Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Maryam S. Seyedsadr"'
Autor:
Maryam S. Seyedsadr, Oliver Weinmann, Ana Amorim, Benjamin V. Ineichen, Matteo Egger, Javad Mirnajafi-Zadeh, Burkhard Becher, Mohammad Javan, Martin E. Schwab
Publikováno v:
Neurobiology of Disease, Vol 124, Iss , Pp 189-201 (2019)
Multiple sclerosis is an inflammatory disease of the central nervous system (CNS) in which multiple sites of blood-brain barrier (BBB) disruption, focal inflammation, demyelination and tissue destruction are the hallmarks. Here we show that sphingosi
Externí odkaz:
https://doaj.org/article/5e4c82486955409889c724d5c9e6f988
Autor:
Daniel Hwang, Larissa Lumi Watanabe Ishikawa, Maryam S. Seyedsadr, Elisabeth Mari, Ezgi Kasimoglu, Ziver Sahin, Alexandra Boehm, Soohwa Jang, Javad Rasouli, Courtney Vaccaro, Michael Gonzalez, Hakon Hakonarson, Abdolmohamad Rostami, Guang-Xian Zhang, Bogoljub Ciric
Publikováno v:
Blood advances. 6(23)
GATA-binding factor 1 (GATA1) is a transcription factor that governs the development and function of multiple hematopoietic cell lineages. GATA1 is expressed in hematopoietic stem and progenitor cells (HSPCs) and is essential for erythroid lineage co
Autor:
Daniel Hwang, Maryam S. Seyedsadr, Larissa Lumi Watanabe Ishikawa, Alexandra Boehm, Ziver Sahin, Giacomo Casella, Soohwa Jang, Michael V. Gonzalez, James P. Garifallou, Hakon Hakonarson, Weifeng Zhang, Dan Xiao, Abdolmohamad Rostami, Guang-Xian Zhang, Bogoljub Ciric
Publikováno v:
Proceedings of the National Academy of Sciences. 119
The receptor for colony stimulating factor 1 (CSF-1R) is important for the survival and function of myeloid cells that mediate pathology during experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS). CSF-1 and IL-
Autor:
Sandra Kapitza, Julia Kaiser, Nicolas Good, Patricia S. Plattner, Christiane Bleul, Michael Linnebank, Roland Martin, Marc P. Schneider, Björn Zörner, Maryam S. Seyedsadr, Benjamin V. Ineichen, Martin E. Schwab
Publikováno v:
Acta Neuropathologica. 134:423-440
Two hallmarks of chronic multiple sclerosis lesions are the absence of significant spontaneous remyelination and primary as well as secondary neurodegeneration. Both characteristics may be influenced by the presence of inhibitory factors preventing m
Autor:
Dianne P. Figlewicz, Martin E. Schwab, Nicolas Good, Maryam S. Seyedsadr, Martin Hlavica, Aro Delparente, Andrin Good, Benjamin V. Ineichen, Patricia S. Plattner
Publikováno v:
Neuroscience Letters. 648:41-46
One main pathological hallmark of multiple sclerosis (MS) is demyelination. Novel therapies which enhance myelin repair are urgently needed. Insulin and insulin-like growth factor 1 (IGF-1) have strong functional relationships. Here, we addressed the
Gpr17, a Player in Lysolecithin-Induced Demyelination, Oligodendrocyte Survival, and Differentiation
Publikováno v:
The Journal of Neuroscience. 37:2273-2275
Multiple sclerosis (MS) is a neuro-inflammatory disease of the CNS. The symptoms start with the entrance of immune cells to the CNS, which causes inflammation, demyelination, and axonal degeneration. After demyelination, oligodendrocyte progenitor ce
Autor:
Maryam S. Seyedsadr, Sepideh Dashti
Publikováno v:
Journal of Human Genetics and Genomics.
Autor:
Javad Mirnajafi-Zadeh, Benjamin V. Ineichen, Martin E. Schwab, Oliver Weinmann, Matteo Egger, Mohammad Javan, Ana Amorim, Burkhard Becher, Maryam S. Seyedsadr
Publikováno v:
Neurobiology of Disease, Vol 124, Iss, Pp 189-201 (2019)
Multiple sclerosis is an inflammatory disease of the central nervous system (CNS) in which multiple sites of blood-brain barrier (BBB) disruption, focal inflammation, demyelination and tissue destruction are the hallmarks. Here we show that sphingosi