Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Mauro Racaniello"'
Autor:
Giovanna De Chiara, Mauro Racaniello, Cristiana Mollinari, Maria Elena Marcocci, Giorgia Aversa, Alessio Cardinale, Anna Giovanetti, Enrico Garaci, Anna Teresa Palamara, Daniela Merlo
Publikováno v:
Frontiers in Aging Neuroscience, Vol 8 (2016)
Several findings suggest that HSV-1 infection plays a role in the neurodegenerative processes that characterize Alzheimer’s disease, but the underlying mechanisms have yet to be fully elucidated. Here we show that HSV-1 productive infection in cort
Externí odkaz:
https://doaj.org/article/dadbfb689b744248b3b6f89f553c736d
Autor:
Laura Narciso, Eleonora Parlanti, Mauro Racaniello, Valeria Simonelli, Alessio Cardinale, Daniela Merlo, Eugenia Dogliotti
Publikováno v:
Neural Plasticity, Vol 2016 (2016)
Externí odkaz:
https://doaj.org/article/2822e8ac694046f1bb11df0d94cb96b5
Autor:
Enrico Garaci, Maria Chiara de Stefano, Cristiana Mollinari, Alessio Cardinale, Mauro Racaniello, Daniela Merlo
Publikováno v:
Neurochemical research (2015). doi:10.1007/s11064-014-1465-1
info:cnr-pdr/source/autori:Cardinale A.; De Stefano M.C.; Mollinari C.; Racaniello M.; Garaci E.; Merlo D./titolo:Biochemical Characterization of Sirtuin 6 in the Brain and Its Involvement in Oxidative Stress Response/doi:10.1007%2Fs11064-014-1465-1/rivista:Neurochemical research/anno:2015/pagina_da:/pagina_a:/intervallo_pagine:/volume
info:cnr-pdr/source/autori:Cardinale A.; De Stefano M.C.; Mollinari C.; Racaniello M.; Garaci E.; Merlo D./titolo:Biochemical Characterization of Sirtuin 6 in the Brain and Its Involvement in Oxidative Stress Response/doi:10.1007%2Fs11064-014-1465-1/rivista:Neurochemical research/anno:2015/pagina_da:/pagina_a:/intervallo_pagine:/volume
Sirtuin 6 (SIRT6) is a member of nicotinamide adenine dinucleotide-dependent deacetylase protein family and has been implicated in the control of glucose and lipid metabolism, cancer, genomic stability and DNA repair. Moreover, SIRT6 regulates the ex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b8748102759e6b975ba07441cd435c8
https://doi.org/10.1007/s11064-014-1465-1
https://doi.org/10.1007/s11064-014-1465-1
Autor:
Claudio Frank, A Zaratti, Stefano Rufini, Alessio Cardinale, Mauro Racaniello, Virginia Tancredi, Daniele Grossi, Alessandro Cutarelli, Daniela Merlo, Giovanna D'Arcangelo
Publikováno v:
Neural Plasticity
Neural Plasticity, Vol 2016 (2016)
Neural Plasticity, Vol 2016 (2016)
Niemann-Pick type C disease is an autosomal recessive storage disorder, characterized by abnormal sequestration of unesterified cholesterol within the late endolysosomal compartment of cells and accumulation of gangliosides and other sphingolipids. P
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a3552fa2209610e80d6d695721e009f3
http://hdl.handle.net/2108/146487
http://hdl.handle.net/2108/146487
Autor:
Cristiana Mollinari, Maurizio Pocchiari, Maria Chiara de Stefano, Serena Saladini, Giovanna De Chiara, Enrico Garaci, Alessio Cardinale, Daniela Merlo, Mauro Racaniello
Publikováno v:
Journal of Biological Chemistry. 287:2618-2631
Accumulation of DNA damage and deficiency in DNA repair potentially contribute to the progressive neuronal loss in neurodegenerative disorders, including Alzheimer disease (AD). In multicellular eukaryotes, double strand breaks (DSBs), the most letha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f6644f0864ad6a09abd27a5a2a27e512
https://doi.org/10.1074/jbc.m111.276550
https://doi.org/10.1074/jbc.m111.276550
Publikováno v:
Current Alzheimer research. 13(11)
Accumulation of DNA damage and impairment of DNA repair systems are involved in the pathogenesis of different neurodegenerative diseases. Whenever DNA damage is too extensive, the DNA damage response pathway provides for triggering cellular senescenc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::37b13cfedbae3d66f831b28282abbe04
https://pubmed.ncbi.nlm.nih.gov/27033054
https://pubmed.ncbi.nlm.nih.gov/27033054
Autor:
Mauro Racaniello, Cristina Zona, Alessio Cardinale, Massimo Pieri, Enrico Garaci, Alessandra Berry, M C de Stefano, Daniela Merlo, Francesca Cirulli, Cristiana Mollinari
Publikováno v:
Cell death and disease (2015). doi:10.1038/cddis.2014.589.
info:cnr-pdr/source/autori:Mollinari C, Racaniello M, Berry A, Pieri M, de Stefano MC, Cardinale A, Zona C, Cirulli F, Garaci E, Merlo D./titolo:miR-34a regulates cell proliferation, morphology and function of newborn neurons resulting in improved behavioural outcomes/doi:10.1038%2Fcddis.2014.589./rivista:Cell death and disease/anno:2015/pagina_da:/pagina_a:/intervallo_pagine:/volume
Cell Death & Disease
info:cnr-pdr/source/autori:Mollinari C, Racaniello M, Berry A, Pieri M, de Stefano MC, Cardinale A, Zona C, Cirulli F, Garaci E, Merlo D./titolo:miR-34a regulates cell proliferation, morphology and function of newborn neurons resulting in improved behavioural outcomes/doi:10.1038%2Fcddis.2014.589./rivista:Cell death and disease/anno:2015/pagina_da:/pagina_a:/intervallo_pagine:/volume
Cell Death & Disease
miR-34a is involved in the regulation of the fate of different cell types. However, the mechanism by which it controls the differentiation programme of neural cells remains largely unknown. Here, we investigated the role of miR-34a in neurogenesis an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::57584c314c63f21c3e301834fb015527
http://www.nature.com/cddis/journal/v6/n1/full/cddis2014589a.html
http://www.nature.com/cddis/journal/v6/n1/full/cddis2014589a.html
Autor:
Virginia Tancredi, Daniela Merlo, Cristiana Mollinari, Alessio Cardinale, Margherita D'Antuono, Giovanna De Chiara, Mauro Racaniello
Publikováno v:
Neurochemical research 35 (2010): 239–246. doi:10.1007/s11064-009-0047-0
info:cnr-pdr/source/autori:Racaniello M.1,2; Cardinale A.1; Mollinari C.3,4; D'Antuono M.2; De Chiara G.3; Tancredi V.2; Merlo D.1,3/titolo:Phosphorylation changes of CaMKII, ERK1%2F2, PKB%2FAkt kinases and CREB activation during early long-term potentiation at Schaffer collateral-CA1 mouse hippocampal synapses/doi:10.1007%2Fs11064-009-0047-0/rivista:Neurochemical research/anno:2010/pagina_da:239/pagina_a:246/intervallo_pagine:239–246/volume:35
info:cnr-pdr/source/autori:Racaniello M.1,2; Cardinale A.1; Mollinari C.3,4; D'Antuono M.2; De Chiara G.3; Tancredi V.2; Merlo D.1,3/titolo:Phosphorylation changes of CaMKII, ERK1%2F2, PKB%2FAkt kinases and CREB activation during early long-term potentiation at Schaffer collateral-CA1 mouse hippocampal synapses/doi:10.1007%2Fs11064-009-0047-0/rivista:Neurochemical research/anno:2010/pagina_da:239/pagina_a:246/intervallo_pagine:239–246/volume:35
Protein phosphorylation is the main signaling system known to trigger synaptic changes underlying long-term potentiation (LTP). The timing of these phosphorylations plays an essential role to maintain the potentiated state of synapses. However, in mi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5c57efccd73add1bc4cfeed41e783374
https://publications.cnr.it/doc/166916
https://publications.cnr.it/doc/166916
Autor:
Enrico Garaci, Lucia Ricci-Vitiani, Daniela Merlo, Corrado Lucantoni, Anna Maria Rinaldi, Mauro Racaniello, Cristina Zona, Roberto Pallini, Ruggero De Maria, Cristiana Mollinari, Massimo Pieri
Publikováno v:
Journal of cell science 122 (2009): 4195–4207. doi:10.1242/jcs.056895
info:cnr-pdr/source/autori:Mollinari, C1; Ricci-Vitiani, L2; Pieri, M3,4; Lucantoni, C5; Rinaldi, AM3; Racaniello, M3,6; De Maria, R2; Zona, C3,4; Pallini, R5; Merlo, D1,6; Garaci, E7/titolo:Downregulation of thymosin beta4 in neural progenitor grafts promotes spinal cord regeneration/doi:10.1242%2Fjcs.056895/rivista:Journal of cell science/anno:2009/pagina_da:4195/pagina_a:4207/intervallo_pagine:4195–4207/volume:122
info:cnr-pdr/source/autori:Mollinari, C1; Ricci-Vitiani, L2; Pieri, M3,4; Lucantoni, C5; Rinaldi, AM3; Racaniello, M3,6; De Maria, R2; Zona, C3,4; Pallini, R5; Merlo, D1,6; Garaci, E7/titolo:Downregulation of thymosin beta4 in neural progenitor grafts promotes spinal cord regeneration/doi:10.1242%2Fjcs.056895/rivista:Journal of cell science/anno:2009/pagina_da:4195/pagina_a:4207/intervallo_pagine:4195–4207/volume:122
Thymosin beta4 (Tbeta4) is an actin-binding peptide whose expression in developing brain correlates with migration and neurite extension of neurons. Here, we studied the effects of the downregulation of Tbeta4 expression on growth and differentiation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::27e674e9bcde175f45d125bf4bf7b4bf
https://pubmed.ncbi.nlm.nih.gov/19861493
https://pubmed.ncbi.nlm.nih.gov/19861493
Autor:
Guido Valesini, Elena Ortona, Paola Margutti, Alessandra Siracusano, Fabrizio Conti, Rachele Riganò, Mauro Racaniello, Cristiano Alessandri, Federica Delunardo, Elisabetta Profumo, Maurizio Sorice
Publikováno v:
Arthritis Research & Therapy. 7:R896
Anti-endothelial-cell antibodies are associated with psychiatric manifestations in systemic lupus erythematosus (SLE). Our primary aim in this study was to seek and characterize molecules that behave as endothelial autoantigens in SLE patients with p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::70b8c9c90357f6810b27dbf4ba7a0c20
https://doi.org/10.1186/ar1759
https://doi.org/10.1186/ar1759