Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Martina Mareková"'
Publikováno v:
Acta Medica, Vol 44, Iss 1, Pp 7-13 (2001)
Cultivation with 4-8 μmol/l hypericin (specific proteinkinase C inhibitor) activated by light induced high inhibition of the rate of HL-60 cell growth. When hypericin treated cells were not exposed to light growth inhibition was insignificant. Culti
Externí odkaz:
https://doaj.org/article/7803a048a6764bd4bfca360203059379
Publikováno v:
Acta Medica, Vol 43, Iss 2, Pp 69-73 (2000)
TP-53 deficient cells of human leukaemia HL-60 die by massive apoptosis after treatment by high (50-100 nmol/l) doses of DNA damaging agent Idarubicin, regardless of the cell-cycle phase, in which they are affected. In contrary, after relatively low
Externí odkaz:
https://doaj.org/article/2dce0a6542ff4fe3a617d2d747816808
Autor:
Josef Marek, Stanislav Nemecek, Martina Mareková, Jan Cap, Miroslav Červinka, Emil Rudolf, Jaroslav Cerman
Publikováno v:
Annals of the New York Academy of Sciences. 1010:520-524
The aim of the study was to investigate the mechanism of action of somatostatin analogues (SSA), ionizing radiation, and their combination on pituitary adenoma cells with special emphasis on proliferative and apoptotic activity. In the 14 GH-secretin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::77e65755b0e4b3f3e6d7eb070b5c3406
https://doi.org/10.1196/annals.1299.096
https://doi.org/10.1196/annals.1299.096
Publikováno v:
Radiation and Environmental Biophysics. 42:193-199
Most cell lines that lack functional p53 protein are arrested in the G(2) phase of the cell cycle due to DNA damage. It was previously found that the human promyelocyte leukemia cells HL-60 (TP53 negative) that had been exposed to ionizing radiation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5b712f803c45cde853253c1a310bd6e
https://doi.org/10.1007/s00411-003-0209-4
https://doi.org/10.1007/s00411-003-0209-4
Publikováno v:
Journal of Clinical Neuroscience. 10:444-448
With the aim to investigate histopathological changes and proliferative and apoptotic activity in GH-secreting adenomas we compared 14 cases pre-treated with somatostatin analogues before surgery with a reference group of 17 un-pretreated ones. Besid
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46e90f3aa4bb38bba431da94150e1228
https://doi.org/10.1016/s0967-5868(03)00072-9
https://doi.org/10.1016/s0967-5868(03)00072-9
Publikováno v:
Acta Medica, Vol 44, Iss 1, Pp 7-13 (2001)
Scopus-Elsevier
Scopus-Elsevier
Cultivation with 4-8 μmol/l hypericin (specific proteinkinase C inhibitor) activated by light induced high inhibition of the rate of HL-60 cell growth. When hypericin treated cells were not exposed to light growth inhibition was insignificant. Culti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2df5a26dfd07b0707b53ab6dc924085b
https://doi.org/10.14712/18059694.2019.80
https://doi.org/10.14712/18059694.2019.80
Publikováno v:
Endocrine journal. 50(5)
Ionising radiation is used for the treatment of pituitary tumours as fractionated radiotherapy, where the total dose reaching the tumour area is in the range of 40-50 Gy, or during stereotactic radiosurgery, where the total dose reaching the tumour a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5152ac8d58833c6e26fb9ef248fb5fe4
https://pubmed.ncbi.nlm.nih.gov/14614219
https://pubmed.ncbi.nlm.nih.gov/14614219
Publikováno v:
Acta Medica, Vol 43, Iss 2, Pp 69-73 (2000)
TP-53 deficient cells of human leukaemia HL-60 die by massive apoptosis after treatment by high (50-100 nmol/l) doses of DNA damaging agent Idarubicin, regardless of the cell-cycle phase, in which they are affected. In contrary, after relatively low
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::849a919988983ce5c506150477b616ca
https://pubmed.ncbi.nlm.nih.gov/10953380
https://pubmed.ncbi.nlm.nih.gov/10953380
Publikováno v:
Radiation & Environmental Biophysics; Oct2003, Vol. 42 Issue 3, p193, 7p