Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Marlieke G. Veldhuis"'
Autor:
Yuan Ji, Marlieke G. Veldhuis, Jantien Zandvoort, Fee L. Romunde, Marien J. C. Houtman, Karen Duran, Gijs van Haaften, Eva-Maria Zangerl-Plessl, Hiroki Takanari, Anna Stary-Weinzinger, Marcel A. G. van der Heyden
Publikováno v:
Journal of Biomedical Science, Vol 24, Iss 1, Pp 1-10 (2017)
Abstract Background The inward rectifier potassium current IK1 contributes to a stable resting membrane potential and phase 3 repolarization of the cardiac action potential. KCNJ2 gain-of-function mutations V93I and D172N associate with increased IK1
Externí odkaz:
https://doaj.org/article/efbd78c2599d487c96e896e9cb6f2763
Autor:
Bart Kok, Marlieke G. Veldhuis, Marien J C Houtman, Fee Romunde, Yuan Ji, Marcel A.G. van der Heyden, Muge Qile
Publikováno v:
Frontiers in Physiology, Vol 10 (2019)
Frontiers in Physiology, 10(JUL). Frontiers Research Foundation
Frontiers in Physiology, 10(JUL). Frontiers Research Foundation
K IR2.1 potassium channels, producing inward rectifier potassium current (I K1), are important for final action potential repolarization and a stable resting membrane potential in excitable cells like cardiomyocytes. Abnormal K IR2.1 function, either
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28644186d9318522dce90156e7e38ddf
https://dspace.library.uu.nl/handle/1874/387120
https://dspace.library.uu.nl/handle/1874/387120
Autor:
Anna Stary-Weinzinger, Karen Duran, M. Houtman, Marcel A.G. van der Heyden, Hiroki Takanari, J. Zandvoort, Marlieke G. Veldhuis, Em. Zangerl-Plessl, G van Haaften, Yuan Ji
Publikováno v:
EP Europace. 19:iii2-iii2
Autor:
Muge Qile, Marcel A.G. van der Heyden, M. Houtman, Yuan Ji, M A Vos, Marlieke G. Veldhuis, Bart Kok, Fee Romunde
Publikováno v:
EP Europace. 19:iii238-iii238
Publikováno v:
Adaptive Medicine, 7, 1. Taipei : Society of Adaptive Science in Taiwan
Inward rectifier currents carried by K_(IR)2.1 proteins have an important role in cardiac electrophysiology. Animal knock-outs and human loss-of-function mutation carriers experience cardiac pro-arrhythmia, but phenotypes are not confined to the hear