Zobrazeno 1 - 10
of 34
pro vyhledávání: '"Markus Riessland"'
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 18 (2024)
Externí odkaz:
https://doaj.org/article/c61f12a6aa54419aa57bb689f2bb6140
Autor:
Taylor Russo, Markus Riessland
Publikováno v:
Frontiers in Aging Neuroscience, Vol 14 (2022)
Immune responses are arising as a common feature of several neurodegenerative diseases, such as Parkinson’s disease (PD), Alzheimer’s disease (AD), and Amyotrophic Lateral Sclerosis (ALS), but their role as either causative or consequential remai
Externí odkaz:
https://doaj.org/article/ab52b00c173840dda177591f00082a9d
Autor:
Markus Riessland
Publikováno v:
Life, Vol 11, Iss 6, p 541 (2021)
Sixty years ago (1961), Hayflick and Moorhead reported that primary cells terminate their growth and stop dividing after ~50 passages or one year in culture [...]
Externí odkaz:
https://doaj.org/article/dcd0d50402ab46c0abe45babc6c30eff
Autor:
Magali Michaud, Thomas Arnoux, Serena Bielli, Estelle Durand, Yann Rotrou, Sibylle Jablonka, Fabrice Robert, Marc Giraudon-Paoli, Markus Riessland, Marie-Geneviève Mattei, Emile Andriambeloson, Brunhilde Wirth, Michael Sendtner, Jorge Gallego, Rebecca M. Pruss, Thierry Bordet
Publikováno v:
Neurobiology of Disease, Vol 38, Iss 1, Pp 125-135 (2010)
Spinal muscular atrophy (SMA) is caused by insufficient levels of the survival motor neuron (SMN) protein leading to muscle paralysis and respiratory failure. In mouse, introducing the human SMN2 gene partially rescues Smn-/- embryonic lethality. How
Externí odkaz:
https://doaj.org/article/3036b72971154587972567e407e550ca
Autor:
Tae Wan Kim, So Yeon Koo, Markus Riessland, Hyunwoo Cho, Fayzan Chaudhry, Benjamin Kolisnyk, Marco Vincenzo Russo, Nathalie Saurat, Sanjoy Mehta, Ralph Garippa, Doron Betel, Lorenz Studer
Publikováno v:
bioRxiv
Ongoing, first-in-human clinical trials illustrate the feasibility and translational potential of human pluripotent stem cell (hPSC)-based cell therapies in Parkinson’s disease (PD). However, a major unresolved challenge in the field is the extensi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a4f5aed9cea6a40c4718d48a481222d0
https://europepmc.org/articles/PMC10081262/
https://europepmc.org/articles/PMC10081262/
Autor:
Markus Riessland
Publikováno v:
Expert opinion on pharmacotherapy. 20(9)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3fe6df76464e055132e9ac9fcce7e28c
https://pubmed.ncbi.nlm.nih.gov/30892979
https://pubmed.ncbi.nlm.nih.gov/30892979
Autor:
Paul Greengard, Jae-Won Shim, Jason Ni, Kevin Dam, Jia Cheng, Emily J. Park, Lavoisier Ramos-Espiritu, Devrim Acehan, Jordan A. Pearson, Markus Riessland, Jack Zhang, Lars Brichta, Gabriele Ciceri, Tae Wan Kim, Benjamin Kolisnyk, Lorenz Studer, Wei Wang
Cellular senescence is a mechanism used by mitotic cells to prevent uncontrolled cell division. As senescent cells persist in tissues, they cause local inflammation and are harmful to surrounding cells, contributing to aging. Generally, neurodegenera
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e92dec3c02c7efbfcbf9e4920c50c7de
https://doi.org/10.1101/452243
https://doi.org/10.1101/452243
Publikováno v:
Expert Opinion on Investigational Drugs. 24:867-881
Currently, there is no cure available for the hereditary neurodegenerative disease proximal spinal muscular atrophy (SMA), which is the number one genetic killer in early childhood. However, growing knowledge of SMA pathophysiology has opened new ave
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::898d270e12fdc8f29da11d9e6b85befe
https://doi.org/10.1517/13543784.2015.1038341
https://doi.org/10.1517/13543784.2015.1038341
Autor:
Seyyedmohsen Hosseinibarkooie, Markus Riessland, Laura Torres-Benito, Natalia Mendoza-Ferreira, Eva Janzen, Svenja Schneider, Frank Rigo, Kristina Hupperich, Min Jeong Kye, Vanessa Grysko, Matthias Hammerschmidt, Brunhilde Wirth, Theresa Tschanz, C. Frank Bennett
Publikováno v:
Brain
Plastin3 is a protective modifier of spinal muscular atrophy (SMA). Janzen et al. report that CHP1 interacts directly with plastin3, and that CHP1 suppression reduces SMA pathology in cellular and animal models by restoring impaired endocytosis. CHP1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1728b8f988d42bd4b09fbaee70a6664
https://pubmed.ncbi.nlm.nih.gov/29961886
https://pubmed.ncbi.nlm.nih.gov/29961886