Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Matteo Melensi"'
Autor:
Maurizio Aricò, Elena Boggio, Valentina Cetica, Matteo Melensi, Elisabetta Orilieri, Nausicaa Clemente, Giuseppe Cappellano, Sara Buttini, Maria Felicia Soluri, Cristoforo Comi, Carlo Dufour, Daniela Pende, Irma Dianzani, Steven R Ellis, Sara Pagliano, Stefania Marcenaro, Ugo Ramenghi, Annalisa Chiocchetti, Umberto Dianzani
Publikováno v:
PLoS ONE, Vol 8, Iss 7, p e68045 (2013)
Autoimmune lymphoproliferative syndrome (ALPS) is caused by genetic defects decreasing Fas function and is characterized by lymphadenopathy/splenomegaly and expansion of CD4/CD8 double-negative T cells. This latter expansion is absent in the ALPS var
Externí odkaz:
https://doaj.org/article/d2f08b5859d14113bd79ed499b9f84e0
Publikováno v:
Computers & Mathematics with Applications. 66:1010-1023
How do we recast the effects of molecular mimicry and genetic alterations affecting the T -cell response against self and non-self antigens into a mathematical model for the development of autoimmune disorders? Bearing this question in mind, we propo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d98919cd4183288d673f63f14a696119
https://doi.org/10.1016/j.camwa.2013.06.026
https://doi.org/10.1016/j.camwa.2013.06.026
Autor:
Matteo Melensi, Umberto Dianzani, Giorgio Leigheb, Ghislain Emmanuel Sopoh, Elisa Zavattaro, Roch Christian Johnson, Ange Dodji Dossou, Riccardo Mesturini
Publikováno v:
International Journal of Dermatology. 49:1297-1302
Background Buruli Ulcer (BU) is a severe cutaneous and subcutaneous disease due to Mycobacterium ulcerans infection, mainly distributed in sub-Saharan Africa and tropical areas. The role of T helper (TH) cytokines in the development and clinical cour
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ae6202ca0fdf9e6695d7505131d5c574
https://doi.org/10.1111/j.1365-4632.2010.04615.x
https://doi.org/10.1111/j.1365-4632.2010.04615.x
Autor:
Alexander Lorz, Marcello Edoardo Delitala, Rebecca H. Chisholm, Tommaso Lorenzi, Matteo Melensi
Publikováno v:
Immunology
Immunology, Wiley, 2015, 146 (2), pp.271-280. ⟨10.1111/imm.12500⟩
Immunology, 2015, 146 (2), pp.271-280. ⟨10.1111/imm.12500⟩
Immunology, Wiley, 2015, 146 (2), pp.271-280. ⟨10.1111/imm.12500⟩
Immunology, 2015, 146 (2), pp.271-280. ⟨10.1111/imm.12500⟩
International audience; T cells are key players in immune action against the invasion of target cells expressing non-self antigens. During an immune response, antigen-specific T cells dynamically sculpt the antigenic distribution of target cells, and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3cf391ffb42753a0346b0192489d3b5e
https://hal.inria.fr/hal-01237852
https://hal.inria.fr/hal-01237852
Publikováno v:
ITM Web of Conferences, Vol 5, p 00005 (2015)
T cells are key players in the immune action against the invasion of can- cer cells. During an immune response, antigen-specific T cells dynamically sculpt the antigenic distribution of cancer cells, and cancer cells concurrently shape the repertoire
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4e60b37460a3e9ddf7815c718f75ee61
https://doaj.org/article/9d1ec3305417465b88ca3443d0cfb715
https://doaj.org/article/9d1ec3305417465b88ca3443d0cfb715
Publikováno v:
Mathematical Models of Tumor-Immune System Dynamics ISBN: 9781493917921
How does immunotherapy affect the evolutionary dynamics of cancer cells? Can we enhance the anti-cancer efficacy of T cells by using different types of immune boosters in combination? Bearing these questions in mind, we present a mathematical model o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::569c61900f9795082a0f3f8cf44d1c81
http://hdl.handle.net/11583/2671308
http://hdl.handle.net/11583/2671308
Autor:
Irma Dianzani, Maurizio Aricò, Matteo Melensi, Ugo Ramenghi, Umberto Dianzani, Annalisa Chiocchetti, Elena Boggio
Publikováno v:
Pediatrics. 132(4)
This article presents a case report for a child presenting with mixed clinical features of autoimmune lymphoproliferative syndrome (ALPS), familial hemophagocytic lymphohistiocytosis (FHL), and X-linked lymphoproliferative (XLP) disease. From 6 month
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6d22f910a6067d2d39e03f0a6a5de7cb
https://pubmed.ncbi.nlm.nih.gov/24043286
https://pubmed.ncbi.nlm.nih.gov/24043286
Autor:
Giuseppe Cappellano, Cristoforo Comi, Elisabetta Orilieri, Stefania Marcenaro, Maria Felicia Soluri, Steven R. Ellis, Nausicaa Clemente, Annalisa Chiocchetti, Ugo Ramenghi, Irma Dianzani, Valentina Cetica, Sara Pagliano, Daniela Pende, Maurizio Aricò, Elena Boggio, Umberto Dianzani, Sara Buttini, Matteo Melensi, Carlo Dufour
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 7, p e68045 (2013)
PLoS ONE, Vol 8, Iss 7, p e68045 (2013)
Autoimmune lymphoproliferative syndrome (ALPS) is caused by genetic defects decreasing Fas function and is characterized by lymphadenopathy/splenomegaly and expansion of CD4/CD8 double-negative T cells. This latter expansion is absent in the ALPS var
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::294786f17077191406d406da2a3ec225
http://europepmc.org/articles/PMC3698121
http://europepmc.org/articles/PMC3698121
Autor:
Umberto Dianzani, Franco Aina, Francesca Uberti, Elena Grossini, Ezio Micalizzi, Annalisa Chiocchetti, Philippe Primo Caimmi, Cristiano Ariatti, Chiara Dianzani, Enrico Crosio, Giuseppe Cappellano, Giovanni Vacca, David A.S.G. Mary, Gabriella Nicosia, Renzo Boldorini, Matteo Melensi, Maria Prat, Stefano Pietronave
Publikováno v:
The Canadian journal of cardiology. 29(4)
Background The endocannabinoid system reportedly plays a role in the pathogenesis of cardiovascular diseases. This system is expressed also in adipose tissue, which could thus be involved in cardiac disorders through modulation of metabolically trigg
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3fad94f4f727149e2b3b9cbf1b987b79
https://pubmed.ncbi.nlm.nih.gov/22926037
https://pubmed.ncbi.nlm.nih.gov/22926037
Autor:
Elisabetta Orilieri, Cristoforo Comi, Umberto Dianzani, Rosella Mechelli, Marco Salvetti, Ugo Ramenghi, Sara Bocca, Maria Felicia Soluri, Nausicaa Clemente, Alessandro Poggi, Matteo Melensi, Elena Boggio, Sandra D'Alfonso, Giovanna Gentile, Annalisa Chiocchetti
Inherited defects decreasing function of the Fas death receptor cause autoimmune lymphoproliferative syndrome (ALPS) and its variant Dianzani autoimmune lymphoproliferative disease (DALD). Since a deleterious mutation of the SH2D1A gene protects MRLl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5f81e37d5268d80c731539918afecce1
http://hdl.handle.net/2318/108904
http://hdl.handle.net/2318/108904