Zobrazeno 1 - 10
of 442
pro vyhledávání: '"Masayoshi Takeuchi"'
Publikováno v:
International Journal of Molecular Sciences, Vol 25, Iss 13, p 7409 (2024)
Advanced glycation end-products (AGEs) form through non-enzymatic glycation of various proteins. Optic nerve degeneration is a frequent complication of diabetes, and retinal AGE accumulation is strongly linked to the development of diabetic retinopat
Externí odkaz:
https://doaj.org/article/15a62359eb9c414fbb6656c3940c4765
Publikováno v:
Biomolecules, Vol 14, Iss 2, p 202 (2024)
Advanced glycation end-products (AGEs) have recently been implicated in the onset/progression of lifestyle-related diseases (LSRDs); therefore, the suppression of AGE-induced effects may be used in both the prevention and treatment of these diseases.
Externí odkaz:
https://doaj.org/article/cdbb1a6d0eb540d9b5dc6cc380ae9132
Publikováno v:
Cells, Vol 12, Iss 24, p 2838 (2023)
Advanced glycation end-products (AGEs), formed through glyceraldehyde (GA) as an intermediate in non-enzymatic reactions with intracellular proteins, are cytotoxic and have been implicated in the pathogenesis of various diseases. Despite their signif
Externí odkaz:
https://doaj.org/article/14ba11bbbf7642c68b82c91671b79c6a
Autor:
Masao Jinno, Ryoji Nagai, Masayoshi Takeuchi, Aiko Watanabe, Koji Teruya, Hikari Sugawa, Naohisa Hatakeyama, Yuichi Jinno
Publikováno v:
Reproductive Biology and Endocrinology, Vol 19, Iss 1, Pp 1-14 (2021)
Abstract Background Advanced glycation end-products (AGE), which accumulate with insulin resistance and aging, impair folliculogenesis and may decrease endometrial receptivity. Hishi (Trapa bispinosa Roxb.) extract, a safe herbal medicine, strongly i
Externí odkaz:
https://doaj.org/article/d0e5a313905e4b2bbb2ac0704e5b1796
Publikováno v:
Diabetology & Metabolic Syndrome, Vol 13, Iss 1, Pp 1-8 (2021)
Abstract Background 1,5-anhydroglucitol is a reduction product of 1,5-anhydrofructose. Circulating 1,5-anhydroglucitol is usually excreted by the kidneys and is reabsorbed via sodium-glucose co-transporter 4 in the renal tubules. In patients on hemod
Externí odkaz:
https://doaj.org/article/d639423bef2f4bb98b98518c15c17a7f
Publikováno v:
Frontiers in Pharmacology, Vol 13 (2022)
Diabetes mellitus (DM) has been identified as a risk factor for the onset and progression of Alzheimer’s disease (AD). In our previous study, we demonstrated that glyceraldehyde (GA)-derived toxic advanced glycation end-products (toxic AGEs, TAGE)
Externí odkaz:
https://doaj.org/article/dd31d2acdc744f1ab222b8603ed4e58c
Publikováno v:
Antioxidants, Vol 12, Iss 3, p 748 (2023)
The repeated excessive intake of sugar, a factor that contributes to the onset of nonalcoholic fatty liver disease (NAFLD) and its progression to the chronic form of nonalcoholic steatohepatitis (NASH), markedly increases the hepatocyte content of gl
Externí odkaz:
https://doaj.org/article/1efd386b2b734fafac279e4d4780cf85
Autor:
Jun-ichi Takino, Takuma Sato, Takumi Kanetaka, Kasumi Okihara, Kentaro Nagamine, Masayoshi Takeuchi, Takamitsu Hori
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-10 (2021)
Abstract Advanced glycation end-products (AGEs) are formed by the non-enzymatic reaction of sugars and proteins. Among the AGEs, glyceraldehyde-derived toxic AGEs (TAGE) are associated with various diseases, including diabetic complications such as d
Externí odkaz:
https://doaj.org/article/46e40f30d5db4e96b709246f60f79f9b
Autor:
Masayoshi Takeuchi
Publikováno v:
Diabetology & Metabolic Syndrome, Vol 12, Iss 1, Pp 1-12 (2020)
Abstract Background The habitual excessive intake of sugar (i.e., sucrose and high-fructose corn syrup), which has been implicated in the onset of diabetes mellitus, induces excessive production of glyceraldehyde, a metabolite produced during glucose
Externí odkaz:
https://doaj.org/article/a480b4b0a9814671b57c71126ca48fae
Publikováno v:
Diabetology & Metabolic Syndrome, Vol 12, Iss 1, Pp 1-8 (2020)
Abstract Background Sarcopenia is a progressive condition that is characterized by decreases in skeletal muscle mass and function. Although sarcopenia is associated with lifestyle-related diseases (LSRD), the mechanisms underlying cell death in myobl
Externí odkaz:
https://doaj.org/article/998c4f264ef24bd19be53d439effd37c