Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Matt Bere"'
Autor:
Sharon B. Cantor, Neil Johnson, Lihua J. Zhu, Bin Deng, Jianhong Ou, Matt Bere, Mihir B. Doshi, Jennifer A. Calvo, Samuel M. Bond, Sumeet U. Nayak, Michelle Mosqueda, Min Peng, Ke Cong, John J. Krais, Nicholas J. Panzarino
Defects in DNA repair and the protection of stalled DNA replication forks are thought to underlie the chemosensitivity of tumors deficient in the hereditary breast cancer genes BRCA1 and BRCA2 (BRCA). Challenging this assumption are recent findings t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::06de07f4f7ad1796e1fb15bd0f3b947c
https://doi.org/10.1158/0008-5472.c.6512572.v1
https://doi.org/10.1158/0008-5472.c.6512572.v1
Autor:
Sharon B. Cantor, Neil Johnson, Lihua J. Zhu, Bin Deng, Jianhong Ou, Matt Bere, Mihir B. Doshi, Jennifer A. Calvo, Samuel M. Bond, Sumeet U. Nayak, Michelle Mosqueda, Min Peng, Ke Cong, John J. Krais, Nicholas J. Panzarino
Supplemental Figure Legends
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5154b074718c1c75fc74fd60d98c0d25
https://doi.org/10.1158/0008-5472.22427155
https://doi.org/10.1158/0008-5472.22427155
Autor:
Sharon B. Cantor, Neil Johnson, Lihua J. Zhu, Bin Deng, Jianhong Ou, Matt Bere, Mihir B. Doshi, Jennifer A. Calvo, Samuel M. Bond, Sumeet U. Nayak, Michelle Mosqueda, Min Peng, Ke Cong, John J. Krais, Nicholas J. Panzarino
Supplemental Figures 1-6. Figure S1: BRCA-deficient cancer cells fail to restrain replication in the presence of stress and ssDNA gaps develop. Figure S2: Replication restraint, depletion, and PDX controls. Figure S3: Depletion of SMARCAL1 or inhibit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4895446f2204506ea34735722026f78a
https://doi.org/10.1158/0008-5472.22427161
https://doi.org/10.1158/0008-5472.22427161
Autor:
Ke Cong, John J. Krais, Michelle Mosqueda, Sharon B. Cantor, Sumeet Nayak, Lihua Julie Zhu, Jianhong Ou, Min Peng, Matt Bere, Neil Johnson, Samuel M. Bond, Jennifer A. Calvo, Bin Deng, Nicholas J. Panzarino, Mihir B. Doshi
Publikováno v:
Cancer Res
Defects in DNA repair and the protection of stalled DNA replication forks are thought to underlie the chemosensitivity of tumors deficient in the hereditary breast cancer genes BRCA1 and BRCA2 (BRCA). Challenging this assumption are recent findings t
Autor:
Neil Johnson, Nicholas J. Panzarino, Arne Nedergaard Kousholt, Jennifer A. Calvo, Sharon B. Cantor, Matt Bere, Jos Jonkers, Sumeet Nayak, Wei Ting C. Lee, Eli Rothenberg, Ke Cong, Min Peng, John J. Krais
BRCA1 or BRCA2 (BRCA)-deficient tumor cells have defects in DNA double strand break repair by homologous recombination (HR) and fork protection (FP) that are thought to underlie the sensitivity to poly(ADP-ribose) polymerase inhibitor (PARPi). Given
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::11726f93dbf7fe1c10652ca816760bca
https://doi.org/10.1101/781989
https://doi.org/10.1101/781989