Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Mascha S. Recks"'
Autor:
Marie Wunsch, Wenji Zhang, Jodi Hanson, Richard Caspell, Alexey Y. Karulin, Mascha S. Recks, Stefanie Kuerten, Srividya Sundararaman, Paul V. Lehmann
Publikováno v:
Viruses, Vol 7, Iss 8, Pp 4414-4437 (2015)
Most humans become infected with human cytomegalovirus (HCMV). Typically, the immune system controls the infection, but the virus persists and can reactivate in states of immunodeficiency. While substantial information is available on the contributio
Externí odkaz:
https://doaj.org/article/51e0bee4a5e54b91a2acc027775c046b
Autor:
Ramu A. Subbramanian, Wenji Zhang, Edith Karacsony, Mascha S. Recks, Helena Batoulis, Stefanie Kuerten, Paul V. Lehmann
Publikováno v:
Cells, Vol 1, Iss 3, Pp 409-427 (2012)
T cell monitoring is increasingly performed using cryopreserved PBMC. It has been suggested that resting of PBMC after thawing, that is, culturing them overnight in test medium, produces higher antigen-induced spot counts in ELISPOT assays. To evalua
Externí odkaz:
https://doaj.org/article/5742bfec50c54046a5479b7f20f1a06d
Publikováno v:
PLoS ONE, Vol 11, Iss 5, p e0155197 (2016)
[This corrects the article DOI: 10.1371/journal.pone.0144847.].
Externí odkaz:
https://doaj.org/article/970274d733df45a58aff5485a8d3b6ba
Autor:
Franziska van der Broeck, Nicole Wagner, Nicolai B. Grether, Michael Schroeter, Süleyman Ergün, Stefanie Kuerten, Alla Ganscher, Erik Henke, Mascha S. Recks
Publikováno v:
Clinical Immunology. 159:93-106
Here we studied the autoantibody specificity elicited by proteolipid protein (PLP) in MP4-induced experimental autoimmune encephalomyelitis, a mouse model of multiple sclerosis (MS). In C57BL/6 (B6) mice, antibodies were induced by immunization with
Autor:
Mascha S. Recks, Stefanie Kuerten, Eva R. Stormanns, Klaus Addicks, Jonas Bader, Stefan Arnhold
Publikováno v:
Clinical Immunology. 149:32-45
Studies of MS histopathology are largely dependent on suitable animal models. While light microscopic analysis gives an overview of tissue pathology, it falls short in evaluating detailed changes in nerve fiber morphology. The ultrastructural data pr
Autor:
Stefanie Kuerten, Richard O. Williams, Helena Batoulis, F. O. Holland, F. Thomalla, Mascha S. Recks
Summary In various autoimmune diseases, anti-tumour necrosis factor (TNF)-α treatment has been shown to reduce both clinical disease severity and T helper type 1 (Th1)1/Th17 responses. In experimental autoimmune encephalomyelitis (EAE), however, the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::24dd1aa92c534007b322308c3b767b99
https://doi.org/10.1111/cei.12209
https://doi.org/10.1111/cei.12209
Autor:
Christian Kerkloh, Mascha S. Recks, Achim Schickel, Klaus Addicks, Nancy H. Ruddle, Stefanie Kuerten, Paul V. Lehmann
Publikováno v:
Acta Neuropathologica. 124:861-873
While the role of T cells has been studied extensively in multiple sclerosis (MS), the pathogenic contribution of B cells has only recently attracted major attention, when it was shown that B cell aggregates can develop in the meninges of a subset of
Publikováno v:
APMIS. 119:819-830
Multiple sclerosis (MS) is an autoimmune disorder of the CNS. Different subtypes of the disease have been noted, and characterized by distinct clinical courses and histopathologic manifestations. The most intensively studied animal model of MS, exper
Publikováno v:
Neuroscience Letters. 494:227-231
In the present study, we demonstrate that the histopathologic features of myelin oligodendrocyte glycoprotein (MOG) peptide 35-55-induced experimental autoimmune encephalomyelitis (EAE) in C57BL/6 mice closely mirror the hallmarks of MS pathology. On
Autor:
J. Bader, Stefanie Kuerten, C. C. Kaiser, K. Addicks, Mascha S. Recks, Michael Schroeter, Gereon R. Fink
Publikováno v:
Fortschritte der Neurologie · Psychiatrie. 79:161-170
In spite of tremendous scientific effort, the mechanisms underlying multiple sclerosis (MS) still remain to be elucidated. The prevalent pathogenetic concept adheres to the assumption of a strict hierarchical sequence of the triad inflammation, demye