Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Matthew J. Leventhal"'
Autor:
Vanitha Nithianandam, Hassan Bukhari, Matthew J. Leventhal, Rachel A. Battaglia, Xianjun Dong, Ernest Fraenkel, Mel B. Feany
Publikováno v:
Nature Communications, Vol 14, Iss 1, Pp 1-19 (2023)
Abstract Aβ peptides derived from the amyloid precursor protein (APP) have been strongly implicated in the pathogenesis of Alzheimer’s disease. However, the normal function of APP and the importance of that role in neurodegenerative disease is les
Externí odkaz:
https://doaj.org/article/189e34beec2d4e72b27b05854a87e2ee
Autor:
Max Jan, Matthew J. Leventhal, Elizabeth A. Morgan, Jordan C. Wengrod, Anwesha Nag, Samantha D. Drinan, Bruce M. Wollison, Matthew D. Ducar, Aaron R. Thorner, Scott Leppanen, Jane Baronas, Jonathan Stevens, William J. Lane, Natasha Kekre, Vincent T. Ho, John Koreth, Corey S. Cutler, Sarah Nikiforow, Edwin P. Alyea, III, Joseph H. Antin, Robert J. Soiffer, Jerome Ritz, R. Coleman Lindsley, Benjamin L. Ebert
Publikováno v:
Blood Advances, Vol 3, Iss 14, Pp 2199-2204 (2019)
Abstract: Immune evasion is a hallmark of cancer and a central mechanism underlying acquired resistance to immune therapy. In allogeneic hematopoietic cell transplantation (alloHCT), late relapses can arise after prolonged alloreactive T-cell control
Externí odkaz:
https://doaj.org/article/dd67bff001a141b5ae1e7eda4fd3a48d
Autor:
Mathieu Gigoux, Morten O. Holmström, Roberta Zappasodi, Joseph J. Park, Stephane Pourpe, Cansu Cimen Bozkus, Levi M. B. Mangarin, David Redmond, Svena Verma, Sara Schad, Mariam M. George, Divya Venkatesh, Arnab Ghosh, David Hoyos, Zaki Molvi, Baransel Kamaz, Anna E. Marneth, William Duke, Matthew J. Leventhal, Max Jan, Vincent T. Ho, Gabriela S. Hobbs, Trine Alma Knudsen, Vibe Skov, Lasse Kjær, Thomas Stauffer Larsen, Dennis Lund Hansen, R. Coleman Lindsley, Hans Hasselbalch, Jacob H. Grauslund, Thomas L. Lisle, Özcan Met, Patrick Wilkinson, Benjamin Greenbaum, Manuel A. Sepulveda, Timothy Chan, Raajit Rampal, Mads H. Andersen, Omar Abdel-Wahab, Nina Bhardwaj, Jedd D. Wolchok, Ann Mullally, Taha Merghoub
Publikováno v:
Science Translational Medicine. 14
The majority of JAK2V617F-negative myeloproliferative neoplasms (MPNs) have disease-initiating frameshift mutations in calreticulin (CALR), resulting in a common carboxyl-terminal mutant fragment (CALRMUT), representing an attractive source of neoant