Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Masako Kuga"'
Autor:
Misato Kobayashi, Tamio Ohno, Natsuko Hada, Masato Fujiyoshi, Masako Kuga, Masahiko Nishimura, Atsushi Murai, Fumihiko Horio
Publikováno v:
Journal of Lipid Research, Vol 51, Iss 12, Pp 3463-3469 (2010)
Each abdominal fat depot, such as mesenteric or epididymal, differently contributes to the development of insulin resistance. The aim of this study was to identify the genetic regions that contribute to fat accumulation in epididymal/mesenteric fat a
Externí odkaz:
https://doaj.org/article/cff98f59de314539930ca5d6ea6b2ab5
Autor:
Yuki Miyasaka, Takeshi Kobayashi, Naoya Gotoh, Masako Kuga, Misato Kobayashi, Fumihiko Horio, Katsunori Hashimoto, Tsutomu Kawabe, Tamio Ohno
Publikováno v:
Mammalian Genome. 34:32-43
Autor:
Miyoko Matsushima, Yoshiaki Kikkawa, Yuki Miyasaka, Masahide Takahashi, Masashi Mizuno, Tsutomu Kawabe, Masako Kuga, Tamio Ohno, Kaori Ushida
Publikováno v:
Experimental Animals
Malaria is caused by Plasmodium parasites and is one of the most life-threatening infectious diseases in humans. Infection can result in severe complications such as cerebral malaria, acute lung injury/acute respiratory distress syndrome, and acute r
Autor:
Masato Fujiyoshi, Masahiko Nishimura, Atsushi Murai, Misato Kobayashi, Fumihiko Horio, Tamio Ohno, Masako Kuga, Natsuko Hada
Publikováno v:
Journal of Lipid Research, Vol 51, Iss 12, Pp 3463-3469 (2010)
Each abdominal fat depot, such as mesenteric or epididymal, differently contributes to the development of insulin resistance. The aim of this study was to identify the genetic regions that contribute to fat accumulation in epididymal/mesenteric fat a
Autor:
Masato Fujiyoshi, Masako Kuga, Fumihiko Horio, Masahiko Nishimura, Akira Ishikawa, Misato Kobayashi, Tamio Ohno, Natsuko Hada, Shizufumi Ebihara
Publikováno v:
Physiological genomics. 35(1)
The SMXA-5 recombinant inbred strain, which was established from nondiabetic parental SM/J and A/J mice, develops diabetic phenotypes such as impaired glucose tolerance. The combination of diabetogenic genes in the SM/J and A/J genomes impairs glucos
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