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pro vyhledávání: '"Mark J. Somers"'
Autor:
Margaret Tarpey, Tohru Fukai, David G. Harrison, S Kurz, Mark J. Somers, Jørn Bech Laursen, Bruce A. Freeman, Ascan Warnholtz, Louise McCann
Publikováno v:
Circulation. 103:1282-1288
Background —Altered endothelial cell nitric oxide (NO · ) production in atherosclerosis may be due to a reduction of intracellular tetrahydrobiopterin, which is a critical cofactor for NO synthase (NOS). In addition, previous literature suggests t
Publikováno v:
Antioxidants & Redox Signaling. 2:779-787
A growing body of evidence has suggested that a membrane-bound NADH/NADPH oxidase is the predominant source of reactive oxygen species (ROS) in vascular cells. Prior studies have used indirect assessments of superoxide including lucigenin-enhanced ch
Publikováno v:
Circulation. 101:1722-1728
Background —Angiotensin II–induced hypertension is associated with increased vascular superoxide production, which contributes to hypertension caused by the octapeptide. In cell culture, stretch increases endothelial and vascular smooth muscle pr
Autor:
David G. Harrison, Mark J. Somers
Publikováno v:
Current hypertension reports. 1(1)
During the past 15 years it has become clear that nitric oxide (NO(*)) released by endothelial cells plays a crucial role in vascular homeostasis. In addition to its role as a vasodilator, NO(*) inhibits platelet aggregation and smooth muscle prolife
Publikováno v:
Developments in Cardiovascular Medicine ISBN: 9781461371038
In the past decade, substantial attention has been directed toward understanding how reactive oxygen species contribute to the pathophysiology of vascular disease. Increasing evidence suggests that increases in vascular production of oxygenderived ra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::50d646370cb375ac1debd59315288534
https://doi.org/10.1007/978-1-4615-4649-8_17
https://doi.org/10.1007/978-1-4615-4649-8_17
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