Zobrazeno 1 - 10 of 79 pro vyhledávání: '"McAleer, MA"'
1
Sphingolipids in atopic dermatitis
Autor: Jakasa I, Jurakic Toncic R, McAleer MA, Irvine AD, Kezic S
Dysfunctional skin barrier plays a key role in the pathophysiology of atopic dermatitis (AD), a common inflammatory skin disease. Altered composition of ceramides is regarded as a major cause of skin barrier dysfunction, however it is not clear wheth
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=57a035e5b1ae::a2e39cf07fe8cdf1636c00d24b54d8c5
https://www.bib.irb.hr/1233033
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2
Systemic and stratum corneum biomarkers of severity in infant atopic dermatitis include markers of innate and T helper cell-related immunity and angiogenesis
Autor: McAleer, MA, Jakasa, I, Hurault, G, Sarvari, P, McLean, WHI, Tanaka, RJ, Kezic, S, Irvine, AD
Background Biomarkers of atopic dermatitis (AD) are largely lacking, especially in infant AD. Those that have been examined to date have focused mostly on serum cytokines, with few on noninvasive biomarkers in the skin. Objectives We aimed to explore
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=57a035e5b1ae::2bacd234f2496b35fd1132a8fbd0a986
https://www.bib.irb.hr/987685
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3
The widespread use of topical antimicrobials enriches for resistance in Staphylococcus aureus isolated from patients with atopic dermatitis
Autor: Harkins, CP, McAleer, MA, Bennett, D, McHugh, M, Fleury, OM, Pettigrew, KA, Oravcová, K, Parkhill, J, Proby, CM, Dawe, RS, Geoghegan, JA, Irvine, AD, Holden, MTG
BACKGROUND: Carriage rates of Staphylococcus aureus on affected skin in atopic dermatitis (AD) are approximately 70%. Increasing disease severity during flares and overall disease severity correlate with increased burden of S. aureus. Treatment in AD
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f68875b0fea49c6547b2d3451462445
https://www.repository.cam.ac.uk/handle/1810/296028
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4
Systemic and stratum corneum biomarkers of severity in infant AD include markers of innate and Th-related immunity and angiogenesis
Autor: McAleer, MA, Jakasa, I, Hurault, G, Sarvari, P, McLean, WHI, Tanaka, RJ, Kezic, S, Irvine, AD
BACKGROUND: Biomarkers of atopic dermatitis (AD) are largely lacking, especially in infant AD. Those that have been examined to date have focused mostly on serum cytokines with few on non-invasive biomarkers in the skin. OBJECTIVES: We aimed to explo
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______1032::3ca47b0b4b2547819f4f325d41a70359
http://hdl.handle.net/10044/1/62001
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7
Multi-ancestry genome-wide association study of 21,000 cases and 95,000 controls identifies new risk loci for atopic dermatitis
Autor: Paternoster, L, Standl, M, Waage, J, Baurecht, H, Hotze, M, Strachan, DP, Curtin, JA, Bonnelykke, K, Tian, C, Takahashi, A, Esparza-Gordillo, J, Alves, AC, Thyssen, JP, den Dekker, HT, Ferreira, MA, Altmaier, E, Sleiman, PMA, Xiao, FL, Gonzalez, JR, Marenholz, I, Kalb, B, Pino-Yanes, M, Xu, C-J, Carstensen, L, Groen-Blokhuis, MM, Venturini, C, Pennell, CE, Barton, SJ, Levin, AM, Curjuric, I, Bustamante, M, Kreiner-Moller, E, Lockett, GA, Bacelis, J, Bunyavanich, S, Myers, RA, Matanovic, A, Kumar, A, Tung, JY, Hirota, T, Kubo, M, McArdle, WL, Henderson, AJ, Kemp, JP, Zheng, J, Smith, GD, Rueschendorf, F, Bauerfeind, A, Lee-Kirsch, MA, Arnold, A, Homuth, G, Schmidt, CO, Mangold, E, Cichon, S, Keil, T, Rodriguez, E, Peters, A, Franke, A, Lieb, W, Novak, N, Foelster-Holst, R, Horikoshi, M, Pekkanen, J, Sebert, S, Husemoen, LL, Grarup, N, De Jongste, JC, Rivadeneira, F, Hofman, A, Jaddoe, VWV, Pasmans, SGMA, Elbert, NJ, Uitterlinden, AG, Marks, GB, Thompson, PJ, Matheson, MC, Robertson, CF, Ried, JS, Li, J, Zuo, XB, Zheng, XD, Yin, XY, Sun, LD, McAleer, MA, O'Regan, GM, Fahy, CMR, Campbell, LE, Macek, M, Kurek, M, Hu, D, Eng, C, Postma, DS, Feenstra, B, Geller, F, Hottenga, JJ, Middeldorp, CM, Hysi, P, Bataille, V, Spector, T, Tiesler, CMT, Thiering, E, Pahukasahasram, B, Yang, JJ, Imboden, M, Huntsman, S, Vilor-Tejedor, N, Relton, CL, Myhre, R, Nystad, W, Custovic, A, Weiss, ST, Meyers, DA, Soederhaell, C, Melen, E, Ober, C, Raby, BA, Simpson, A, Jacobsson, B, Holloway, JW, Bisgaard, H, Sunyer, J, Probst-Hensch, NM, Williams, LK, Godfrey, KM, Wang, CA, Boomsma, DI, Melbye, M, Koppelman, GH, Jarvis, D, McLean, WHI, Irvine, AD, Zhang, XJ, Hakonarson, H, Gieger-, C, Burchard, EG, Martin, NG, Duijts, L, Linneberg, A, Jarvelin, M-R, Noethen, MM, Lau, S, Huebner, N, Lee, Y-A, Tamari, M, Hinds, DA, Glass, D, Brown, SJ, Heinrich, J, Evans, DM, Weidinger, S, AAGC, AAGC, Epidemio, EGL
Publikováno v: Nature Genetics
Genetic association studies have identified 21 loci associated with atopic dermatitis risk predominantly in populations of European ancestry. To identify further susceptibility loci for this common, complex skin disease, we performed a meta-analysis
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=core_ac_uk__::fe015907bf2695c75e6d9e570aa8583c
https://openaccess.sgul.ac.uk/id/eprint/107705/7/NG-LE40675R1_Paternoster_200815_with_fig.pdf
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8
Insight into IKBKG/NEMO locus: report of new mutations and complex genomic rearrangements leading to Incontinentia pigmenti disease
Autor: Conte MI, Pescatore A, Paciolla M, Esposito E, Miano MG, Lioi MB, McAleer MA, Giardino G, Pignata C, Irvine AD, Scheuerle AE, Royer G, Hadj-Rabia S, Bodemer C, Bonnefont J-P, Munnich A, Smahi A, Steffann J, Fusco F, Ursini MV
Publikováno v: Human mutation 35 (2014): 165–177. doi:10.1002/humu.22483.
info:cnr-pdr/source/autori:Conte MI, Pescatore A, Paciolla M, Esposito E, Miano MG, Lioi MB, McAleer MA, Giardino G, Pignata C, Irvine AD, Scheuerle AE, Royer G, Hadj-Rabia S, Bodemer C, Bonnefont J-P, Munnich A, Smahi A, Steffann J, Fusco F and Ursini MV/titolo:Insight into IKBKG%2FNEMO locus: report of new mutations and complex genomic rearrangements leading to Incontinentia pigmenti disease/doi:10.1002%2Fhumu.22483./rivista:Human mutation/anno:2014/pagina_da:165/pagina_a:177/intervallo_pagine:165–177/volume:35
Incontinentia Pigmenti (IP) is an X-linked-dominant Mendelian disorder caused by mutation in the IKBKG/NEMO gene, encoding for NEMO/IKKgamma, a regulatory protein of NF-kB signaling. In more than 80% of cases, IP is due to recurrent or non-recurrent
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=cnr_________::d3fa48a662df7e1a2f6256e62030ef0d
https://publications.cnr.it/doc/271032
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9
Akademický článek
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10
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