Zobrazeno 1 - 10
of 50
pro vyhledávání: '"Marta, Casal Moura"'
Autor:
John H Stone, Peter C Grayson, Ulrich Specks, E William St Clair, Peter A Merkel, Philip Seo, Carol A Langford, Paul A Monach, Cees G M Kallenberg, Robert F Spiera, Fernando C Fervenza, Zuoming Deng, Wei Tew, Marta Casal Moura, Stephen R Brooks, Marco Prunotto
Publikováno v:
RMD Open, Vol 9, Iss 1 (2023)
Background The frequency of proteinase 3 gene (PRTN3) polymorphisms in patients with antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis (AAV) is not fully characterised. We hypothesise that the presence of a PRTN3 gene polymorphism (sin
Externí odkaz:
https://doaj.org/article/6bc5f2edf8284a2885c3e9a25bbce574
Autor:
Marta Casal Moura, Carolina Branco, Joana Martins-Martinho, José Luís Ferraro, Alvise Berti, Estela Nogueira, Cristina Ponte
Publikováno v:
Therapeutic Advances in Musculoskeletal Disease, Vol 14 (2022)
In the past decade, unprecedented progress has been made in understanding the pathogenesis, diagnosis, assessment, and treatment of anti-neutrophil cytoplasmic antibody (ANCA)-associated vasculitides (AAVs). International collaborations and input fro
Externí odkaz:
https://doaj.org/article/105ab7310d2e49108f3e3e3af25fc023
Publikováno v:
Chest. 163:e207-e210
Autor:
Alvise Berti, Sophie Hillion, Maximilian F. Konig, Marta Casal Moura, Amber M. Hummel, Eva Carmona, Tobias Peikert, Fernando C. Fervenza, Cees G.M. Kallenberg, Carol A. Langford, Peter A. Merkel, Paul A. Monach, Philip Seo, Robert F. Spiera, Paul Brunetta, E. William St. Clair, Kristina M. Harris, John H. Stone, Guido Grandi, Jacques‐Olivier Pers, Ulrich Specks, Divi Cornec
Publikováno v:
Arthritis & Rheumatology. 75:736-747
Autor:
Marta Casal, Moura, Gwen E, Thompson, Darlene R, Nelson, Lynn A, Fussner, Amber M, Hummel, Dieter E, Jenne, Daniel, Emerling, Fernando C, Fervenza, Cees G M, Kallenberg, Carol A, Langford, W Joseph, McCune, Peter A, Merkel, Paul A, Monach, Philip, Seo, Robert F, Spiera, E William, St Clair, Steven R, Ytterberg, John H, Stone, William H, Robinson, Ulrich, Specks
Publikováno v:
Arthritis & Rheumatology. 75:748-759
Proteinase 3 (PR3) is the major antigen for anti-neutrophil cytoplasmic antibodies (ANCAs) in the systemic autoimmune vasculitis, granulomatosis with polyangiitis (GPA). PR3 anti-neutrophil cytoplasmic antibodies (PR3-ANCAs) recognize different epito
Autor:
Sanjeev Sethi, Benjamin Madden, Marta Casal Moura, Raman Deep Singh, Samih H. Nasr, Jean Hou, Alok Sharma, Karl A. Nath, Ulrich Specks, Fernando C. Fervenza, Mark Haas
Publikováno v:
Journal of the American Society of Nephrology. 34:374-384
Autor:
Marta Casal Moura, Ulrich Specks, Shahrzad Tehranian, Sanjeev Sethi, Dalia Zubidat, Luca Nardelli, Fernanda G. dos Santos, Ciria Sousa, Juan León-Róman, Shane A. Bobart, Eddie Greene, Ladan Zand, Fernando C. Fervenza
Publikováno v:
Clinical Journal of the American Society of Nephrology. 18:47-59
Autor:
Baptiste Chevet, Divi Cornec, Marta Casal Moura, Emilie Cornec-Le Gall, Fernando C Fervenza, Kenneth J Warrington, Ulrich Specks, Alvise Berti
Publikováno v:
Rheumatology. 62:1787-1803
ANCA-associated vasculitides (AAV) are a group of rare, primary, systemic necrotizing small-vessel vasculitides. Granulomatosis with polyangiitis and microscopic polyangiitis account for ∼80–90% of all AAV. Exposure to silica dust, farming and ch
Autor:
Marta Casal Moura, Cynthia S Crowson, Ulrich Specks, Kenneth J Warrington, Ladan Zand, Sanjeev Sethi, Fernando C Fervenza
Publikováno v:
Clinical Kidney Journal. 16:432-436
The risk of progression to end-stage kidney disease (ESKD) in patients with antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis (AAV) and glomerulonephritis (AAV-GN) remains high. At 5 years of follow-up, 14–25% of patients will evolve
Autor:
Yuan-Ping Pang, Marta Casal Moura, Gwen E. Thompson, Darlene R. Nelson, Amber M. Hummel, Dieter E. Jenne, Daniel Emerling, Wayne Volkmuth, William H. Robinson, Ulrich Specks
Publikováno v:
Frontiers in Immunology, Vol 10 (2019)
Mutants of a catalytically inactive variant of Proteinase 3 (PR3)—iPR3-Val103 possessing a Ser195Ala mutation relative to wild-type PR3-Val103—offer insights into how autoantigen PR3 interacts with antineutrophil cytoplasmic antibodies (ANCAs) in
Externí odkaz:
https://doaj.org/article/315a254bfbd1410fbcbda94f11de70d8