Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Matilde Lleonart"'
Autor:
Santiago Ramon y Cajal, Inés de Torres, Javier Hernández-Losa, Matilde Lleonart, Elisabet Argelaguet, Laura Coch, Berta Pons, Gemma Armengol, Laura López-Vicente
Supplementary Data from Regulation of Replicative and Stress-Induced Senescence by RSK4, which is Down-regulated in Human Tumors
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8726971fef44bdce535efa03e6ac9a03
https://doi.org/10.1158/1078-0432.22439952
https://doi.org/10.1158/1078-0432.22439952
Autor:
Santiago Ramon y Cajal, Inés de Torres, Javier Hernández-Losa, Matilde Lleonart, Elisabet Argelaguet, Laura Coch, Berta Pons, Gemma Armengol, Laura López-Vicente
Purpose: The control of senescence and its biochemical pathways is a crucial factor for understanding cell transformation. In a large RNA interference screen, the RSK4 gene was found to be related to p53-dependent arrest. The purpose of the present s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b2ecd49b49f2a9bf1b97191d0ba71d3
https://doi.org/10.1158/1078-0432.c.6517335.v1
https://doi.org/10.1158/1078-0432.c.6517335.v1
Autor:
David Beach, Amancio Carnero, Gordon Peters, Juan Cruz Cigudosa, David Bernard, Matilde Lleonart, Preeti Kerai, Jesús Gil
Supplementary Figure 1 from Immortalization of Primary Human Prostate Epithelial Cells by c-Myc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::16fb97a6f2f971789f921912337718c6
https://doi.org/10.1158/0008-5472.22364787
https://doi.org/10.1158/0008-5472.22364787
Autor:
David Beach, Amancio Carnero, Gordon Peters, Juan Cruz Cigudosa, David Bernard, Matilde Lleonart, Preeti Kerai, Jesús Gil
A significant percentage of prostate tumors have amplifications of the c-Myc gene, but the precise role of c-Myc in prostate cancer is not fully understood. Immortalization of human epithelial cells involves both inactivation of the Rb/p16INK4a pathw
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::50d4ce40e5ee0710d7b8bf848f247a8a
https://doi.org/10.1158/0008-5472.c.6494361.v1
https://doi.org/10.1158/0008-5472.c.6494361.v1
Autor:
David Beach, Amancio Carnero, Gordon Peters, Juan Cruz Cigudosa, David Bernard, Matilde Lleonart, Preeti Kerai, Jesús Gil
Supplementary Figure 2 from Immortalization of Primary Human Prostate Epithelial Cells by c-Myc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73ca9e380de527c398e436cd103c0ad7
https://doi.org/10.1158/0008-5472.22364784
https://doi.org/10.1158/0008-5472.22364784
Autor:
Jesús Gil, Preeti Kerai, Matilde Lleonart, David Bernard, Juan Cruz Cigudosa, Gordon Peters, Amancio Carnero, David Beach
Publikováno v:
Cancer Research. 82:2656-2656
MAP17 and SGLT1 protein expression levels as prognostic markers for cervical tumor patient survival.
Autor:
Marco Perez, Juan M Praena-Fernandez, Blanca Felipe-Abrio, Maria A Lopez-Garcia, Antonio Lucena-Cacace, Angel Garcia, Matilde Lleonart, Guiovanna Roncador, Juan J Marin, Amancio Carnero
Publikováno v:
PLoS ONE, Vol 8, Iss 2, p e56169 (2013)
MAP17 is a membrane-associated protein that is overexpressed in human tumors. Because the expression of MAP17 increases reactive oxygen species (ROS) generation through SGLT1 in cancer cells, in the present work, we investigated whether MAP17 and/or
Externí odkaz:
https://doaj.org/article/346cee41b8ee4f1eac63fe110e3363f8
Autor:
Amancio Carnero, Matilde Lleonart
Publikováno v:
Inside the Cell. 1:96-105
Publikováno v:
The Open Pathology Journal. 2:57-62
Primary mammalian cells reach replicative exhaustion after several passages in vitro, a process called replica- tive senescence. During such a state of permanent growth arrest, senescent cells are refractory to physiological prolifera- tion stimuli w
Autor:
Amancio, Carnero, Matilde, Lleonart
Publikováno v:
BioEssays : news and reviews in molecular, cellular and developmental biology. 38
Tumors are often viewed as unique entities with specific behaviors. However, tumors are a mixture of differentially evolved subpopulations of cells in constant Darwinian evolution, selecting the fittest clone and allowing it to outgrow the rest. As i