Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Marzieh Bardestani"'
Autor:
Javad Dehbozorgian, Sanaz Safaei, Maryam Abdollahi, Narges Rezaei, Sezaneh Haghpanah, Marzieh Bardestani, Fatemeh Hassani, Naser Honar, Mehran Karimi, Masoomeh Esmaeilzadeh
Publikováno v:
Iranian Red Crescent Medical Journal
Background: Hepcidin is a key regulator of iron absorption in humans. It is mainly affected by hypoxia and iron stores. Objectives: The current study aimed to determine the correlation between serum hepcidin and ferritin levels in patients with Thala
Autor:
Sezaneh Haghpanah, Gholamhossein Ajami, Sara Matin, Zahra Majd, Marzieh Bardestani, Mehran Karimi, Hamid Namazi
Publikováno v:
Iranian Red Crescent Medical Journal
Background: Iron-loaded cardiac complication is the essential cause of mortality in patients with thalassemia. Early detection and treatment of cardiac over-load can reduce mortality. Objectives: The current study aimed to evaluate the relationship b
Autor:
Amin Abolhasani Foroughi, Masoume Nazeri, Mehran Karimi, Sezaneh Haghpanah, Roghieh Ghaffari, Marzieh Bardestani, Hosein Ghaffari
Publikováno v:
Iranian Red Crescent Medical Journal
Background: In patients with thalassemia, chronic anemia causes bone marrow expansion and consequently skeletal manifestation in spine, skull, face and rib bones. Objectives: We aimed to compare chest radiographic findings and facial bone deformity i
Autor:
Sezaneh Haghpanah, Azin Hamidi, Mehran Karimi, Marzieh Bardestani, Maryam Khavari, Mohammad Hadi Bagheri, Razieh Hantooshzadeh
Publikováno v:
Iranian Red Crescent Medical Journal
Background: Recent studies regarding the effect of hydroxyurea (HU) in thalassemia have revealed favorable effects on the reduction of ineffective erythropoiesis. Objectives: The aim of the current study was to evaluate whether or not HU can have an
Autor:
Roza Kamfiroozi, Samir Silavizadeh, Marzieh Bardestani, Mohammadreza Bordbar, Sezaneh Haghpanah, Mehran Karimi
Publikováno v:
Iranian Red Crescent Medical Journal
Background: β-Thalassemia is an inherited hemoglobin disorder caused by defective synthesis of s-globin chains. Hemoglobin (Hb) F induction is a possible therapeutic approach which can partially compensate for α and non-α globin chains imbalance.