Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Matteo Falappa"'
Autor:
Lorenzo Muzzi, Donatella Di Lisa, Matteo Falappa, Sara Pepe, Alessandro Maccione, Laura Pastorino, Sergio Martinoia, Monica Frega
Publikováno v:
Bioengineering, Vol 10, Iss 4, p 449 (2023)
With the advent of human-induced pluripotent stem cells (hiPSCs) and differentiation protocols, methods to create in-vitro human-derived neuronal networks have been proposed. Although monolayer cultures represent a valid model, adding three-dimension
Externí odkaz:
https://doaj.org/article/42cbfbcc955f45719c5b0ba4486d744b
Autor:
Marta Pace, Matteo Falappa, Patricia Machado, Laura Facchin, Dirk M Hermann, Claudio L Bassetti
Publikováno v:
Clinical and Translational Neuroscience, Vol 4 (2020)
Background: Ischemic stroke causes hypoexcitability in the peri-infarct motor neocortex that stems from increased tonic γ-amino-butyric acid (GABA) activity in neurons. This hypoexcitability, while neuroprotective in the acute phase, may impair neur
Externí odkaz:
https://doaj.org/article/883ec7d55c3e43c285f26034f6a9df82
Autor:
Lorenzo Muzzi, Donatella Di Lisa, Matteo Falappa, Sara Pepe, Alessandro Maccione, Laura Pastorino, Monica Frega, Sergio Martinoia
Publikováno v:
SSRN Electronic Journal.
Autor:
Dirk M. Hermann, Patricia Machado, Marta Pace, Matteo Falappa, Claudio L. Bassetti, Laura Facchin
Publikováno v:
Clinical and Translational Neuroscience, Vol 4 (2020)
Background: Ischemic stroke causes hypoexcitability in the peri-infarct motor neocortex that stems from increased tonic γ-amino-butyric acid (GABA) activity in neurons. This hypoexcitability, while neuroprotective in the acute phase, may impair neur
Autor:
Giovanna Zoccoli, Matteo Falappa, Matteo Cerri, Chiara Berteotti, Andrea Freschi, Valter Tucci, Fatemeh Kaveh, Edoardo Balzani, Alfonso Urbanucci, Eivind Hovig, Roberto Amici, Viviana Lo Martire, Marta Pace
Publikováno v:
JCI Insight
Imprinted genes are highly expressed in the hypothalamus; however, whether specific imprinted genes affect hypothalamic neuromodulators and their functions is unknown. It has been suggested that Prader–Willi syndrome (PWS), a neurodevelopmental dis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d705fc6615e6dcec06c17459887084d3
https://europepmc.org/articles/PMC7406246/
https://europepmc.org/articles/PMC7406246/
Autor:
Matteo Cerri, Andrea Freschi, Blanco María Encarnación, Mojtaba Bandarabadi, Andrea Armirotti, Marta Pace, Antoine Roger Adamantidis, Michela Chiappalone, Ilaria Colombi, Matteo Falappa, Valter Tucci, Roberto Amici
Publikováno v:
Pace, Marta; Colombi, Ilaria; Falappa, Matteo; Freschi, Andrea; Bandarabadi, Mojtaba; Armirotti, Andrea; Encarnación, Blanco María; Adamantidis, Antoine R; Amici, Roberto; Cerri, Matteo; Chiappalone, Michela; Tucci, Valter (2020). Loss of Snord116 alters cortical neuronal activity in mice: a preclinical investigation of Prader-Willi syndrome. Human molecular genetics, 29(12), pp. 2051-2064. Oxford University Press 10.1093/hmg/ddaa084
Prader–Willi syndrome (PWS) is a neurodevelopmental disorder that is characterized by metabolic alteration and sleep abnormalities mostly related to rapid eye movement (REM) sleep disturbances. The disease is caused by genomic imprinting defects th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5c886f8f6ea44c0cf0794fe226541e4b
http://hdl.handle.net/11585/780279
http://hdl.handle.net/11585/780279
Autor:
Laura Facchin, Marta Pace, Patricia Machado, Dirk M. Hermann, Matteo Falappa, Claudio L. Bassetti
BackgroundIschemic stroke induces hypoexcitability of the peri-infarct cortex by increased tonic GABA activity, which impairs stroke recovery. The GABAA α5 antagonist S44819 has recently been shown to promote post-ischemic motor-coordination recover
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::09e942c71805d9ae2a881811114b1595