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Publikováno v:
Leukemialymphoma. 63(3)
Cancer cells reprogram their metabolism to maintain sustained proliferation, which creates unique metabolic dependencies between malignant and healthy cells that can be exploited for therapy. In acute myeloid leukemia (AML), mitochondrial inhibitors
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b827e2e6193974758b9800514cbb20a4
https://pubmed.ncbi.nlm.nih.gov/34704521
https://pubmed.ncbi.nlm.nih.gov/34704521
