Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Mathew J. Sharman"'
Autor:
Mathew J Sharman, Seyyed H Moussavi Nik, Mengqi M Chen, Daniel Ong, Linda Wijaya, Simon M Laws, Kevin Taddei, Morgan Newman, Michael Lardelli, Ralph N Martins, Giuseppe Verdile
Publikováno v:
PLoS ONE, Vol 8, Iss 6, p e66235 (2013)
We investigated the guinea pig, Cavia porcellus, as a model for Alzheimer's disease (AD), both in terms of the conservation of genes involved in AD and the regulatory responses of these to a known AD risk factor - high cholesterol intake. Unlike rats
Externí odkaz:
https://doaj.org/article/596e4a984a77483ebce43491eef47eb2
Autor:
Ralph N. Martins, M P E Ng, Sam Gandy, Guanghou Shui, Markus R. Wenk, Wei Ling Florence Lim, Mathew J. Sharman, Andrew M. Jenner
Publikováno v:
Neuroscience. 169:109-115
Despite apolipoprotein E's important role in cholesterol transport and metabolism in the brain as well as its influence on Alzheimer's disease, the impact of the human APOE genotype on cholesterol metabolism in brain has not been fully examined. This
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::546802717a5c82bc744acd872eb1b913
https://doi.org/10.1016/j.neuroscience.2010.04.026
https://doi.org/10.1016/j.neuroscience.2010.04.026
Autor:
Mathew J. Sharman, Markus R. Wenk, Sam Gandy, Guanghou Shui, Andrew M. Jenner, Wei Ling Florence Lim, Mary Pei Ern Ng, Ralph N. Martins
Publikováno v:
Alzheimer's & Dementia. 6
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4836c19f110053666d8bedbba808430e
https://doi.org/10.1016/j.jalz.2010.05.835
https://doi.org/10.1016/j.jalz.2010.05.835
Autor:
Giuseppe Verdile, Daniel Ong, Simon M. Laws, Mathew J. Sharman, Kevin Taddei, Linda K. Wijaya, Michael Lardelli, Mengqi M. Chen, Morgan Newman, Ralph N. Martins, Seyyed Hani Moussavi Nik
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 6, p e66235 (2013)
PLoS ONE, Vol 8, Iss 6, p e66235 (2013)
We investigated the guinea pig, Cavia porcellus, as a model for Alzheimer's disease (AD), both in terms of the conservation of genes involved in AD and the regulatory responses of these to a known AD risk factor - high cholesterol intake. Unlike rats
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::acf46b30b40f5ffd01f387073e8cf865
https://doi.org/10.1371/journal.pone.0066235
https://doi.org/10.1371/journal.pone.0066235