Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Martin F. Bubik"'
Autor:
Egon Koch, Olivier Potterat, Jacqueline Bräutigam, Clemens Erdelmeier, Martin F. Bubik, Andreas Hensel, Maria Teresa Faleschini, Elisabeth A. Willer, Iris Bischoff, S Fuchs, Milena Bauhart, Stefan Zahler, Maria De Mieri, Robert Fürst, Matthias Hamburger
Publikováno v:
Planta medica. 83(8)
The hawthorn (Crataegus spp.) extract WS 1442 is used against mild forms of chronic heart failure. This disease is associated with endothelial barrier dysfunction and edema formation. We have recently shown that WS 1442 protects against this dysfunct
Autor:
Fritz Krombach, Elisabeth A. Willer, Stefan Zahler, Hermann Ammer, Martin F. Bubik, Angelika M. Vollmar, Guido Jürgenliemk, Peter Bihari, Robert Fürst
Publikováno v:
Journal of molecular and cellular cardiology. 52(1)
Endothelial hyperpermeability followed by edema formation is a hallmark of many severe disorders. Effective drugs directly targeting endothelial barrier function are widely lacking. We hypothesized that the hawthorn (Crataegus spp.) extract WS® 1442
Autor:
H Ammer, Robert Fürst, Martin F. Bubik, Angelika M. Vollmar, Stefan Zahler, Elisabeth A. Willer
Publikováno v:
Planta Medica. 76
Autor:
P Bihari, H Ammer, Guido Jürgenliemk, Martin F. Bubik, Angelika M. Vollmar, F Krombach, Stefan Zahler, Robert Fürst
Publikováno v:
Planta Medica. 76
Autor:
Martin F. Bubik, Bettina A. Mayer, Angelika M. Vollmar, Fritz Krombach, Peter Bihari, Robert Fürst, Alexander G. Khandoga, Florian Hoffmann, Markus Rehberg, Stefan Zahler
Publikováno v:
Molecular pharmacology. 74(1)
Endothelial barrier dysfunction is a hallmark of many severe pathologies, including sepsis or atherosclerosis. The cardiovascular hormone atrial natriuretic peptide (ANP) has increasingly been suggested to counteract endothelial leakage. Surprisingly
Autor:
Alexandra K. Kiemer, Martin F. Bubik, Robert Fürst, Angelika M. Vollmar, Stefan Zahler, Hanna M. Eilken, Timm Schroeder
Publikováno v:
FASEB J. 21, 74-80 (2007)
Glucocorticoids are well-established anti-inflammatory drugs thought to mainly act by inhibition of proinflammatory transcription factors like NF-{kappa}B. In recent years, however, transcription factor-independent mechanisms of glucocorticoid action
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ba9785a8045415374f80772ba9de5a8d
https://push-zb.helmholtz-muenchen.de/frontdoor.php?source_opus=2367
https://push-zb.helmholtz-muenchen.de/frontdoor.php?source_opus=2367