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pro vyhledávání: '"Matthew R Lynch"'
Autor:
Matthew R. Lynch, Susie L Hu
Publikováno v:
Journal of Onco-Nephrology. 4:7-14
With increasing prevalence of both kidney disease and cancer, patients who have both are common. Toxicity from treatment or direct kidney injury by the cancer itself can lead to acute kidney injury or progression of pre-existing chronic kidney diseas
Autor:
Matthew R. Lynch, Ankur D. Shah
Publikováno v:
Kidney360
Accessibility to dialysis facilities plays a central role when deciding on a patient's long-term dialysis modality. Studies investigating the effect of distance to nearest dialysis-providing unit on modality choice have yielded conflicting results. W
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b3b824a25dc9aa9da3ccaec501f960f4
https://europepmc.org/articles/PMC8986047/
https://europepmc.org/articles/PMC8986047/
Publikováno v:
American Journal of Kidney Diseases
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) continues to spread around the world. As of the end of June 2021, there were approximately 181 million confirmed cases and more than 3.9 million deaths across the globe. The colossal impact
Autor:
Matthew R, Lynch, Jie, Tang
Publikováno v:
Rhode Island medical journal (2013). 103(8)
Acute kidney injury (AKI) has been reported as a complication of COVID-19. However, the epidemiology, management, and associated outcomes have varied greatly between studies. The pathophysiology remains unclear. Summary: The etiology of AKI in the se
Publikováno v:
Hemodialysis international. International Symposium on Home Hemodialysis. 25(1)
Autor:
Isaac E. Stillman, Toren Finkel, Xiuying Chen, Ilsa I. Rovira, Samir M. Parikh, Kensei Taguchi, Matthew R Lynch, Daniel A. Brown, Mei T. Tran, Swati S. Bhasin, Manoj Bhasin, Kenneth M. Ralto, Vinod Raman, Nuo Sun, Craig R. Brooks, Zsuzsanna K. Zsengellér
Publikováno v:
JCI Insight
Because injured mitochondria can accelerate cell death through the elaboration of oxidative free radicals and other mediators, it is striking that proliferator γ coactivator 1-α (PGC1α), a stimulator of increased mitochondrial abundance, protects
Autor:
Anders H. Berg, Sushrut S. Waikar, Adam Lerner, Eugene P. Rhee, Samir M. Parikh, Matthew R Lynch, Mary E. Trovato, Clary B. Clish, Steven H. Kim, Ravi Thadhani, David E. Leaf, Ali Poyan Mehr, Joseph Messmer, Kamal R. Khabbaz, Charbel C. Khoury, Shoshana J. Herzig, Mei T. Tran, Ajay Kher, Noemie Simon-Tillaux, Kenneth M. Ralto, Vaughan Washco
Publikováno v:
Nature medicine
Nicotinamide adenine dinucleotide (NAD+) extends longevity in experimental organisms, raising interest in its impact on human health. De novo NAD+ biosynthesis from tryptophan is evolutionarily conserved yet considered supplanted among higher species
Publikováno v:
American Journal of Physiology-Renal Physiology. 314:F1-F8
Acute kidney injury (AKI) arising from diverse etiologies is characterized by mitochondrial dysfunction. The peroxisome proliferator-activated receptor γ coactivator-1alpha (PGC1α), a master regulator of mitochondrial biogenesis, has been shown to