Zobrazeno 1 - 10
of 80
pro vyhledávání: '"Melinda M. Dean"'
Autor:
Michelle Roets, David J. Sturgess, Ching-Yu Chen, Jaisil E. J. Punnasseril, Lee Jones, Andre A. van Zundert, Melinda M. Dean
Publikováno v:
Frontiers in Anesthesiology, Vol 3 (2024)
Background and objectivesTransfusion-related immune modulation (TRIM) and associated adverse outcomes during major surgery are increasingly important to patients and health services internationally. A panel of pre-operative blood tests is an essentia
Externí odkaz:
https://doaj.org/article/6897ea0113144076a6baf3627062368d
Autor:
Flavia Torres Vasconcelos, Helen M. Faddy, Katharina M.D. Merollini, Robert L.P. Flower, Melinda M. Dean, Elvina Viennet
Publikováno v:
Health Sciences Review, Vol 7, Iss , Pp 100087- (2023)
Background: Natural disasters and pandemics can be highly challenging to blood supply chains. This review aimed to assess the impacts of pandemics and natural disasters on blood donation globally, appraise any similarities and differences, and provid
Externí odkaz:
https://doaj.org/article/1e6e4e78c22c4cd49196b8ebf794ee4d
Publikováno v:
Platelets, Vol 29, Iss 4, Pp 373-382 (2018)
Platelet transfusion has been reported to modulate the recipients’ immune system. To date, the precise mechanism(s) driving poor patient outcomes (e.g., increased rate of mortality, morbidity, infectious complications and prolonged hospital stays)
Externí odkaz:
https://doaj.org/article/5acaf62d1acf4c99bef2b78e5d80ea9c
Autor:
Katrina Kildey, Robert L. Flower, Thu V. Tran, Robert Tunningley, Jonathan Harris, Melinda M. Dean
Publikováno v:
Computational and Structural Biotechnology Journal, Vol 6, Iss 7 (2013)
Murine models with modified gene function as a result of N-ethyl-N-nitrosourea (ENU) mutagenesis have been used to study phenotypes resulting from genetic change. This study investigated genetic factors associated with red blood cell (RBC) physiology
Externí odkaz:
https://doaj.org/article/ef17b406fae24c9e960242920a22df7b
Publikováno v:
Pathology. 55:S98-S99
Autor:
Sara, Chiaretti, Mark, Burton, Penny, Hassel, Filip, Radenkovic, Nilam, Devikashri, Annette J, Sultana, Fergal T, Temple, Melinda M, Dean, John-Paul, Tung
Publikováno v:
Blood Transfus
BACKGROUND: Antibodies against human neutrophil antigen (HNA)-3a are associated with severe cases of transfusion-related acute lung injury (TRALI). The HNA-3 system is located on choline transporter-like 2 (CTL-2) protein. CTL-2 is encoded by the gen
Autor:
John F. Fraser, Rishendran Naidoo, Fenny Chong, Elise K. Hewlett, John-Paul Tung, Arlanna Esguerra-Lallen, Marc Ziegenfuss, Robert L. Flower, Alexis J. Perros, Peter Tesar, Susan Smith, Melinda M. Dean, S. Engkilde-Pedersen, Donalee O’Brien, Helen M. Faddy, Kelly M Rooks
Publikováno v:
Journal of Cellular and Molecular Medicine
Coronary artery bypass grafting (CABG) triggers a systemic inflammatory response that may contribute to adverse outcomes. Dendritic cells (DC) and monocytes are immunoregulatory cells potentially affected by CABG, contributing to an altered immune st
Autor:
John-Paul Tung, Sara Chiaretti, Melinda M. Dean, Annette J. Sultana, Michael C. Reade, Yoke Lin Fung
Publikováno v:
Blood reviews. 53
Transfusion-related acute lung injury (TRALI) can occur during or after a transfusion, and remains a leading cause of transfusion-associated morbidity and mortality. TRALI is caused by the transfusion of either anti-leukocyte antibodies or biological
Autor:
Mark Gillett, Jeremy Hsu, Peter K.M. Maitz, Melinda M. Dean, Natalie Lott, Zsolt J. Balogh, Anthony Joseph, Susan Taggart, David O. Irving, Rena Hirani, Lesley Survela, Julie Seggie
Publikováno v:
Molecular Immunology. 103:229-234
Background The potential for the co-existence of genetically disparate cells (microchimerism) and associated cytokine profiles following red blood cell (RBC) transfusion in trauma patients has not been well characterized to date. This study investiga
Autor:
Melinda M. Dean, Assia Moussa, Nicole S. Fraser, Terry Walsh, Catherine A. Hyland, Andrew C. Perkins, Christine Knauth, Robert L. Flower, Glenda M. Millard, Elizna M. Schoeman
Publikováno v:
Transfusion. 58:2414-2420
KLF1 is an essential transcriptional activator that drives erythropoiesis. KLF1 variants can result in the Inhibitor of Lutheran, or In(Lu), phenotype where red blood cells (RBCs) have reduced BCAM (LU) and CD44 (IN). Other RBC surface molecules also