Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Matheus P.S. Magalhães-Gomes"'
Autor:
Marco A. M. Prado, Silvia Guatimosim, Vânia F. Prado, Matheus P.S. Magalhães-Gomes, Wallace Camargos, Jéssica Neves Andrade, Vanessa P. Teixeira, Priscila Aparecida Costa Valadão, Hermann A. Rodrigues, Lígia Araujo Naves, Walter L.G. Cavalcante, Marcia Gallaci, Cristina Guatimosim, Rubens Garcias
Publikováno v:
Scopus
Repositório Institucional da UNESP
Universidade Estadual Paulista (UNESP)
instacron:UNESP
Repositório Institucional da UNESP
Universidade Estadual Paulista (UNESP)
instacron:UNESP
Made available in DSpace on 2021-06-25T10:54:34Z (GMT). No. of bitstreams: 0 Previous issue date: 2021-04-15 Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES) Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c465c0a7fbca92b6b85e13f6f7c0772
Autor:
Ornela Kljakic, Matheus P.S. Magalhães-Gomes, Marco A. M. Prado, Priscila Aparecida Costa Valadão, Julliane V. Joviano-Santos, Vania F. Prado, Cristina Guatimosim, Leonardo Rossi de Oliveira
Publikováno v:
The FEBS journalReferences. 288(18)
Motoneurons (MNs) control muscle activity by releasing the neurotransmitter acetylcholine (ACh) at the level of neuromuscular junctions. ACh is packaged into synaptic vesicles by the vesicular ACh transporter (VAChT), and disruptions in its release c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ed8bb34e250b24e899f954319404b28
https://pubmed.ncbi.nlm.nih.gov/33730374
https://pubmed.ncbi.nlm.nih.gov/33730374
Autor:
Matheus P.S. Magalhães-Gomes, Jéssica Neves Andrade, Julliane V. Joviano-Santos, Fabiola M. Ribeiro, Cristina Guatimosim, José Carlos Nogueira, Giselle Foureaux, Priscila Aparecida Costa Valadão, Bárbara Campos de Aragão, Juan Carlos Tapia
Publikováno v:
European Journal of Neuroscience. 45:785-796
Involuntary choreiform movements are clinical hallmark of Huntington's disease, an autosomal dominant neurodegenerative disorder caused by an increased number of CAG trinucleotide repeats in the huntingtin gene. Involuntary movements start with an im
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d1effcae443eeb1925355f047a69ff87
https://doi.org/10.1111/ejn.13510
https://doi.org/10.1111/ejn.13510
Autor:
Cristina Guatimosim, Thatiane C.G. Machado, Danuza Montijo Diniz, Marina Ladeira, Marcus Vinicius Gomez, Julliane V. Joviano-Santos, Kivia B. Soares, André R. Massensini, Lorena F. Fernandes, Aline Silva de Miranda, Priscila Aparecida Costa Valadão, Matheus P.S. Magalhães-Gomes
Publikováno v:
Neuropeptides. 85:102111
Abnormal calcium influx and glutamatergic excitotoxicity have been extensively associated with neuronal death in Huntington's disease (HD), a genetic movement disorder. Currently, there is no effective treatment for this fatal condition. The neurotox
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8cb0393f0cc659c547a7ce9057057bae
https://doi.org/10.1016/j.npep.2020.102111
https://doi.org/10.1016/j.npep.2020.102111
Autor:
Diogo Guimarães, Cristina Guatimosim, Jéssica Neves Andrade, Silvia Guatimosim, Vânia F. Prado, Matheus P.S. Magalhães-Gomes, Grace S. Pereira, Patrícia Massara Martinelli, Luana Pereira Leite Schetino, Sydney K. Vaughan, Cristiane P. Bastos, Daisy Motta-Santos, Marco A. M. Prado, Candido Celso Coimbra, Gregorio Valdez
Publikováno v:
Anatomy and Cell Biology Publications
Congenital myasthenic syndromes (CMS) result from reduced cholinergic transmission at neuromuscular junctions (NMJs). While the etiology of CMS varies, the disease is characterized by muscle weakness. To date, it remains unknown if CMS causes long-te
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46b9e541109f202a0657228ef969183a
https://ir.lib.uwo.ca/anatomypub/143
https://ir.lib.uwo.ca/anatomypub/143
Autor:
Ana Flávia Machado Botelho, Jader S. Cruz, Artur Santos-Miranda, Silvia Guatimosim, Tatiane de Oliveira Barreto, Matheus P.S. Magalhães-Gomes, Priscila Aparecida Costa Valadão, Itamar Couto Guedes de Jesus, Julliane V. Joviano-Santos, Marília Martins Melo, Cristina Guatimosim, Jéssica Neves Andrade
Publikováno v:
The FEBS journal. 286(1)
Huntington's disease (HD) is a neurodegenerative genetic disorder. Although described as a brain pathology, there is evidence suggesting that defects in other systems can contribute to disease progression. In line with this, cardiovascular defects ar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::683552053b32705a9c2d361f15e54d35
https://pubmed.ncbi.nlm.nih.gov/30451379
https://pubmed.ncbi.nlm.nih.gov/30451379
Autor:
Silvia Guatimosim, Itamar Couto Guedes de Jesus, Luísa Helena Figueiredo Peixoto, Julliane V. Joviano-Santos, Fabiola M. Ribeiro, Priscila Aparecida Costa Valadão, Giselle Foureaux, Rayan Silva de Paula, Matheus P.S. Magalhães-Gomes, Thatiane C.G. Machado, Cristina Guatimosim, Erika Cristina Jorge, Juan Carlos Tapia, José Carlos Nogueira, Bárbara Campos de Aragão, Jéssica Neves Andrade, Julia Meireles Nogueira
Publikováno v:
ASN NEURO
ASN Neuro, Vol 11 (2019)
ASN Neuro, Vol 11 (2019)
Huntington’s disease (HD) is a disorder characterized by chronic involuntary movements, dementia, and psychiatric symptoms. It is caused by a mutation in the gene that encodes for huntingtin protein (HTT), leading to the formation of mutant protein
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eeea6f89fd7cd060de99eb50ef5e3a91
https://doi.org/10.1177/1759091419886212
https://doi.org/10.1177/1759091419886212