Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Matthew D. McLean"'
Autor:
Michael F. Papacostas, Dwight M. Robertson, Matthew D. McLean, Keisha D. Wolfe, Hui Liu, Timothy R. Shope
Publikováno v:
Pediatrics. 149(3)
OBJECTIVES To characterize the outcomes of ABO incompatible direct antiglobulin test (DAT) positive newborns and determine the predictive ability of a sixth-hour transcutaneous bilirubin (TcB for needing phototherapy ≤24 hours of age. METHODS Retro
Publikováno v:
The Anatomical Record. 297:1585-1595
The tropomyosin (TM) gene family produces a set of related TM proteins with important functions in striated and smooth muscle, and nonmuscle cells. In vertebrate striated muscle, the thin filament consists largely of actin, TM, the troponin (Tn) comp
Autor:
Belinda J. Spinner, Nancy Dobbins, Matthew D. McLean, Dipak K. Dube, Larry F. Lemanski, Christopher R. Denz, Robert W. Zajdel, Aruna Choudhury, Masako Nakatsugawa, Syamalima Dube, Sonali Mehta
Publikováno v:
Journal of Cellular Biochemistry. 85:747-761
A striated muscle isoform of a Tropomyosin (TM-4) gene was characterized and found to be necessary for contractile function in embryonic heart. The full-length clone of this isoform was isolated from the Mexican axolotl (Ambystoma mexicanum) and name
Autor:
David F. Wieczorek, Larry F. Lemanski, Matthew D. McLean, Sharon L. Lemanski, Robert W. Zajdel, Mariappan Muthuchamy, Dipak K. Dube
Publikováno v:
Developmental Dynamics. 213:412-420
Expression of tropomyosin protein, an essential component of the thin filament, has been found to be drastically reduced in cardiac mutant hearts of the Mexican axolotl (Ambystoma mexicanum) with no formation of sarcomeric myofibrils. Therefore, this
Publikováno v:
Anatomical record (Hoboken, N.J. : 2007). 297(9)
The tropomyosin (TM) gene family produces a set of related TM proteins with important functions in striated and smooth muscle, and nonmuscle cells. In vertebrate striated muscle, the thin filament consists largely of actin, TM, the troponin (Tn) comp
Publikováno v:
Cardiovascular Pharmacology: Open Access.
The Mexican axolotl (Ambystoma mexicanum) is a unique model to study vertebrate heart development for several reasons. In addition to the wild-type animal, there is also an embryonic lethal condition caused by a homozygous recessive mutation in cardi
Publikováno v:
Journal of Clinical & Experimental Cardiology.
Copyright: © 2013 Dubev DK, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and
Autor:
Matthew D McLean, Aaron Bass Brady
Publikováno v:
Pediatrics. 137:222A-222A
Autor:
Bernard J. Poiesz, Dipak K. Dube, Harold L. Thurston, Matthew D. McLean, Christopher R. Denz, Syamalima Dube, Robert W. Zajdel
Publikováno v:
Journal of cellular biochemistry. 99(3)
The Mexican axolotl, Ambystoma mexicanum, serves as an intriguing model to investigate myofibril organization and heart development in vertebrates. The axolotl has a homozygous recessive cardiac lethal gene "c" which causes a failure of ventricular m
Publikováno v:
Cardiovascular toxicology. 6(2)
In the axolotl, Ambystoma mexicanum, a simple, recessive cardiac-lethal mutation in gene "c" results in the hearts of c/c homozygous animals being deficient in sarcomeric tropomyosin (TM) and failing to form mature myofibrils. Subsequently, the mutan