Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Matthias Pillath‐Eilers"'
Autor:
Ayan Mohamud Yusuf, Mina Borbor, Tanja Hussner, Carolin Weghs, Britta Kaltwasser, Matthias Pillath-Eilers, Bernd Walkenfort, Richard Kolesnick, Erich Gulbins, Dirk M. Hermann, Ulf Brockmeier
Publikováno v:
Cell Death and Disease, Vol 15, Iss 9, Pp 1-14 (2024)
Abstract Acid sphingomyelinase (ASM) inhibitors are widely used for the treatment of post-stroke depression. They promote neurological recovery in animal stroke models via neurorestorative effects. In a previous study, we found that antidepressants i
Externí odkaz:
https://doaj.org/article/bb96a3a7cca44e52960359d37b31a205
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 15 (2021)
Astrocytic networks are critically involved in regulating the activity of neuronal networks. However, a comprehensive and ready-to-use data analysis tool for investigating functional interactions between the astrocytes is missing. We developed the no
Externí odkaz:
https://doaj.org/article/66ab24120ffe4e26bc37b6982b375706
Autor:
Mina Borbor, Dongpei Yin, Ulf Brockmeier, Chen Wang, Marius Doeckel, Matthias Pillath‐Eilers, Britta Kaltwasser, Dirk M. Hermann, Egor Dzyubenko
Astrocytic responses are critical for the maintenance of neuronal networks in health and disease. In stroke, reactive astrocytes undergo functional changes potentially contributing to secondary neurodegeneration, but the mechanisms of astrocyte-media
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef5a72cbf4cbf133f3b48096dc8ab8eb
https://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&origin=inward&scp=85148596173
https://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&origin=inward&scp=85148596173
Autor:
Egor Dzyubenko, Daniel Manrique-Castano, Matthias Pillath-Eilers, Paraskevi Vasileiadou, Jacqueline Reinhard, Andreas Faissner, Dirk M Hermann
Publikováno v:
Matrix biology : journal of the International Society for Matrix Biology. 110
Cellular responses in glia play a key role in regulating brain remodeling post-stroke. However, excessive glial reactivity impedes post-ischemic neuroplasticity and hampers neurological recovery. While damage-associated molecular patterns and activat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f698c15d42358800c3a4481ffccc6fac
https://pubmed.ncbi.nlm.nih.gov/35430380
https://pubmed.ncbi.nlm.nih.gov/35430380