Zobrazeno 1 - 10
of 57
pro vyhledávání: '"Masakazu Okamoto"'
Autor:
Kenichiro Saito, Yusuke Inagaki, Yoshinobu Uchihara, Masakazu Okamoto, Yuki Nishimura, Akihito Kawai, Tatsuro Sugino, Kensuke Okamura, Munehiro Ogawa, Akira Kido, Yasuhito Tanaka
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-10 (2024)
Abstract Allogeneic bone grafts are used to treat bone defects in orthopedic surgery, but the osteogenic potential of artificial bones remains a challenge. In this study, we developed a β-tricalcium phosphate (β-TCP) formulation containing MgO, ZnO
Externí odkaz:
https://doaj.org/article/d8a1686c82c54f189c8ebcd4cc235b8e
Publikováno v:
2022 IEEE 19th Annual Consumer Communications & Networking Conference (CCNC).
Autor:
Everett Rand Sutherland, Masakazu Okamoto, Yi Yeong Jeong, Joanne Domenico, Sang-Ha Kim, Erwin W. Gelfand, Katsuyuki Takeda, Naohiro Terada, Fangkun Ning, Yoshiki Shiraishi, Shigeru Ashino
Publikováno v:
Stem Cells Translational Medicine
All-trans retinoic acid (ATRA) or mesenchymal stem cells (MSCs) have been shown to promote lung tissue regeneration in animal models of emphysema. However, the reparative effects of the combination of the two and the role of p70S6 kinase-1 (p70S6k1)
Autor:
Yi Jia, Michaela Schedel, Masakazu Okamoto, Shigeru Ashino, Anna Lluis, Erwin W. Gelfand, Katsuyuki Takeda, Anthony Joetham, Azzeddine Dakhama
Publikováno v:
The Journal of Immunology. 193:2238-2247
Glucocorticoid-induced TNFR family–related protein (GITR)–mediated activation of JNK was shown to regulate the suppressive activity of CD4+CD25+ naturally occurring T regulatory cells (nTregs) in wild-type (WT) hosts. In this study, CD4+CD25+ T c
Autor:
Azzeddine Dakhama, Erwin W. Gelfand, Junyan Han, Masakazu Okamoto, Wanjiang Zeng, Meiqin Wang, Yi Jia, Yoshiki Shiraishi, Katsuyuki Takeda
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology. 51:143-154
Respiratory syncytial virus (RSV) is the leading cause of lower respiratory tract illnesses in infants worldwide. Both RSV-G and RSV-F glycoproteins play pathogenic roles during infection with RSV. The objective of this study was to compare the effec
Autor:
Shinichi Nakano, Toshiyuki Kita, Masakazu Okamoto, Yasushi Kaneta, Akifumi Hoshino, Yoichi Nagao
Publikováno v:
IEICE Transactions on Fundamentals of Electronics, Communications and Computer Sciences. :554-561
Autor:
Toru Hara, Naozumi Hashimoto, Yoshinori Hasegawa, Tamio Ohno, Kaoru Shimokata, Tsutomu Kawabe, Miyoko Matsushima, Masahiko Nishimura, Kazuyoshi Imaizumi, Masakazu Okamoto
Publikováno v:
Immunogenetics. 65:17-24
Asthma is regarded as a multifactorial inflammatory disorder arising as a result of inappropriate immune responses in genetically susceptible individuals to common environmental antigens. However, the precise molecular basis is unknown. To identify g
Autor:
Masakazu Okamoto, Katsuyuki Takeda, Michaela Schedel, Anthony Joetham, Hiroshi Ohnishi, Joanne Domenico, Erwin W. Gelfand, Azzeddine Dakhama
Publikováno v:
Journal of Biological Chemistry. 287:17100-17108
Naturally occurring Foxp3(+)CD4(+)CD25(+) T regulatory cell (nTreg)-mediated suppression of lung allergic responses is abrogated following ligation of glucocorticoid-induced tumor necrosis receptor (GITR) family-related protein. In vitro stimulation
Autor:
Erwin W. Gelfand, Yoshiki Shiraishi, Stephen Tomlinson, Viviana P. Ferreira, V. Michael Holers, Michael K. Pangburn, Joshua M. Thurman, Katsuyuki Takeda, Masakazu Okamoto, Claudio Cortes
Publikováno v:
The Journal of Immunology. 188:661-667
Activation of the alternative pathway of complement plays a critical role in the development of allergen-induced airway hyperresponsiveness (AHR) and inflammation in mice. Endogenous factor H, a potent inhibitor of the alternative pathway, is increas
Autor:
Azzeddine Dakhama, Michaela Schedel, Katsuyuki Takeda, Hiroshi Ohnishi, Anthony Joetham, Erwin W. Gelfand, Masakazu Okamoto
Publikováno v:
The Journal of Immunology. 186:113-120
Naturally occurring CD4+CD25+Foxp3+ T regulatory cells (nTregs) regulate lung allergic responses through production of IL-10 and TGF-β. nTregs from CD8−/− mice failed to suppress lung allergic responses and were characterized by reduced levels o