca2+ sparks

Autor: Melissa A. Riddle, Jessica M. Osmond, Nancy L. Kanagy, Jay S. Naik, Benjimen R. Walker, Laura V. Gonzalez Bosc, Olan Jackson-Weaver

Publikováno v: American Journal of Physiology-Heart and Circulatory Physiology. 304:H1446-H1454

We have previously shown that hydrogen sulfide (H2S) reduces myogenic tone and causes relaxation of phenylephrine (PE)-constricted mesenteric arteries. This effect of H2S to cause vasodilation and vascular smooth muscle cell (VSMC) hyperpolarization

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::218086c914705987aa1320f8c8c64934
https://doi.org/10.1152/ajpheart.00506.2012

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Role of caveolin-1 in endothelial BKCachannel regulation of vasoreactivity

Autor: Benjimen R. Walker, Melissa A. Riddle, Jennifer M Hughes

Publikováno v: American Journal of Physiology-Cell Physiology. 301:C1404-C1414

A novel vasodilatory influence of endothelial cell (EC) large-conductance Ca2+-activated K+(BKCa) channels is present following in vivo exposure to chronic hypoxia (CH) and may exist in other pathological states. However, the mechanism of channel act

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::978b4de938fab715146b6278905013b2
https://doi.org/10.1152/ajpcell.00013.2011

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Altered membrane lipid domains limit pulmonary endothelial calcium entry following chronic hypoxia

Autor: Jay S. Naik, Thomas C. Resta, Melissa A. Riddle, Michael L. Paffett, Steven D. Menicucci, Antonio J. Gonzales, Benjimen R. Walker

Publikováno v: American Journal of Physiology-Heart and Circulatory Physiology. 301:H1331-H1340

Agonist-induced Ca2+entry into the pulmonary endothelium depends on activation of both store-operated Ca2+(SOC) entry and receptor-operated Ca2+(ROC) entry. We previously reported that pulmonary endothelial cell SOC entry and ROC entry are reduced in

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6d67463b8e7c741c82afc26e56b126fa
https://doi.org/10.1152/ajpheart.00980.2010

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Altered Protein Kinase C Regulation of Pulmonary Endothelial Store- and Receptor-Operated Ca2+Entry after Chronic Hypoxia

Autor: Michael L. Paffett, Benjimen R. Walker, Nancy L. Kanagy, Thomas C. Resta, Melissa A. Riddle

Publikováno v: Journal of Pharmacology and Experimental Therapeutics. 334:753-760

Chronic hypoxia (CH)-induced pulmonary hypertension is associated with decreased basal pulmonary artery endothelial cell (EC) Ca2+, which correlates with reduced store-operated Ca2+ (SOC) entry. Protein kinase C (PKC) attenuates SOC entry in ECs. The

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6d22410f36ae3dc75848f37d1af2f3a5
https://doi.org/10.1124/jpet.110.165563

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