Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Mathew C. Easterday"'
Autor:
Sarah Knox, Hong Ge, Brian D Dimitroff, Yi Ren, Katie A Howe, Andrew M Arsham, Mathew C Easterday, Thomas P Neufeld, Michael B O'Connor, Scott B Selleck
Publikováno v:
PLoS ONE, Vol 2, Iss 4, p e375 (2007)
Tuberous sclerosis complex is a dominant genetic disorder produced by mutations in either of two tumor suppressor genes, TSC1 and TSC2; it is characterized by hamartomatous tumors, and is associated with severe neurological and behavioral disturbance
Externí odkaz:
https://doaj.org/article/6c8842a87e6b4b098be9b378eb623f96
Autor:
Kenneth Ryan, Andreas P. Russ, Robert J. Levy, David J. Wehr, Jingtao You, Mathew C. Easterday
Publikováno v:
Human Gene Therapy. 15:842-855
Autor:
Daniel H. Geschwind, Ichiro Nakano, Dwain K. Irvin, Robert L. Jackson, Mehrnoosh Dianati, Andres A. Paucar, Babak Roobini, Alexey V. Terskikh, Joseph D. Dougherty, Mathew C. Easterday, Irving L. Weissman, Harley I. Kornblum, Jing Ou
Publikováno v:
Developmental Biology. 264:309-322
The identification of the genes regulating neural progenitor cell (NPC) functions is of great importance to developmental neuroscience and neural repair. Previously, we combined genetic subtraction and microarray analysis to identify genes enriched i
Autor:
Mathew C. Easterday, Joerg Dietrich
Publikováno v:
Trends in Neurosciences. 25:129-131
Stem Cells in the Mammalian Brain: the 4th Brain Research Interactive Symposium, at the 2001 Annual Conference of the Society for Neuroscience, San Diego, CA, USA from November 8–10 2001.
Autor:
Michael B. O'Connor, Mathew C. Easterday, Brian D. Dimitroff, Hong Ge, Yi Ren, Andrew M. Arsham, Thomas P. Neufeld, Sarah M. Knox, Scott B. Selleck, Katie A. Howe
Publikováno v:
PLoS ONE, Vol 2, Iss 4, p e375 (2007)
PloS one, vol 2, iss 4
PLoS ONE
PloS one, vol 2, iss 4
PLoS ONE
Tuberous sclerosis complex is a dominant genetic disorder produced by mutations in either of two tumor suppressor genes, TSC1 and TSC2; it is characterized by hamartomatous tumors, and is associated with severe neurological and behavioral disturbance
Publikováno v:
Human gene therapy. 15(9)
Congenital heart disease is the most prevalent cause of infant morbidity and mortality in developed countries. The mechanisms responsible for many specific types of congenital cardiac malformations are strongly associated with gene abnormalities. How
Autor:
Mathew C, Easterday, Joseph D, Dougherty, Robert L, Jackson, Jing, Ou, Ichiro, Nakano, Andres A, Paucar, Babak, Roobini, Mehrnoosh, Dianati, Dwain K, Irvin, Irving L, Weissman, Alexey V, Terskikh, Daniel H, Geschwind, Harley I, Kornblum
Publikováno v:
Developmental biology. 264(2)
The identification of the genes regulating neural progenitor cell (NPC) functions is of great importance to developmental neuroscience and neural repair. Previously, we combined genetic subtraction and microarray analysis to identify genes enriched i
Autor:
Mathew C. Easterday, Harley I. Kornblum, Irving L. Weissman, Heath J. Antoine, Zugen Chen, Stanley F. Nelson, Joseph D. Dougherty, Alexey V. Terskikh, Robert L. Jackson, Daniel H. Geschwind, Jing Ou
Publikováno v:
Neuron. 29(2)
Genetic mechanisms regulating CNS progenitor function and differentiation are not well understood. We have used microarrays derived from a representational difference analysis (RDA) subtraction in a heterogeneous stem cell culture system to systemati
Publikováno v:
Developmental neuroscience. 22(1-2)
The epidermal growth factor receptor family consists of four related tyrosine kinases: the epidermal growth factor receptor (EGF-R or ErbB), ErbB2, ErbB3, and ErbB4. These receptors are capable of extensive cross-activation upon the binding of their
Autor:
Daniel H. Geschwind, Leroy Hood, Alexey V. Terskikh, Irving L. Weissman, Harley I. Kornblum, Linheng Li, Mathew C. Easterday
Publikováno v:
Journal of Neurochemistry. 81:81-81
It is reasonable to propose that gene expression profiles of purified stem cells could give clues for the molecular mechanisms of stem cell behavior. We took advantage of cDNA subtraction to identify a set of genes selectively expressed in mouse adul