Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Matthew F. Rathgeber"'
Autor:
Shang-Chuen Wu, Hau-Ming Jan, Mary L. Vallecillo-Zúniga, Matthew F. Rathgeber, Caleb S. Stowell, Kaleb L. Murdock, Kashyap R. Patel, Hirotomo Nakahara, Carter J. Stowell, Moon H. Nahm, Connie M. Arthur, Richard D. Cummings, Sean R. Stowell
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-14 (2023)
Abstract Microbial glycan microarrays (MGMs) populated with purified microbial glycans have been used to define the specificity of host immune factors toward microbes in a high throughput manner. However, a limitation of such arrays is that glycan pr
Externí odkaz:
https://doaj.org/article/c4280ad7b7c745f8a5a8f62ba788f3bd
Autor:
Shang-Chuen Wu, Connie M. Arthur, Hau-Ming Jan, Wilfredo F Garcia-Beltran, Kashyap R. Patel, Matthew F. Rathgeber, Hans Verkerke, Narayanaiah Cheedarla, Ryan Philip Jajosky, Anu Paul, Andrew S. Neish, John D. Roback, Cassandra D. Josephson, Duane R. Wesemann, Daniel Kalman, Seth Rakoff-Nahoum, Richard D. Cummings, Sean R. Stowell
Publikováno v:
Blood. 140:79-80
Autor:
Hailie N. Gill, Mary L. Vallecillo-Zúniga, Matthew F. Rathgeber, Christian J Arnold, Spencer Hayes, Pam M. Van Ry, Braden C. Kartchner, Peter Daniel Poulson, Ethan L. Dodson, Jacob S. Luddington, Seth R. Garfield, Sean R. Stowell, Connie M Arthur, Jonathan L Spallino, Jonard Corpuz Valdoz
Publikováno v:
Cells
Volume 10
Issue 11
Cells, Vol 10, Iss 3210, p 3210 (2021)
Volume 10
Issue 11
Cells, Vol 10, Iss 3210, p 3210 (2021)
Two of the main pathologies characterizing dysferlinopathies are disrupted muscle membrane repair and chronic inflammation, which lead to symptoms of muscle weakness and wasting. Here, we used recombinant human Galectin-1 (rHsGal-1) as a therapeutic
Autor:
Shang-Chuen Wu, Nourine A. Kamili, Marcelo Dias-Baruffi, Cassandra D. Josephson, Matthew F. Rathgeber, Melissa Y. Yeung, William J. Lane, Jianmei Wang, Hau-Ming Jan, Seth Rakoff-Nahoum, Richard D. Cummings, Sean R. Stowell, Connie M. Arthur
Publikováno v:
iScience. 25:104482
Adaptive immunity can target a nearly infinite range of antigens, yet it is tempered by tolerogenic mechanisms that limit autoimmunity. Such immunological tolerance, however, creates a gap in adaptive immunity against microbes decorated with self-lik
Autor:
Mary L Vallecillo-Zúniga, Matthew F Rathgeber, P Daniel Poulson, Spencer Hayes, Jacob S Luddington, Hailie N Gill, Matthew Teynor, Braden C Kartchner, Jonard Valdoz, Caleb Stowell, Ashley R Markham, Connie Arthur, Sean Stowell, Pam M Van Ry
Publikováno v:
PLoS ONE, Vol 15, Iss 9, p e0238441 (2020)
Limb-girdle muscular dystrophy type 2B (LGMD2B) is caused by mutations in the dysferlin gene, resulting in non-functional dysferlin, a key protein found in muscle membrane. Treatment options available for patients are chiefly palliative in nature and
Externí odkaz:
https://doaj.org/article/1b0d5091b3e842558ddcb01bd5b3fcc0