Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Martina, Muckenthaler"'
Autor:
Mariateresa Pettinato, Alessandro Dulja, Silvia Colucci, Valeria Furiosi, Franca Fette, Andrea Steinbicker, Martina Muckenthaler, Antonella Nai, Alessia Pagani, Laura Silvestri
Publikováno v:
HemaSphere, Vol 7, p e680541c (2023)
Externí odkaz:
https://doaj.org/article/e3a9f67393ff469ead38d7036099febb
Publikováno v:
HemaSphere, Vol 7, p e032586a (2023)
Externí odkaz:
https://doaj.org/article/7b25b9855e2645df91eabbc364c9972a
Autor:
Jana Samarin, Piotr Fabrowski, Roman Kurilov, Hana Nuskova, Johanna Hummel-Eisenbeiss, Hannelore Pink, Nan Li, Vivienn Weru, Hamed Alborzinia, Umut Yildiz, Laura Grob, Minerva Taubert, Marie Czech, Michael Morgen, Christina Brandstädter, Katja Becker, Lianghao Mao, Ashok Kumar Jayavelu, Angela Goncalves, Ulrike Uhrig, Jeanette Seiler, Yanhong Lyu, Sven Diederichs, Ursula Klingmüller, Martina Muckenthaler, Annette Kopp-Schneider, Aurelio Teleman, Aubry K. Miller, Nikolas Gunkel
Publikováno v:
Redox Biology, Vol 62, Iss , Pp 102639- (2023)
Despite a strong rationale for why cancer cells are susceptible to redox-targeting drugs, such drugs often face tumor resistance or dose-limiting toxicity in preclinical and clinical studies. An important reason is the lack of specific biomarkers to
Externí odkaz:
https://doaj.org/article/43f98fcf2a844645aeeecc65e57523ec
Autor:
Achille Iolascon, Lucia De Franceschi, Martina Muckenthaler, Ali Taher, David Rees, Mariane de Montalembert, Stefano Rivella, Androulla Eleftheriou, Maria Domenica Cappellini
Publikováno v:
HemaSphere, Vol 3, Iss 3 (2019)
Abstract. The inherited disorders of hemoglobin, which include sickle cell disease and thalassemias, are the most common and widespread distributed monogenic disorders. Due to a selective advantage in malaria regions, these hemoglobin defects are par
Externí odkaz:
https://doaj.org/article/34c8ec1b1d874770861db774d1bcd32b
Autor:
Stefanie Junk, Elke Schaeffeler, Martin Zimmermann, Anja Möricke, Rita Beier, Peter Schütte, Birthe Fedders, Julia Alten, Laura Hinze, Norman Klein, Andreas Kulozik, Martina Muckenthaler, Rolf Koehler, Arndt Borkhardt, Jayaram Vijayakrishnan, David Ellinghaus, Michael Forster, Andre Franke, Astrid Wintering, Christian Kratz, Martin Schrappe, Matthias Schwab, Richard Houlston, Gunnar Cario, Martin Stanulla
Background Characterization of clinical phenotypes in context with tumor and host genomic information can aid in the development of more effective and less toxic risk-adapted and targeted treatment strategies. To analyze the impact of therapy-related
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::37dea6150bdd3c4dad2748b61aacb46e
https://zenodo.org/record/7539533
https://zenodo.org/record/7539533
Autor:
Walee Chamulitrat, Chutima Jansakun, Warangkana Chunglok, Sandro Altamura, Martina Muckenthaler, Simone Staffer, Sabine Tuma-Kellner, Uta Merle
Publikováno v:
39. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber.
Publikováno v:
HemaSphere, Vol 2, Iss 1 (2018)
Externí odkaz:
https://doaj.org/article/4c4e6820af6647c580b3dd3f0d70b86f
Publikováno v:
Blood. 140(19)
Anemia of inflammation (AI) is a highly prevalent comorbidity in patients affected by chronic inflammatory disorders, such as chronic kidney disease, inflammatory bowel disease, or cancer, that negatively affect disease outcome and quality of life. T
Autor:
LukasJohannes Klement, Chutima Jansakun, Sabine Tuma-Keller, Simone Staffer, Sandro Altamura, Martina Muckenthaler, Uta Merle, Walee Chamulitrat
Publikováno v:
Zeitschrift für Gastroenterologie.
Autor:
Chutima, Jansakun, Warangkana, Chunglok, Sandro, Altamura, Martina, Muckenthaler, Simone, Staffer, Sabine, Tuma-Kellner, Uta, Merle, Walee, Chamulitrat
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1869:166590
Polymorphisms of phospholipase A2VIA (iPLA2β or PLA2G6) are associated with body weights and blood C-reactive protein. The role of iPLA2β/PLA2G6 in non-alcoholic steatohepatitis (NASH) is still elusive because female iPla2β-null mice showed attenu