Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Meagan A. Jenkins"'
Autor:
Arun Singh, Meagan A. Jenkins, Kenneth J. Burke, Jr., Goichi Beck, Andrew Jenkins, Annalisa Scimemi, Stephen F. Traynelis, Stella M. Papa
Publikováno v:
Cell Reports, Vol 22, Iss 4, Pp 941-952 (2018)
Dopamine (DA) loss in Parkinson’s disease (PD) alters the function of striatal projection neurons (SPNs) and causes motor deficits, but DA replacement can induce further abnormalities. A key pathological change in animal models and patients is SPN
Externí odkaz:
https://doaj.org/article/aa0dd9bfbdb64e38a7d24fafa33c0931
Autor:
Meagan M. Jenkins, Donna Phan Tran, Evelyn A. Flores, Deborah Kupferwasser, Harry Pickering, Ying Zheng, David W. Gjertson, Ted M. Ross, Joanna M. Schaenman, Loren G. Miller, Michael R. Yeaman, Elaine F. Reed
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
IntroductionSARS-CoV-2 is the etiologic agent of coronavirus disease 2019 (COVID-19). Questions remain regarding correlates of risk and immune protection against COVID-19.MethodsWe prospectively enrolled 200 participants with a high risk of SARS-CoV-
Externí odkaz:
https://doaj.org/article/273b14870a78446f823e91edaa627091
Publikováno v:
Infectious Diseases (London, England)
Infectious Diseases
Infectious Diseases
SARS-CoV-2 was identified as the causative pathogen in an outbreak of viral pneumonia cases originating in Wuhan, China, with an ensuing rapid global spread that led it to be declared a pandemic by the WHO on March 11, 2020. Given the threat to publi
Publikováno v:
Methods in Molecular Biology ISBN: 9781071617359
Within the last several years, great strides have been made in understanding the molecular and cellular mechanisms that control the generation of T follicular helper (TFH), T regulatory (TREG), and T follicular regulatory (TFR) cells. As a result, it
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c97e74559f932cc885f85f9ce2f7d213
https://doi.org/10.1007/978-1-0716-1736-6_16
https://doi.org/10.1007/978-1-0716-1736-6_16
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 2380
Within the last several years, great strides have been made in understanding the molecular and cellular mechanisms that control the generation of T follicular helper (T
Autor:
Alexander F. Rosenberg, Beatriz León, André Ballesteros-Tato, Meagan M. Jenkins, Holly Bachus, Davide Botta, Michael D. Schultz
Publikováno v:
Science Immunology. 6
CD8+ T cell responses to pulmonary challenges are primed by lung migratory dendritic cells (mDCs), which capture antigens in the lungs and migrate to the lung-draining mediastinal lymph node (med-L...
Autor:
Meagan M, Jenkins, Holly, Bachus, Davide, Botta, Michael D, Schultz, Alexander F, Rosenberg, Beatriz, León, André, Ballesteros-Tato
Publikováno v:
Sci Immunol
CD8(+) T cell responses to pulmonary challenges are primed by lung migratory dendritic cells (mDCs), which capture antigens in the lungs and migrate to the lung-draining mediastinal lymph node (med-LN) to activate T cells. Notably, the lungs and the
Autor:
Sahil Talwar, Paul S. García, Andrew Jenkins, Amanda A. H. Freeman, Prabhjyot Saini, Donald L. Bliwise, Meagan A. Jenkins, Lynn Marie Trotti, Neil Feldman, David B. Rye, Olivia A. Moody, Brad Cherson, Joseph W. Lynch, Eric M. Hernandez
Publikováno v:
Annals of Neurology. 81:904-907
Autor:
Meagan M Jenkins, Holly Bachus, Davide Botta, Michael Schultz, Alexander Rosenberg, Beatriz León, André Ballesteros-Tato
Publikováno v:
The Journal of Immunology. 206:103.06-103.06
CD8+ T cell responses to influenza virus infection are canonically known to be activated solely in the lung draining, mediastinal lymph node (mLN). Mechanistically, migratory dendritic cells (mDCs) are activated in the lungs and traffic to the mLN wh
Publikováno v:
The Journal of Immunology. 204:93.6-93.6
Necroptosis, a programmed form of lytic cell death, is initiated by various viral and bacterial pathogens as result of irreparable ion dysregulation and energy depletion. This cellular damage results in the activation of the kinases RIPK1 and RIPK3,