Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Mathilde Prud'homme"'
Autor:
Mathilde Prud’homme, PhD, Maxime Coutrot, MD, MSc, Thibault Michel, MD, MSc, Louis Boutin, MD, MSc, Magali Genest, PhD, Françoise Poirier, PhD, Jean-Marie Launay, PharmD, PhD, Bocar Kane, Satoshi Kinugasa, MD, PhD, Niki Prakoura, PhD, Sophie Vandermeersch, Alain Cohen-Solal, MD, PhD, Claude Delcayre, PhD, Jane-Lise Samuel, MD, PhD, Ravindra Mehta, MD, Etienne Gayat, MD, PhD, Alexandre Mebazaa, MD, PhD, Christos E. Chadjichristos, PhD, Matthieu Legrand, MD, PhD
Publikováno v:
JACC: Basic to Translational Science, Vol 4, Iss 6, Pp 717-732 (2019)
Summary: Acute kidney injury is associated with increased risk of heart failure and mortality. This study demonstrates that acute kidney injury induces remote cardiac dysfunction, damage, injury, and fibrosis via a galectin-3 (Gal-3) dependent pathwa
Externí odkaz:
https://doaj.org/article/7f1da282244a4324a0d112605eb113cb
Publikováno v:
Anesthésie & Réanimation. 4:386-396
Resume Le terme de « syndrome cardio-renal » est defini comme tout desordre cardiaque et renal par lequel un dysfonctionnement aigu ou chronique dans un organe peut induire un dysfonctionnement aigu ou chronique dans l’autre. Cinq sous types ont
Autor:
Alain Cohen-Solal, Claude Delcayre, Mathilde Prud’homme, Dalila Boudia, Valérie Domergue, Héloïse Prigent, Jane-Lise Samuel, Renée Ventura-Clapier, Loubina Fazal, Anne Garnier, Philippe Mateo
Publikováno v:
Journal of Applied Physiology. 123:1579-1591
Exercise training has been demonstrated to have beneficial effects in patients with heart failure (HF) or diabetes. However, it is unknown whether diabetic patients with HF will benefit from exercise training. Male Wistar rats were fed either a stand
Publikováno v:
Nephron. 137:273-276
Acute kidney injury (AKI) has been associated with both short- and long-term outcomes. To date, there is still a debate whether the increase risk of morbidity and mortality is directly due to AKI occurrence. There is, however, a potential causal impa
Autor:
Françoise Poirier, Etienne Gayat, Claude Delcayre, Jean-Marie Launay, Thibault Michel, Bocar Kane, Satoshi Kinugasa, Alain Cohen-Solal, Magali Genest, Matthieu Legrand, Maxime Coutrot, Louis Boutin, Sophie Vandermeersch, Christos E. Chadjichristos, Alexandre Mebazaa, Mathilde Prud’homme, Ravindra L. Mehta, Niki Prakoura, Jane-Lise Samuel
Publikováno v:
JACC. Basic to translational science, vol 4, iss 6
JACC: Basic to Translational Science
JACC: Basic to Translational Science, Vol 4, Iss 6, Pp 717-732 (2019)
JACC : Basic to Translational Science
JACC : Basic to Translational Science, Elsevier on behalf of the American College of Cardiology Foundation, 2019, 4, pp.717-732. ⟨10.1016/j.jacbts.2019.06.005⟩
JACC : Basic to Translational Science, 2019, 4 (6), pp.717-732. ⟨10.1016/j.jacbts.2019.06.005⟩
JACC: Basic to Translational Science
JACC: Basic to Translational Science, Vol 4, Iss 6, Pp 717-732 (2019)
JACC : Basic to Translational Science
JACC : Basic to Translational Science, Elsevier on behalf of the American College of Cardiology Foundation, 2019, 4, pp.717-732. ⟨10.1016/j.jacbts.2019.06.005⟩
JACC : Basic to Translational Science, 2019, 4 (6), pp.717-732. ⟨10.1016/j.jacbts.2019.06.005⟩
Visual Abstract
Highlights • In 2 different mouse models, AKI increased Gal-3 expression and induced cardiac dysfunction, cardiac and systemic inflammation, cardiac macrophage infiltration, and fibrosis. • Cardiac consequences of AKI were de
Highlights • In 2 different mouse models, AKI increased Gal-3 expression and induced cardiac dysfunction, cardiac and systemic inflammation, cardiac macrophage infiltration, and fibrosis. • Cardiac consequences of AKI were de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::948d78c938448798fca803e3c711b7ad
https://escholarship.org/uc/item/37f467d5
https://escholarship.org/uc/item/37f467d5
Autor:
Alain Cohen-Solal, Mathilde Prud’homme, Régine Merval, Claudio Passino, Giuseppe Vergaro, Claude Delcayre, Jane-Lise Samuel, Michele Emdin
Publikováno v:
Journal of the American College of Cardiology. 65:A798
Galectin-3 (Gal-3) is involved in cardiac inflammation, fibrogenesis and remodeling. Previous evidences show that Gal-3 interacts with aldosterone in promoting macrophage infiltration and vascular fibrosis, and that Gal-3 genetic and pharmacological